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9. A red-shifted, fast-relaxing azobenzene photoswitch for visible light control of an ionotropic glutamate receptor. Kienzler MA; Reiner A; Trautman E; Yoo S; Trauner D; Isacoff EY J Am Chem Soc; 2013 Nov; 135(47):17683-6. PubMed ID: 24171511 [TBL] [Abstract][Full Text] [Related]
10. Pharmacological properties and H+ sensitivity of excitatory amino acid receptor channels in rat cerebellar granule neurones. Traynelis SF; Cull-Candy SG J Physiol; 1991 Feb; 433():727-63. PubMed ID: 1726797 [TBL] [Abstract][Full Text] [Related]
11. Structure-activity analysis of binding kinetics for NMDA receptor competitive antagonists: the influence of conformational restriction. Benveniste M; Mayer ML Br J Pharmacol; 1991 Sep; 104(1):207-21. PubMed ID: 1686203 [TBL] [Abstract][Full Text] [Related]
12. Optical control of muscular nicotinic channels with azocuroniums, photoswitchable azobenzenes bearing two N-methyl-N-carbocyclic quaternary ammonium groups. Herrera-Arozamena C; Estrada-Valencia M; Martí-Marí O; Pérez C; de la Fuente Revenga M; Villalba-Galea CA; Rodríguez-Franco MI Eur J Med Chem; 2020 Aug; 200():112403. PubMed ID: 32447198 [TBL] [Abstract][Full Text] [Related]
17. Blockade by ifenprodil of high voltage-activated Ca2+ channels in rat and mouse cultured hippocampal pyramidal neurones: comparison with N-methyl-D-aspartate receptor antagonist actions. Church J; Fletcher EJ; Baxter K; MacDonald JF Br J Pharmacol; 1994 Oct; 113(2):499-507. PubMed ID: 7834201 [TBL] [Abstract][Full Text] [Related]
18. A novel class of amino-alkylcyclohexanes as uncompetitive, fast, voltage-dependent, N-methyl-D-aspartate (NMDA) receptor antagonists--in vitro characterization. Gilling K; Jatzke C; Wollenburg C; Vanejevs M; Kauss V; Jirgensons A; Parsons CG J Neural Transm (Vienna); 2007; 114(12):1529-37. PubMed ID: 17728997 [TBL] [Abstract][Full Text] [Related]
19. Distribution, density, and clustering of functional glutamate receptors before and after synaptogenesis in hippocampal neurons. Cottrell JR; Dubé GR; Egles C; Liu G J Neurophysiol; 2000 Sep; 84(3):1573-87. PubMed ID: 10980028 [TBL] [Abstract][Full Text] [Related]
20. Glutamate receptor antibodies in neurological diseases: anti-AMPA-GluR3 antibodies, anti-NMDA-NR1 antibodies, anti-NMDA-NR2A/B antibodies, anti-mGluR1 antibodies or anti-mGluR5 antibodies are present in subpopulations of patients with either: epilepsy, encephalitis, cerebellar ataxia, systemic lupus erythematosus (SLE) and neuropsychiatric SLE, Sjogren's syndrome, schizophrenia, mania or stroke. These autoimmune anti-glutamate receptor antibodies can bind neurons in few brain regions, activate glutamate receptors, decrease glutamate receptor's expression, impair glutamate-induced signaling and function, activate blood brain barrier endothelial cells, kill neurons, damage the brain, induce behavioral/psychiatric/cognitive abnormalities and ataxia in animal models, and can be removed or silenced in some patients by immunotherapy. Levite M J Neural Transm (Vienna); 2014 Aug; 121(8):1029-75. PubMed ID: 25081016 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]