These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
265 related articles for article (PubMed ID: 38068992)
41. Targeting of GSK3β promotes imatinib-mediated apoptosis in quiescent CD34+ chronic myeloid leukemia progenitors, preserving normal stem cells. Reddiconto G; Toto C; Palamà I; De Leo S; de Luca E; De Matteis S; Dini L; Passerini CG; Di Renzo N; Maffia M; Coluccia AM Blood; 2012 Mar; 119(10):2335-45. PubMed ID: 22262776 [TBL] [Abstract][Full Text] [Related]
42. Combined Therapy of ATRA and Imatinib Mesylate Decreases BCR-ABL and ABCB1/MDR1 Expression Through Cellular Differentiation in a Chronic Myeloid Leukemia Model. Pinto CA; DE Sousa Portilho AJ; Barbosa MC; DE Moraes MEA; DE Lemos JAR; Burbano RMR; Moreira-Nunes CA In Vivo; 2021; 35(5):2661-2667. PubMed ID: 34410954 [TBL] [Abstract][Full Text] [Related]
43. Discovery of a highly potent kinase inhibitor capable of overcoming multiple imatinib-resistant ABL mutants for chronic myeloid leukemia (CML). Lu T; Cao J; Zou F; Li X; Wang A; Wang W; Liang H; Liu Q; Hu C; Chen C; Hu Z; Wang W; Li L; Ge J; Shen Y; Ren T; Liu J; Xia R; Liu Q Eur J Pharmacol; 2021 Apr; 897():173944. PubMed ID: 33581133 [TBL] [Abstract][Full Text] [Related]
44. The novel anticancer agent JNJ-26854165 is active in chronic myeloid leukemic cells with unmutated BCR/ABL and T315I mutant BCR/ABL through promoting proteosomal degradation of BCR/ABL proteins. You L; Liu H; Huang J; Xie W; Wei J; Ye X; Qian W Oncotarget; 2017 Jan; 8(5):7777-7790. PubMed ID: 27999193 [TBL] [Abstract][Full Text] [Related]
45. Antitumor Effects of Blocking Protein Neddylation in T315I-BCR-ABL Leukemia Cells and Leukemia Stem Cells. Liu C; Nie D; Li J; Du X; Lu Y; Li Y; Zhou J; Jin Y; Pan J Cancer Res; 2018 Mar; 78(6):1522-1536. PubMed ID: 29321163 [TBL] [Abstract][Full Text] [Related]
46. Frequency of ABL gene mutations in chronic myeloid leukemia patients resistant to imatinib and results of treatment switch to second-generation tyrosine kinase inhibitors. Marcé S; Zamora L; Cabezón M; Xicoy B; Boqué C; Fernández C; Grau J; Navarro JT; Fernández de Sevilla A; Ribera JM; Feliu E; Millá F; Med Clin (Barc); 2013 Aug; 141(3):95-9. PubMed ID: 23433665 [TBL] [Abstract][Full Text] [Related]
47. Phenotypic and functional characterization of subpopulation of Imatinib resistant chronic myeloid leukemia cell line. Hekmatshoar Y; Karadag Gurel A; Ozkan T; Rahbar Saadat Y; Koc A; Karabay AZ; Bozkurt S; Sunguroglu A Adv Med Sci; 2023 Sep; 68(2):238-248. PubMed ID: 37421850 [TBL] [Abstract][Full Text] [Related]
48. BMS-214662 potently induces apoptosis of chronic myeloid leukemia stem and progenitor cells and synergizes with tyrosine kinase inhibitors. Copland M; Pellicano F; Richmond L; Allan EK; Hamilton A; Lee FY; Weinmann R; Holyoake TL Blood; 2008 Mar; 111(5):2843-53. PubMed ID: 18156496 [TBL] [Abstract][Full Text] [Related]
49. Histone demethylase PHF8 facilitates the development of chronic myeloid leukaemia by directly targeting BCR::ABL1. Feng H; Fu Y; Cui Z; Zhou M; Zhang L; Gao Z; Ma S; Chen C Br J Haematol; 2023 Sep; 202(6):1178-1191. PubMed ID: 37469124 [TBL] [Abstract][Full Text] [Related]
51. Expression of Hippo signaling pathway and Aurora kinase genes in chronic myeloid leukemia. Marsola APZC; Simões BP; Palma LC; Berzoti-Coelho MG; Burin SM; de Castro FA Med Oncol; 2018 Jan; 35(3):26. PubMed ID: 29387948 [TBL] [Abstract][Full Text] [Related]
52. Dual inhibition of STAT3 and STAT5 may overcome imatinib resistance in chronic myeloid leukemia. Yin L; Xu J; Wu W; Niu M; Li Z; Zhu F; Xu K Hematology; 2023 Dec; 28(1):2224625. PubMed ID: 37345979 [TBL] [Abstract][Full Text] [Related]
53. Axl Blockade by BGB324 Inhibits BCR-ABL Tyrosine Kinase Inhibitor-Sensitive and -Resistant Chronic Myeloid Leukemia. Ben-Batalla I; Erdmann R; Jørgensen H; Mitchell R; Ernst T; von Amsberg G; Schafhausen P; Velthaus JL; Rankin S; Clark RE; Koschmieder S; Schultze A; Mitra S; Vandenberghe P; Brümmendorf TH; Carmeliet P; Hochhaus A; Pantel K; Bokemeyer C; Helgason GV; Holyoake TL; Loges S Clin Cancer Res; 2017 May; 23(9):2289-2300. PubMed ID: 27856601 [No Abstract] [Full Text] [Related]
54. Chronic Myeloid Leukemia in the Era of Tyrosine Kinase Inhibitors: An Evolving Paradigm of Molecularly Targeted Therapy. Ali MA Mol Diagn Ther; 2016 Aug; 20(4):315-33. PubMed ID: 27220498 [TBL] [Abstract][Full Text] [Related]
55. Prosurvival kinase PIM2 is a therapeutic target for eradication of chronic myeloid leukemia stem cells. Ma L; Pak ML; Ou J; Yu J; St Louis P; Shan Y; Hutchinson L; Li S; Brehm MA; Zhu LJ; Green MR Proc Natl Acad Sci U S A; 2019 May; 116(21):10482-10487. PubMed ID: 31068472 [TBL] [Abstract][Full Text] [Related]
56. PD166326, a novel tyrosine kinase inhibitor, has greater antileukemic activity than imatinib mesylate in a murine model of chronic myeloid leukemia. Wolff NC; Veach DR; Tong WP; Bornmann WG; Clarkson B; Ilaria RL Blood; 2005 May; 105(10):3995-4003. PubMed ID: 15657179 [TBL] [Abstract][Full Text] [Related]
57. Imatinib-mesylate enhances the maintenance of chronic myeloid leukemia stem cell potential in the absence of glucose. Bono S; Dello Sbarba P; Lulli M Stem Cell Res; 2018 Apr; 28():33-38. PubMed ID: 29414416 [TBL] [Abstract][Full Text] [Related]
58. Dasatinib (BMS-354825) targets an earlier progenitor population than imatinib in primary CML but does not eliminate the quiescent fraction. Copland M; Hamilton A; Elrick LJ; Baird JW; Allan EK; Jordanides N; Barow M; Mountford JC; Holyoake TL Blood; 2006 Jun; 107(11):4532-9. PubMed ID: 16469872 [TBL] [Abstract][Full Text] [Related]
59. BCR-ABL1 mediated miR-150 downregulation through MYC contributed to myeloid differentiation block and drug resistance in chronic myeloid leukemia. Srutova K; Curik N; Burda P; Savvulidi F; Silvestri G; Trotta R; Klamova H; Pecherkova P; Sovova Z; Koblihova J; Stopka T; Perrotti D; Polakova KM Haematologica; 2018 Dec; 103(12):2016-2025. PubMed ID: 30049824 [TBL] [Abstract][Full Text] [Related]
60. CDKIs p18(INK4c) and p57(Kip2) are involved in quiescence of CML leukemic stem cells after treatment with TKI. Moreno-Lorenzana D; Avilés-Vazquez S; Sandoval Esquivel MA; Alvarado-Moreno A; Ortiz-Navarrete V; Torres-Martínez H; Ayala-Sánchez M; Mayani H; Chavez-Gonzalez A Cell Cycle; 2016 May; 15(9):1276-87. PubMed ID: 26985855 [TBL] [Abstract][Full Text] [Related] [Previous] [Next] [New Search]