517 related articles for article (PubMed ID: 7589089)
1. Major histocompatibility complex class II-associated peptides determine the binding of the superantigen toxic shock syndrome toxin-1.
von Bonin A; Ehrlich S; Malcherek G; Fleischer B
Eur J Immunol; 1995 Oct; 25(10):2894-8. PubMed ID: 7589089
[TBL] [Abstract][Full Text] [Related]
2. Carboxy-terminal residues of major histocompatibility complex class II-associated peptides control the presentation of the bacterial superantigen toxic shock syndrome toxin-1 to T cells.
Wen R; Broussard DR; Surman S; Hogg TL; Blackman MA; Woodland DL
Eur J Immunol; 1997 Mar; 27(3):772-81. PubMed ID: 9079821
[TBL] [Abstract][Full Text] [Related]
3. A recombinant single-chain human class II MHC molecule (HLA-DR1) as a covalently linked heterotrimer of alpha chain, beta chain, and antigenic peptide, with immunogenicity in vitro and reduced affinity for bacterial superantigens.
Zhu X; Bavari S; Ulrich R; Sadegh-Nasseri S; Ferrone S; McHugh L; Mage M
Eur J Immunol; 1997 Aug; 27(8):1933-41. PubMed ID: 9295029
[TBL] [Abstract][Full Text] [Related]
4. Identification from a phage display library of peptides that bind to toxic shock syndrome toxin-1 and that inhibit its binding to major histocompatibility complex (MHC) class II molecules.
Sato A; Ida N; Fukuyama M; Miwa K; Kazami J; Nakamura H
Biochemistry; 1996 Aug; 35(32):10441-7. PubMed ID: 8756700
[TBL] [Abstract][Full Text] [Related]
5. Inhibition of antigen-specific T cell activation by staphylococcal enterotoxins.
Dowd JE; Jenkins RN; Karp DR
J Immunol; 1995 Feb; 154(3):1024-31. PubMed ID: 7822779
[TBL] [Abstract][Full Text] [Related]
6. Intercellular adhesion molecule-1 and leukocyte function-associated antigen-3 provide costimulation for superantigen-induced T lymphocyte proliferation in the absence of a specific presenting molecule.
Lamphear JG; Stevens KR; Rich RR
J Immunol; 1998 Jan; 160(2):615-23. PubMed ID: 9551895
[TBL] [Abstract][Full Text] [Related]
7. Identification of binding domains on the superantigen, toxic shock syndrome-1, for class II MHC molecules.
Soos JM; Russell JK; Jarpe MA; Pontzer CH; Johnson HM
Biochem Biophys Res Commun; 1993 Mar; 191(3):1211-7. PubMed ID: 8466498
[TBL] [Abstract][Full Text] [Related]
8. Binding of toxic shock syndrome toxin-1 to murine major histocompatibility complex class II molecules.
Scholl PR; Sekaly RP; Diez A; Glimcher LH; Geha RS
Eur J Immunol; 1990 Sep; 20(9):1911-6. PubMed ID: 2209697
[TBL] [Abstract][Full Text] [Related]
9. Identification of MHC class II-associated peptides that promote the presentation of toxic shock syndrome toxin-1 to T cells.
Hogan RJ; VanBeek J; Broussard DR; Surman SL; Woodland DL
J Immunol; 2001 Jun; 166(11):6514-22. PubMed ID: 11359802
[TBL] [Abstract][Full Text] [Related]
10. Binary and ternary complexes between T-cell receptor, class II MHC and superantigen in vitro.
Seth A; Stern LJ; Ottenhoff TH; Engel I; Owen MJ; Lamb JR; Klausner RD; Wiley DC
Nature; 1994 May; 369(6478):324-7. PubMed ID: 8183371
[TBL] [Abstract][Full Text] [Related]
11. A novel HLA class II-independent TCR-mediated T cell activation mechanism is distinguished by the V beta specificity of the proliferating oligoclones and their capacity to generate interleukin-2.
Dennig D; Yan Y; Ferguson K; O'Reilly RJ
Cell Immunol; 1996 Aug; 171(2):200-10. PubMed ID: 8806788
[TBL] [Abstract][Full Text] [Related]
12. The alpha 1 domain of the HLA-DR molecule is essential for high-affinity binding of the toxic shock syndrome toxin-1.
Karp DR; Teletski CL; Scholl P; Geha R; Long EO
Nature; 1990 Aug; 346(6283):474-6. PubMed ID: 2377209
[TBL] [Abstract][Full Text] [Related]
13. Vbeta-restricted T cell adherence to endothelial cells: a mechanism for superantigen-dependent vascular injury.
Brogan PA; Shah V; Klein N; Dillon MJ
Arthritis Rheum; 2004 Feb; 50(2):589-97. PubMed ID: 14872503
[TBL] [Abstract][Full Text] [Related]
14. Superantigen natural affinity maturation revealed by the crystal structure of staphylococcal enterotoxin G and its binding to T-cell receptor Vbeta8.2.
Fernández MM; Bhattacharya S; De Marzi MC; Brown PH; Kerzic M; Schuck P; Mariuzza RA; Malchiodi EL
Proteins; 2007 Jul; 68(1):389-402. PubMed ID: 17427250
[TBL] [Abstract][Full Text] [Related]
15. Staphylococcal toxins bind to different sites on HLA-DR.
Chintagumpala MM; Mollick JA; Rich RR
J Immunol; 1991 Dec; 147(11):3876-81. PubMed ID: 1940373
[TBL] [Abstract][Full Text] [Related]
16. Cross-linking of MHC class II molecules by staphylococcal enterotoxin A is essential for antigen-presenting cell and T cell activation.
Tiedemann RE; Fraser JD
J Immunol; 1996 Nov; 157(9):3958-66. PubMed ID: 8892628
[TBL] [Abstract][Full Text] [Related]
17. Dissection of the function of HLA class II and costimulation in B cell-mediated and toxic shock syndrome toxin-1-induced T cell proliferation.
Dennig D; O'Reilly RJ
J Immunol; 1993 Jun; 150(12):5231-40. PubMed ID: 7685791
[TBL] [Abstract][Full Text] [Related]
18. Characterization of T cell receptors engineered for high affinity against toxic shock syndrome toxin-1.
Buonpane RA; Moza B; Sundberg EJ; Kranz DM
J Mol Biol; 2005 Oct; 353(2):308-21. PubMed ID: 16171815
[TBL] [Abstract][Full Text] [Related]
19. A toxic shock syndrome toxin-1 peptide that shows homology to mycobacterial heat shock protein 18 is presented as conventional antigen to T cells by multiple HLA-DR alleles.
Ramesh N; Spertini F; Scholl P; Geha R
J Immunol; 1992 Feb; 148(4):1025-30. PubMed ID: 1737925
[TBL] [Abstract][Full Text] [Related]
20. Effect of isotypes and allelic polymorphism on the binding of staphylococcal exotoxins to MHC class II molecules.
Scholl PR; Diez A; Karr R; Sekaly RP; Trowsdale J; Geha RS
J Immunol; 1990 Jan; 144(1):226-30. PubMed ID: 2404064
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
