185 related articles for article (PubMed ID: 38001078)
1. PDK4 rescues high-glucose-induced senescent fibroblasts and promotes diabetic wound healing through enhancing glycolysis and regulating YAP and JNK pathway.
Ma Z; Ding Y; Ding X; Mou H; Mo R; Tan Q
Cell Death Discov; 2023 Nov; 9(1):424. PubMed ID: 38001078
[TBL] [Abstract][Full Text] [Related]
2. PDK4 facilitates fibroblast functions and diabetic wound healing through regulation of HIF-1α protein stability and gene expression.
Ma Z; Mo R; Yang P; Ding Y; Zhang H; Dong Z; Chen Y; Tan Q
FASEB J; 2023 Oct; 37(10):e23215. PubMed ID: 37737961
[TBL] [Abstract][Full Text] [Related]
3. P-MSC-derived extracellular vesicles facilitate diabetic wound healing via miR-145-5p/ CDKN1A-mediated functional improvements of high glucose-induced senescent fibroblasts.
Su J; Wei Q; Ma K; Wang Y; Hu W; Meng H; Li Q; Zhang Y; Zhang W; Li H; Fu X; Zhang C
Burns Trauma; 2023; 11():tkad010. PubMed ID: 37860579
[TBL] [Abstract][Full Text] [Related]
4. Regenerative and protective effects of dMSC-sEVs on high-glucose-induced senescent fibroblasts by suppressing RAGE pathway and activating Smad pathway.
Bian X; Li B; Yang J; Ma K; Sun M; Zhang C; Fu X
Stem Cell Res Ther; 2020 Apr; 11(1):166. PubMed ID: 32349787
[TBL] [Abstract][Full Text] [Related]
5. Amniotic epithelial cells accelerate diabetic wound healing by protecting keratinocytes and fibroblasts from high-glucose-induced senescence.
Shu F; Gao H; Wu W; Yu S; Zhang L; Liu H; Xiao S; Xia Z; Zheng Y
Cell Biol Int; 2022 May; 46(5):755-770. PubMed ID: 35077602
[TBL] [Abstract][Full Text] [Related]
6. Enzyme-responsive nanospheres target senescent cells for diabetic wound healing by employing chemodynamic therapy.
Wei X; Zheng Z; Liu M; Yang Z; Xie E; Lin J; Gao Y; Tan R; She Z; Ma J; Yang L
Acta Biomater; 2023 Dec; 172():407-422. PubMed ID: 37848101
[TBL] [Abstract][Full Text] [Related]
7. Defective NCOA4-dependent ferroptosis in senescent fibroblasts retards diabetic wound healing.
Wei X; Liu M; Zheng Z; Yu S; Huang L; Ma J; Gao Y; Peng Y; Chen L; Tan R; She Z; Yang L
Cell Death Discov; 2023 Apr; 9(1):138. PubMed ID: 37117222
[TBL] [Abstract][Full Text] [Related]
8. PDK4-dependent hypercatabolism and lactate production of senescent cells promotes cancer malignancy.
Dou X; Fu Q; Long Q; Liu S; Zou Y; Fu D; Xu Q; Jiang Z; Ren X; Zhang G; Wei X; Li Q; Campisi J; Zhao Y; Sun Y
Nat Metab; 2023 Nov; 5(11):1887-1910. PubMed ID: 37903887
[TBL] [Abstract][Full Text] [Related]
9. Plasma endothelial cells-derived extracellular vesicles promote wound healing in diabetes through YAP and the PI3K/Akt/mTOR pathway.
Wei F; Wang A; Wang Q; Han W; Rong R; Wang L; Liu S; Zhang Y; Dong C; Li Y
Aging (Albany NY); 2020 Jun; 12(12):12002-12018. PubMed ID: 32570219
[TBL] [Abstract][Full Text] [Related]
10. Regenerative and protective effects of calcium silicate on senescent fibroblasts induced by high glucose.
Li B; Bian X; Hu W; Wang X; Li Q; Wang F; Sun M; Ma K; Zhang C; Chang J; Fu X
Wound Repair Regen; 2020 May; 28(3):315-325. PubMed ID: 31943524
[TBL] [Abstract][Full Text] [Related]
11. Altered regulation of mesenchymal cell senescence in adipose tissue promotes pathological changes associated with diabetic wound healing.
Kita A; Saito Y; Miura N; Miyajima M; Yamamoto S; Sato T; Yotsuyanagi T; Fujimiya M; Chikenji TS
Commun Biol; 2022 Apr; 5(1):310. PubMed ID: 35383267
[TBL] [Abstract][Full Text] [Related]
12. An analysis of replicative senescence in dermal fibroblasts derived from chronic leg wounds predicts that telomerase therapy would fail to reverse their disease-specific cellular and proteolytic phenotype.
Stephens P; Cook H; Hilton J; Jones CJ; Haughton MF; Wyllie FS; Skinner JW; Harding KG; Kipling D; Thomas DW
Exp Cell Res; 2003 Feb; 283(1):22-35. PubMed ID: 12565817
[TBL] [Abstract][Full Text] [Related]
13. Reduced Expression of YAP in Dermal Fibroblasts is Associated with Impaired Wound Healing in Type 2 Diabetic Mice.
Yu J; Choi S; Um J; Park KS
Tissue Eng Regen Med; 2017 Feb; 14(1):49-55. PubMed ID: 30603461
[TBL] [Abstract][Full Text] [Related]
14. Elevated skin senescence in young mice causes delayed wound healing.
Samdavid Thanapaul RJR; Shvedova M; Shin GH; Crouch J; Roh DS
Geroscience; 2022 Jun; 44(3):1871-1878. PubMed ID: 35399134
[TBL] [Abstract][Full Text] [Related]
15. Possible role of ginsenoside Rb1 in skin wound healing via regulating senescent skin dermal fibroblast.
Hou J; Kim S
Biochem Biophys Res Commun; 2018 May; 499(2):381-388. PubMed ID: 29577907
[TBL] [Abstract][Full Text] [Related]
16. Elevated Local Senescence in Diabetic Wound Healing Is Linked to Pathological Repair via CXCR2.
Wilkinson HN; Clowes C; Banyard KL; Matteuci P; Mace KA; Hardman MJ
J Invest Dermatol; 2019 May; 139(5):1171-1181.e6. PubMed ID: 30684552
[TBL] [Abstract][Full Text] [Related]
17. Cellular Senescence in Diabetes Mellitus: Distinct Senotherapeutic Strategies for Adipose Tissue and Pancreatic β Cells.
Murakami T; Inagaki N; Kondoh H
Front Endocrinol (Lausanne); 2022; 13():869414. PubMed ID: 35432205
[TBL] [Abstract][Full Text] [Related]
18. CCN2 induces cellular senescence in fibroblasts.
Jun JI; Lau LF
J Cell Commun Signal; 2017 Mar; 11(1):15-23. PubMed ID: 27752926
[TBL] [Abstract][Full Text] [Related]
19. Nε-carboxymethyl-lysine promotes calcium deposition in VSMCs via intracellular oxidative stress-induced PDK4 activation and alters glucose metabolism.
Ma WQ; Han XQ; Wang Y; Wang X; Zhu Y; Liu NF
Oncotarget; 2017 Dec; 8(68):112841-112854. PubMed ID: 29348870
[TBL] [Abstract][Full Text] [Related]
20. Rapamycin inhibits the secretory phenotype of senescent cells by a Nrf2-independent mechanism.
Wang R; Yu Z; Sunchu B; Shoaf J; Dang I; Zhao S; Caples K; Bradley L; Beaver LM; Ho E; Löhr CV; Perez VI
Aging Cell; 2017 Jun; 16(3):564-574. PubMed ID: 28371119
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
