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4. Tafazzin deficiency causes substantial remodeling in the lipidome of a mouse model of Barth Syndrome cardiomyopathy. Hachmann M; Gülcan G; Rajendran R; Höring M; Liebisch G; Bachhuka A; Kohlhaas M; Maack C; Ergün S; Dudek J; Karnati S Front Mol Med; 2024; 4():1389456. PubMed ID: 39086433 [TBL] [Abstract][Full Text] [Related]
5. AAV-Mediated TAZ Gene Replacement Restores Mitochondrial and Cardioskeletal Function in Barth Syndrome. Suzuki-Hatano S; Saha M; Rizzo SA; Witko RL; Gosiker BJ; Ramanathan M; Soustek MS; Jones MD; Kang PB; Byrne BJ; Cade WT; Pacak CA Hum Gene Ther; 2019 Feb; 30(2):139-154. PubMed ID: 30070157 [TBL] [Abstract][Full Text] [Related]
6. Restoration of mitophagy ameliorates cardiomyopathy in Barth syndrome. Zhang J; Liu X; Nie J; Shi Y Autophagy; 2022 Sep; 18(9):2134-2149. PubMed ID: 34985382 [TBL] [Abstract][Full Text] [Related]
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14. Phospholipid abnormalities in children with Barth syndrome. Schlame M; Kelley RI; Feigenbaum A; Towbin JA; Heerdt PM; Schieble T; Wanders RJ; DiMauro S; Blanck TJ J Am Coll Cardiol; 2003 Dec; 42(11):1994-9. PubMed ID: 14662265 [TBL] [Abstract][Full Text] [Related]
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