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2. Common origin of pure and interrupted repeat expansions in spinocerebellar ataxia type 2 (SCA2). Ramos EM; Martins S; Alonso I; Emmel VE; Saraiva-Pereira ML; Jardim LB; Coutinho P; Sequeiros J; Silveira I Am J Med Genet B Neuropsychiatr Genet; 2010 Mar; 153B(2):524-531. PubMed ID: 19676102 [TBL] [Abstract][Full Text] [Related]
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6. Linkage disequilibrium at the SCA2 locus. Didierjean O; Cancel G; Stevanin G; Dürr A; Bürk K; Benomar A; Lezin A; Belal S; Abada-Bendid M; Klockgether T; Brice A J Med Genet; 1999 May; 36(5):415-7. PubMed ID: 10353790 [TBL] [Abstract][Full Text] [Related]
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8. CAG repeat instability at SCA2 locus: anchoring CAA interruptions and linked single nucleotide polymorphisms. Choudhry S; Mukerji M; Srivastava AK; Jain S; Brahmachari SK Hum Mol Genet; 2001 Oct; 10(21):2437-46. PubMed ID: 11689490 [TBL] [Abstract][Full Text] [Related]
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10. Linkage analysis and whole-exome sequencing exclude extra mutations responsible for the parkinsonian phenotype of spinocerebellar ataxia-2. Wang C; Xu Y; Feng X; Ma J; Xie S; Zhang Y; Tang BS; Chan P Neurobiol Aging; 2015 Jan; 36(1):545.e1-7. PubMed ID: 25189117 [TBL] [Abstract][Full Text] [Related]
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14. A common disease haplotype segregating in spinocerebellar ataxia 2 (SCA2) pedigrees of diverse ethnic origin. Pang J; Allotey R; Wadia N; Sasaki H; Bindoff L; Chamberlain S Eur J Hum Genet; 1999; 7(7):841-5. PubMed ID: 10573020 [TBL] [Abstract][Full Text] [Related]
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