300 related articles for article (PubMed ID: 36450451)
1. Development and applications of a CRISPR activation system for facile genetic overexpression in Candida albicans.
Gervais NC; La Bella AA; Wensing LF; Sharma J; Acquaviva V; Best M; Cadena López RO; Fogal M; Uthayakumar D; Chavez A; Santiago-Tirado F; Flores-Mireles AL; Shapiro RS
G3 (Bethesda); 2023 Feb; 13(2):. PubMed ID: 36450451
[TBL] [Abstract][Full Text] [Related]
2. Functional genetic characterization of stress tolerance and biofilm formation in
Maroc L; Shaker H; Shapiro RS
mSphere; 2024 Feb; 9(2):e0076123. PubMed ID: 38265239
[TBL] [Abstract][Full Text] [Related]
3. A CRISPR Interference Platform for Efficient Genetic Repression in
Wensing L; Sharma J; Uthayakumar D; Proteau Y; Chavez A; Shapiro RS
mSphere; 2019 Feb; 4(1):. PubMed ID: 30760609
[TBL] [Abstract][Full Text] [Related]
4. Diminished Expression Alleles for Analysis of Virulence Traits and Genetic Interactions in Candida albicans.
Woolford CA; Mitchell AP
Methods Mol Biol; 2021; 2260():1-13. PubMed ID: 33405027
[TBL] [Abstract][Full Text] [Related]
5. Genetic Analysis of
Min K; Biermann A; Hogan DA; Konopka JB
mSphere; 2018 Nov; 3(6):. PubMed ID: 30463924
[TBL] [Abstract][Full Text] [Related]
6. An acquired mechanism of antifungal drug resistance simultaneously enables Candida albicans to escape from intrinsic host defenses.
Hampe IAI; Friedman J; Edgerton M; Morschhäuser J
PLoS Pathog; 2017 Sep; 13(9):e1006655. PubMed ID: 28953977
[TBL] [Abstract][Full Text] [Related]
7. Distinct roles of Candida albicans drug resistance transcription factors TAC1, MRR1, and UPC2 in virulence.
Lohberger A; Coste AT; Sanglard D
Eukaryot Cell; 2014 Jan; 13(1):127-42. PubMed ID: 24243794
[TBL] [Abstract][Full Text] [Related]
8. Rep1p negatively regulating MDR1 efflux pump involved in drug resistance in Candida albicans.
Chen CG; Yang YL; Tseng KY; Shih HI; Liou CH; Lin CC; Lo HJ
Fungal Genet Biol; 2009 Sep; 46(9):714-20. PubMed ID: 19527793
[TBL] [Abstract][Full Text] [Related]
9. Targeted Genetic Changes in Candida albicans Using Transient CRISPR-Cas9 Expression.
Huang MY; Cravener MC; Mitchell AP
Curr Protoc; 2021 Jan; 1(1):e19. PubMed ID: 33491919
[TBL] [Abstract][Full Text] [Related]
10. Implementation of a CRISPR-Based System for Gene Regulation in
Román E; Coman I; Prieto D; Alonso-Monge R; Pla J
mSphere; 2019 Feb; 4(1):. PubMed ID: 30760608
[TBL] [Abstract][Full Text] [Related]
11. Design and Generation of a CRISPR Interference System for Genetic Repression and Essential Gene Analysis in the Fungal Pathogen Candida albicans.
Wensing L; Shapiro RS
Methods Mol Biol; 2022; 2377():69-88. PubMed ID: 34709611
[TBL] [Abstract][Full Text] [Related]
12. A Hyperactive Form of the Zinc Cluster Transcription Factor Stb5 Causes
Ramírez-Zavala B; Manz H; Englert F; Rogers PD; Morschhäuser J
Antimicrob Agents Chemother; 2018 Dec; 62(12):. PubMed ID: 30249688
[TBL] [Abstract][Full Text] [Related]
13. Identification of Genomewide Alternative Splicing Events in Sequential, Isogenic Clinical Isolates of Candida albicans Reveals a Novel Mechanism of Drug Resistance and Tolerance to Cellular Stresses.
Muzafar S; Sharma RD; Shah AH; Gaur NA; Dasgupta U; Chauhan N; Prasad R
mSphere; 2020 Aug; 5(4):. PubMed ID: 32817456
[TBL] [Abstract][Full Text] [Related]
14. Mutations in the multi-drug resistance regulator MRR1, followed by loss of heterozygosity, are the main cause of MDR1 overexpression in fluconazole-resistant Candida albicans strains.
Dunkel N; Blass J; Rogers PD; Morschhäuser J
Mol Microbiol; 2008 Aug; 69(4):827-40. PubMed ID: 18577180
[TBL] [Abstract][Full Text] [Related]
15. A CRISPR-Cas9-based gene drive platform for genetic interaction analysis in Candida albicans.
Shapiro RS; Chavez A; Porter CBM; Hamblin M; Kaas CS; DiCarlo JE; Zeng G; Xu X; Revtovich AV; Kirienko NV; Wang Y; Church GM; Collins JJ
Nat Microbiol; 2018 Jan; 3(1):73-82. PubMed ID: 29062088
[TBL] [Abstract][Full Text] [Related]
16. PKC signaling regulates drug resistance of the fungal pathogen Candida albicans via circuitry comprised of Mkc1, calcineurin, and Hsp90.
LaFayette SL; Collins C; Zaas AK; Schell WA; Betancourt-Quiroz M; Gunatilaka AA; Perfect JR; Cowen LE
PLoS Pathog; 2010 Aug; 6(8):e1001069. PubMed ID: 20865172
[TBL] [Abstract][Full Text] [Related]
17. Competitive Fitness of Fluconazole-Resistant Clinical Candida albicans Strains.
Popp C; Hampe IAI; Hertlein T; Ohlsen K; Rogers PD; Morschhäuser J
Antimicrob Agents Chemother; 2017 Jul; 61(7):. PubMed ID: 28461316
[TBL] [Abstract][Full Text] [Related]
18. The transcription factor Ndt80 does not contribute to Mrr1-, Tac1-, and Upc2-mediated fluconazole resistance in Candida albicans.
Sasse C; Schillig R; Dierolf F; Weyler M; Schneider S; Mogavero S; Rogers PD; Morschhäuser J
PLoS One; 2011; 6(9):e25623. PubMed ID: 21980509
[TBL] [Abstract][Full Text] [Related]
19. Beauvericin Potentiates Azole Activity via Inhibition of Multidrug Efflux, Blocks Candida albicans Morphogenesis, and Is Effluxed via Yor1 and Circuitry Controlled by Zcf29.
Shekhar-Guturja T; Tebung WA; Mount H; Liu N; Köhler JR; Whiteway M; Cowen LE
Antimicrob Agents Chemother; 2016 Dec; 60(12):7468-7480. PubMed ID: 27736764
[TBL] [Abstract][Full Text] [Related]
20. Analysis of a fungus-specific transcription factor family, the Candida albicans zinc cluster proteins, by artificial activation.
Schillig R; Morschhäuser J
Mol Microbiol; 2013 Sep; 89(5):1003-17. PubMed ID: 23844834
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
