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7. Current Knowledge on the Role of Cardiolipin Remodeling in the Context of Lipid Oxidation and Barth Syndrome. Liang Z; Schmidtke MW; Greenberg ML Front Mol Biosci; 2022; 9():915301. PubMed ID: 35693555 [TBL] [Abstract][Full Text] [Related]
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12. Barth Syndrome: From Mitochondrial Dysfunctions Associated with Aberrant Production of Reactive Oxygen Species to Pluripotent Stem Cell Studies. Saric A; Andreau K; Armand AS; Møller IM; Petit PX Front Genet; 2015; 6():359. PubMed ID: 26834781 [TBL] [Abstract][Full Text] [Related]
13. Barth syndrome: A life-threatening disorder caused by abnormal cardiolipin remodeling. Raja V; Reynolds CA; Greenberg ML J Rare Dis Res Treat; 2017; 2(2):58-62. PubMed ID: 31032491 [TBL] [Abstract][Full Text] [Related]
14. Cardiac metabolic pathways affected in the mouse model of barth syndrome. Huang Y; Powers C; Madala SK; Greis KD; Haffey WD; Towbin JA; Purevjav E; Javadov S; Strauss AW; Khuchua Z PLoS One; 2015; 10(6):e0128561. PubMed ID: 26030409 [TBL] [Abstract][Full Text] [Related]
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16. Expression of human monolysocardiolipin acyltransferase-1 improves mitochondrial function in Barth syndrome lymphoblasts. Mejia EM; Zegallai H; Bouchard ED; Banerji V; Ravandi A; Hatch GM J Biol Chem; 2018 May; 293(20):7564-7577. PubMed ID: 29563154 [TBL] [Abstract][Full Text] [Related]
17. Myocardial Energy Metabolism in Non-ischemic Cardiomyopathy. Greenwell AA; Gopal K; Ussher JR Front Physiol; 2020; 11():570421. PubMed ID: 33041869 [TBL] [Abstract][Full Text] [Related]
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