241 related articles for article (PubMed ID: 37531097)
1. Cold and warmth intensify pain-linked sodium channel gating effects and persistent currents.
Kriegeskorte S; Bott R; Hampl M; Korngreen A; Hausmann R; Lampert A
J Gen Physiol; 2023 Sep; 155(9):. PubMed ID: 37531097
[TBL] [Abstract][Full Text] [Related]
2. Inherited pain: sodium channel Nav1.7 A1632T mutation causes erythromelalgia due to a shift of fast inactivation.
Eberhardt M; Nakajima J; Klinger AB; Neacsu C; Hühne K; O'Reilly AO; Kist AM; Lampe AK; Fischer K; Gibson J; Nau C; Winterpacht A; Lampert A
J Biol Chem; 2014 Jan; 289(4):1971-80. PubMed ID: 24311784
[TBL] [Abstract][Full Text] [Related]
3. Nav1.7-A1632G Mutation from a Family with Inherited Erythromelalgia: Enhanced Firing of Dorsal Root Ganglia Neurons Evoked by Thermal Stimuli.
Yang Y; Huang J; Mis MA; Estacion M; Macala L; Shah P; Schulman BR; Horton DB; Dib-Hajj SD; Waxman SG
J Neurosci; 2016 Jul; 36(28):7511-22. PubMed ID: 27413160
[TBL] [Abstract][Full Text] [Related]
4. Erythromelalgia mutation L823R shifts activation and inactivation of threshold sodium channel Nav1.7 to hyperpolarized potentials.
Lampert A; Dib-Hajj SD; Eastman EM; Tyrrell L; Lin Z; Yang Y; Waxman SG
Biochem Biophys Res Commun; 2009 Dec; 390(2):319-24. PubMed ID: 19800314
[TBL] [Abstract][Full Text] [Related]
5. Sodium channel slow inactivation interferes with open channel block.
Hampl M; Eberhardt E; O'Reilly AO; Lampert A
Sci Rep; 2016 May; 6():25974. PubMed ID: 27174182
[TBL] [Abstract][Full Text] [Related]
6. Human voltage-gated sodium channel mutations that cause inherited neuronal and muscle channelopathies increase resurgent sodium currents.
Jarecki BW; Piekarz AD; Jackson JO; Cummins TR
J Clin Invest; 2010 Jan; 120(1):369-78. PubMed ID: 20038812
[TBL] [Abstract][Full Text] [Related]
7. NaV1.7 gain-of-function mutations as a continuum: A1632E displays physiological changes associated with erythromelalgia and paroxysmal extreme pain disorder mutations and produces symptoms of both disorders.
Estacion M; Dib-Hajj SD; Benke PJ; Te Morsche RH; Eastman EM; Macala LJ; Drenth JP; Waxman SG
J Neurosci; 2008 Oct; 28(43):11079-88. PubMed ID: 18945915
[TBL] [Abstract][Full Text] [Related]
8. Novel SCN9A mutations underlying extreme pain phenotypes: unexpected electrophysiological and clinical phenotype correlations.
Emery EC; Habib AM; Cox JJ; Nicholas AK; Gribble FM; Woods CG; Reimann F
J Neurosci; 2015 May; 35(20):7674-81. PubMed ID: 25995458
[TBL] [Abstract][Full Text] [Related]
9. Phosphorylation of a chronic pain mutation in the voltage-gated sodium channel Nav1.7 increases voltage sensitivity.
Kerth CM; Hautvast P; Körner J; Lampert A; Meents JE
J Biol Chem; 2021; 296():100227. PubMed ID: 33361158
[TBL] [Abstract][Full Text] [Related]
10. Structural basis for severe pain caused by mutations in the S4-S5 linkers of voltage-gated sodium channel Na
Wisedchaisri G; Gamal El-Din TM; Zheng N; Catterall WA
Proc Natl Acad Sci U S A; 2023 Apr; 120(14):e2219624120. PubMed ID: 36996107
[TBL] [Abstract][Full Text] [Related]
11. Gain-of-function mutation of a voltage-gated sodium channel Na
Tanaka BS; Nguyen PT; Zhou EY; Yang Y; Yarov-Yarovoy V; Dib-Hajj SD; Waxman SG
J Biol Chem; 2017 Jun; 292(22):9262-9272. PubMed ID: 28381558
[TBL] [Abstract][Full Text] [Related]
12. Paroxysmal extreme pain disorder mutations within the D3/S4-S5 linker of Nav1.7 cause moderate destabilization of fast inactivation.
Jarecki BW; Sheets PL; Jackson JO; Cummins TR
J Physiol; 2008 Sep; 586(17):4137-53. PubMed ID: 18599537
[TBL] [Abstract][Full Text] [Related]
13. Nav1.7 mutations associated with paroxysmal extreme pain disorder, but not erythromelalgia, enhance Navbeta4 peptide-mediated resurgent sodium currents.
Theile JW; Jarecki BW; Piekarz AD; Cummins TR
J Physiol; 2011 Feb; 589(Pt 3):597-608. PubMed ID: 21115638
[TBL] [Abstract][Full Text] [Related]
14. Inhibition of Navβ4 peptide-mediated resurgent sodium currents in Nav1.7 channels by carbamazepine, riluzole, and anandamide.
Theile JW; Cummins TR
Mol Pharmacol; 2011 Oct; 80(4):724-34. PubMed ID: 21788423
[TBL] [Abstract][Full Text] [Related]
15. Alternative splicing of Na(V)1.7 exon 5 increases the impact of the painful PEPD mutant channel I1461T.
Jarecki BW; Sheets PL; Xiao Y; Jackson JO; Cummins TR
Channels (Austin); 2009; 3(4):259-67. PubMed ID: 19633428
[TBL] [Abstract][Full Text] [Related]
16. Mutations at opposite ends of the DIII/S4-S5 linker of sodium channel Na V 1.7 produce distinct pain disorders.
Cheng X; Dib-Hajj SD; Tyrrell L; Wright DA; Fischer TZ; Waxman SG
Mol Pain; 2010 Apr; 6():24. PubMed ID: 20429905
[TBL] [Abstract][Full Text] [Related]
17. Size matters: Erythromelalgia mutation S241T in Nav1.7 alters channel gating.
Lampert A; Dib-Hajj SD; Tyrrell L; Waxman SG
J Biol Chem; 2006 Nov; 281(47):36029-35. PubMed ID: 17008310
[TBL] [Abstract][Full Text] [Related]
18. Temperature dependence of erythromelalgia mutation L858F in sodium channel Nav1.7.
Han C; Lampert A; Rush AM; Dib-Hajj SD; Wang X; Yang Y; Waxman SG
Mol Pain; 2007 Jan; 3():3. PubMed ID: 17239250
[TBL] [Abstract][Full Text] [Related]
19. A Nav1.7 channel mutation associated with hereditary erythromelalgia contributes to neuronal hyperexcitability and displays reduced lidocaine sensitivity.
Sheets PL; Jackson JO; Waxman SG; Dib-Hajj SD; Cummins TR
J Physiol; 2007 Jun; 581(Pt 3):1019-31. PubMed ID: 17430993
[TBL] [Abstract][Full Text] [Related]
20. N1366S mutation of human skeletal muscle sodium channel causes paramyotonia congenita.
Ke Q; Ye J; Tang S; Wang J; Luo B; Ji F; Zhang X; Yu Y; Cheng X; Li Y
J Physiol; 2017 Nov; 595(22):6837-6850. PubMed ID: 28940424
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
