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3. Ovine ceroid-lipofuscinosis. I: Lipopigment composition is indicative of a lysosomal proteinosis. Palmer DN; Martinus RD; Barns G; Reeves RD; Jolly RD Am J Med Genet Suppl; 1988; 5():141-58. PubMed ID: 3146313 [TBL] [Abstract][Full Text] [Related]
4. Ceroid lipofuscinosis in sheep. II. The major component of the lipopigment in liver, kidney, pancreas, and brain is low molecular weight protein. Palmer DN; Barns G; Husbands DR; Jolly RD J Biol Chem; 1986 Feb; 261(4):1773-7. PubMed ID: 3944108 [TBL] [Abstract][Full Text] [Related]
5. Lysosomal storage of the DCCD reactive proteolipid subunit of mitochondrial ATP synthase in human and ovine ceroid lipofuscinoses. Palmer DN; Fearnley IM; Medd SM; Walker JE; Martinus RD; Bayliss SL; Hall NA; Lake BD; Wolfe LS; Jolly RD Adv Exp Med Biol; 1989; 266():211-22; discussion 223. PubMed ID: 2535017 [TBL] [Abstract][Full Text] [Related]
7. Ovine ceroid lipofuscinosis. The major lipopigment protein and the lipid-binding subunit of mitochondrial ATP synthase have the same NH2-terminal sequence. Palmer DN; Martinus RD; Cooper SM; Midwinter GG; Reid JC; Jolly RD J Biol Chem; 1989 Apr; 264(10):5736-40. PubMed ID: 2522438 [TBL] [Abstract][Full Text] [Related]
8. The sequence of the major protein stored in ovine ceroid lipofuscinosis is identical with that of the dicyclohexylcarbodiimide-reactive proteolipid of mitochondrial ATP synthase. Fearnley IM; Walker JE; Martinus RD; Jolly RD; Kirkland KB; Shaw GJ; Palmer DN Biochem J; 1990 Jun; 268(3):751-8. PubMed ID: 2141977 [TBL] [Abstract][Full Text] [Related]
9. Comparative biology of the neuronal ceroid-lipofuscinoses (NCL): an overview. Jolly RD Am J Med Genet; 1995 Jun; 57(2):307-11. PubMed ID: 7668352 [TBL] [Abstract][Full Text] [Related]
10. Ovine ceroid-lipofuscinosis II: Pathologic changes interpreted in light of biochemical observations. Jolly RD; Shimada A; Craig AS; Kirkland KB; Palmer DN Am J Med Genet Suppl; 1988; 5():159-70. PubMed ID: 3146314 [TBL] [Abstract][Full Text] [Related]
11. Bovine ceroid-lipofuscinosis (Batten's disease): the major component stored is the DCCD-reactive proteolipid, subunit C, of mitochondrial ATP synthase. Martinus RD; Harper PA; Jolly RD; Bayliss SL; Midwinter GG; Shaw GJ; Palmer DN Vet Res Commun; 1991; 15(2):85-94. PubMed ID: 1829867 [TBL] [Abstract][Full Text] [Related]
12. Arachidonic acid and other long-chain fatty acids in canine ceroid lipofuscinosis. Distribution in glycerolipids, metabolism, and pathophysiological correlations. Reddy TS; Armstrong D; Bazan NG Neurochem Pathol; 1985; 3(2):83-97. PubMed ID: 4047506 [TBL] [Abstract][Full Text] [Related]
13. Methylated lysine in storage body protein of sheep with hereditary ceroid-lipofuscinosis. Katz ML; Gerhardt KO Biochim Biophys Acta; 1992 Feb; 1138(2):97-108. PubMed ID: 1540666 [TBL] [Abstract][Full Text] [Related]
14. Analysis of phospholipid molecular species in brains from patients with infantile and juvenile neuronal-ceroid lipofuscinosis using liquid chromatography-electrospray ionization mass spectrometry. Käkelä R; Somerharju P; Tyynelä J J Neurochem; 2003 Mar; 84(5):1051-65. PubMed ID: 12603829 [TBL] [Abstract][Full Text] [Related]
15. Metabolomic investigation of CLN6 neuronal ceroid lipofuscinosis in affected South Hampshire sheep. Pears MR; Salek RM; Palmer DN; Kay GW; Mortishire-Smith RJ; Griffin JL J Neurosci Res; 2007 Nov; 85(15):3494-504. PubMed ID: 17510975 [TBL] [Abstract][Full Text] [Related]