215 related articles for article (PubMed ID: 38294810)
1. Dietary sodium alters aldosterone's effect on renal sodium transporter expression and distal convoluted tubule remodelling.
Mutchler SM; Hasan M; Murphy CP; Baty CJ; Boyd-Shiwarski C; Kirabo A; Kleyman TR
J Physiol; 2024 Mar; 602(5):967-987. PubMed ID: 38294810
[TBL] [Abstract][Full Text] [Related]
2. Deletion of renal Nedd4-2 abolishes the effect of high sodium intake (HS) on Kir4.1, ENaC, and NCC and causes hypokalemia during high HS.
Zhang DD; Duan XP; Xiao Y; Wu P; Gao ZX; Wang WH; Lin DH
Am J Physiol Renal Physiol; 2021 May; 320(5):F883-F896. PubMed ID: 33818128
[TBL] [Abstract][Full Text] [Related]
3. SPAK and OSR1 play essential roles in potassium homeostasis through actions on the distal convoluted tubule.
Ferdaus MZ; Barber KW; López-Cayuqueo KI; Terker AS; Argaiz ER; Gassaway BM; Chambrey R; Gamba G; Rinehart J; McCormick JA
J Physiol; 2016 Sep; 594(17):4945-66. PubMed ID: 27068441
[TBL] [Abstract][Full Text] [Related]
4. Regulation of renal Na transporters in response to dietary K.
Yang L; Xu S; Guo X; Uchida S; Weinstein AM; Wang T; Palmer LG
Am J Physiol Renal Physiol; 2018 Oct; 315(4):F1032-F1041. PubMed ID: 29923764
[TBL] [Abstract][Full Text] [Related]
5. Deletion of Kir5.1 abolishes the effect of high Na
Duan XP; Wu P; Zhang DD; Gao ZX; Xiao Y; Ray EC; Wang WH; Lin DH
Am J Physiol Renal Physiol; 2021 Jun; 320(6):F1045-F1058. PubMed ID: 33900854
[TBL] [Abstract][Full Text] [Related]
6. Deletion of renal Nedd4-2 abolishes the effect of high K
Xiao Y; Duan XP; Zhang DD; Wang WH; Lin DH
Am J Physiol Renal Physiol; 2021 Jul; 321(1):F1-F11. PubMed ID: 34029145
[TBL] [Abstract][Full Text] [Related]
7. Na(+) dependence of K(+) -induced natriuresis, kaliuresis and Na(+) /Cl(-) cotransporter dephosphorylation.
Jensen IS; Larsen CK; Leipziger J; Sørensen MV
Acta Physiol (Oxf); 2016 Sep; 218(1):49-61. PubMed ID: 27172453
[TBL] [Abstract][Full Text] [Related]
8. The mineralocorticoid receptor (MR) regulates ENaC but not NCC in mice with random MR deletion.
Czogalla J; Vohra T; Penton D; Kirschmann M; Craigie E; Loffing J
Pflugers Arch; 2016 May; 468(5):849-58. PubMed ID: 26898302
[TBL] [Abstract][Full Text] [Related]
9. Constitutively Active SPAK Causes Hyperkalemia by Activating NCC and Remodeling Distal Tubules.
Grimm PR; Coleman R; Delpire E; Welling PA
J Am Soc Nephrol; 2017 Sep; 28(9):2597-2606. PubMed ID: 28442491
[TBL] [Abstract][Full Text] [Related]
10. Dietary Na
Zachar R; Jensen BL; Svenningsen P
Am J Physiol Renal Physiol; 2019 Dec; 317(6):F1612-F1622. PubMed ID: 31566425
[TBL] [Abstract][Full Text] [Related]
11. Potassium intake modulates the thiazide-sensitive sodium-chloride cotransporter (NCC) activity via the Kir4.1 potassium channel.
Wang MX; Cuevas CA; Su XT; Wu P; Gao ZX; Lin DH; McCormick JA; Yang CL; Wang WH; Ellison DH
Kidney Int; 2018 Apr; 93(4):893-902. PubMed ID: 29310825
[TBL] [Abstract][Full Text] [Related]
12. Altered renal expression of Na(+) transporters and ROMK in protein-deprived rats.
Ruete MC; Carrizo LC; Bocanegra MV; Vallés PG
Nephron Physiol; 2009; 111(3):p17-29. PubMed ID: 19202345
[TBL] [Abstract][Full Text] [Related]
13. The renal thiazide-sensitive Na-Cl cotransporter as mediator of the aldosterone-escape phenomenon.
Wang XY; Masilamani S; Nielsen J; Kwon TH; Brooks HL; Nielsen S; Knepper MA
J Clin Invest; 2001 Jul; 108(2):215-22. PubMed ID: 11457874
[TBL] [Abstract][Full Text] [Related]
14. Dietary potassium restriction attenuates urinary sodium wasting in the generalized form of pseudohypoaldosteronism type 1.
Adachi M; Tajima T; Muroya K
CEN Case Rep; 2020 May; 9(2):133-137. PubMed ID: 31900739
[TBL] [Abstract][Full Text] [Related]
15. AT2R (Angiotensin II Type 2 Receptor)-Mediated Regulation of NCC (Na-Cl Cotransporter) and Renal K Excretion Depends on the K Channel, Kir4.1.
Wu P; Gao ZX; Duan XP; Su XT; Wang MX; Lin DH; Gu R; Wang WH
Hypertension; 2018 Apr; 71(4):622-630. PubMed ID: 29483225
[TBL] [Abstract][Full Text] [Related]
16. Expression and phosphorylation of the Na+-Cl- cotransporter NCC in vivo is regulated by dietary salt, potassium, and SGK1.
Vallon V; Schroth J; Lang F; Kuhl D; Uchida S
Am J Physiol Renal Physiol; 2009 Sep; 297(3):F704-12. PubMed ID: 19570885
[TBL] [Abstract][Full Text] [Related]
17. Sodium transporter abundance profiling in kidney: effect of spironolactone.
Nielsen J; Kwon TH; Masilamani S; Beutler K; Hager H; Nielsen S; Knepper MA
Am J Physiol Renal Physiol; 2002 Nov; 283(5):F923-33. PubMed ID: 12372767
[TBL] [Abstract][Full Text] [Related]
18. Time course of renal Na-K-ATPase, NHE3, NKCC2, NCC, and ENaC abundance changes with dietary NaCl restriction.
Masilamani S; Wang X; Kim GH; Brooks H; Nielsen J; Nielsen S; Nakamura K; Stokes JB; Knepper MA
Am J Physiol Renal Physiol; 2002 Oct; 283(4):F648-57. PubMed ID: 12217855
[TBL] [Abstract][Full Text] [Related]
19. Mg
Ferdaus MZ; Mukherjee A; Nelson JW; Blatt PJ; Miller LN; Terker AS; Staub O; Lin DH; McCormick JA
Am J Physiol Renal Physiol; 2019 Oct; 317(4):F825-F838. PubMed ID: 31364380
[TBL] [Abstract][Full Text] [Related]
20. Plasma Potassium Determines NCC Abundance in Adult Kidney-Specific
Boscardin E; Perrier R; Sergi C; Maillard MP; Loffing J; Loffing-Cueni D; Koesters R; Rossier BC; Hummler E
J Am Soc Nephrol; 2018 Mar; 29(3):977-990. PubMed ID: 29371419
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
