BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

165 related articles for article (PubMed ID: 37522264)

  • 1. Preferential killing of melanoma cells by a p16-related peptide.
    Soo JK; Castle JT; Bennett DC
    Biol Open; 2023 Aug; 12(8):. PubMed ID: 37522264
    [TBL] [Abstract][Full Text] [Related]  

  • 2. p16/cyclin-dependent kinase inhibitor 2A deficiency in human melanocyte senescence, apoptosis, and immortalization: possible implications for melanoma progression.
    Sviderskaya EV; Gray-Schopfer VC; Hill SP; Smit NP; Evans-Whipp TJ; Bond J; Hill L; Bataille V; Peters G; Kipling D; Wynford-Thomas D; Bennett DC
    J Natl Cancer Inst; 2003 May; 95(10):723-32. PubMed ID: 12759390
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Melanin accumulation accelerates melanocyte senescence by a mechanism involving p16INK4a/CDK4/pRB and E2F1.
    Bandyopadhyay D; Medrano EE
    Ann N Y Acad Sci; 2000 Jun; 908():71-84. PubMed ID: 10911949
    [TBL] [Abstract][Full Text] [Related]  

  • 4. p16-Cdk4-Rb axis controls sensitivity to a cyclin-dependent kinase inhibitor PD0332991 in glioblastoma xenograft cells.
    Cen L; Carlson BL; Schroeder MA; Ostrem JL; Kitange GJ; Mladek AC; Fink SR; Decker PA; Wu W; Kim JS; Waldman T; Jenkins RB; Sarkaria JN
    Neuro Oncol; 2012 Jul; 14(7):870-81. PubMed ID: 22711607
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Deregulation of cell growth and apoptosis in UV-induced melanomagenesis.
    Ouhtit A; Gupta I; Gaur RL; Fernando A; Abd El-Azim AO; Eid A
    Front Biosci (Elite Ed); 2020 Mar; 12(2):223-236. PubMed ID: 32114459
    [TBL] [Abstract][Full Text] [Related]  

  • 6. p16(Ink4a) in melanocyte senescence and differentiation.
    Sviderskaya EV; Hill SP; Evans-Whipp TJ; Chin L; Orlow SJ; Easty DJ; Cheong SC; Beach D; DePinho RA; Bennett DC
    J Natl Cancer Inst; 2002 Mar; 94(6):446-54. PubMed ID: 11904317
    [TBL] [Abstract][Full Text] [Related]  

  • 7. The relative contributions of the p53 and pRb pathways in oncogene-induced melanocyte senescence.
    Haferkamp S; Tran SL; Becker TM; Scurr LL; Kefford RF; Rizos H
    Aging (Albany NY); 2009 May; 1(6):542-56. PubMed ID: 20157537
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Genome-Wide Overexpression Screen Identifies Genes Able to Bypass p16-Mediated Senescence in Melanoma.
    Lee WJ; Škalamera D; Dahmer-Heath M; Shakhbazov K; Ranall MV; Fox C; Lambie D; Stevenson AJ; Yaswen P; Gonda TJ; Gabrielli B
    SLAS Discov; 2017 Mar; 22(3):298-308. PubMed ID: 27872202
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Loss of retinoblastoma but not p16 function allows bypass of replicative senescence in human fibroblasts.
    Wei W; Herbig U; Wei S; Dutriaux A; Sedivy JM
    EMBO Rep; 2003 Nov; 4(11):1061-6. PubMed ID: 14566323
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Tumor suppressor p16INK4A is necessary for survival of cervical carcinoma cell lines.
    McLaughlin-Drubin ME; Park D; Munger K
    Proc Natl Acad Sci U S A; 2013 Oct; 110(40):16175-80. PubMed ID: 24046371
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Molecular regulation of melanocyte senescence.
    Bennett DC; Medrano EE
    Pigment Cell Res; 2002 Aug; 15(4):242-50. PubMed ID: 12100489
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Activation of a cAMP pathway and induction of melanogenesis correlate with association of p16(INK4) and p27(KIP1) to CDKs, loss of E2F-binding activity, and premature senescence of human melanocytes.
    Haddad MM; Xu W; Schwahn DJ; Liao F; Medrano EE
    Exp Cell Res; 1999 Dec; 253(2):561-72. PubMed ID: 10585280
    [TBL] [Abstract][Full Text] [Related]  

  • 13. E2FBP1 antagonizes the p16(INK4A)-Rb tumor suppressor machinery for growth suppression and cellular senescence by regulating promyelocytic leukemia protein stability.
    Fukuyo Y; Takahashi A; Hara E; Horikoshi N; Pandita TK; Nakajima T
    Int J Oral Sci; 2011 Oct; 3(4):200-8. PubMed ID: 22010578
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Melanomagenesis: overcoming the barrier of melanocyte senescence.
    Ha L; Merlino G; Sviderskaya EV
    Cell Cycle; 2008 Jul; 7(13):1944-8. PubMed ID: 18604170
    [TBL] [Abstract][Full Text] [Related]  

  • 15. p16INK4a-induced senescence is disabled by melanoma-associated mutations.
    Haferkamp S; Becker TM; Scurr LL; Kefford RF; Rizos H
    Aging Cell; 2008 Oct; 7(5):733-45. PubMed ID: 18843795
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Adenovirus-mediated overexpression of p15INK4B inhibits human glioma cell growth, induces replicative senescence, and inhibits telomerase activity similarly to p16INK4A.
    Fuxe J; Akusjärvi G; Goike HM; Roos G; Collins VP; Pettersson RF
    Cell Growth Differ; 2000 Jul; 11(7):373-84. PubMed ID: 10939591
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Cellular senescence in naevi and immortalisation in melanoma: a role for p16?
    Gray-Schopfer VC; Cheong SC; Chong H; Chow J; Moss T; Abdel-Malek ZA; Marais R; Wynford-Thomas D; Bennett DC
    Br J Cancer; 2006 Aug; 95(4):496-505. PubMed ID: 16880792
    [TBL] [Abstract][Full Text] [Related]  

  • 18. In vitro and in vivo tumor growth inhibition by a p16-mimicking peptide in p16INK4A-defective, pRb-positive human melanoma cells.
    Noonan DM; Severino A; Morini M; Tritarelli A; Manente L; D'Agnano I; Starace G; Baldi A; Lombardi D; Albini A; Felsani A; Paggi MG
    J Cell Physiol; 2005 Mar; 202(3):922-8. PubMed ID: 15389561
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Human melanocyte senescence and melanoma susceptibility genes.
    Bennett DC
    Oncogene; 2003 May; 22(20):3063-9. PubMed ID: 12789281
    [TBL] [Abstract][Full Text] [Related]  

  • 20. P16INK4a is required for hSNF5 chromatin remodeler-induced cellular senescence in malignant rhabdoid tumor cells.
    Oruetxebarria I; Venturini F; Kekarainen T; Houweling A; Zuijderduijn LM; Mohd-Sarip A; Vries RG; Hoeben RC; Verrijzer CP
    J Biol Chem; 2004 Jan; 279(5):3807-16. PubMed ID: 14604992
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 9.