BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

200 related articles for article (PubMed ID: 7862672)

  • 1. Superoxide dismutase is an abundant component in cell bodies, dendrites, and axons of motor neurons and in a subset of other neurons.
    Pardo CA; Xu Z; Borchelt DR; Price DL; Sisodia SS; Cleveland DW
    Proc Natl Acad Sci U S A; 1995 Feb; 92(4):954-8. PubMed ID: 7862672
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Somatodendritic accumulation of misfolded SOD1-L126Z in motor neurons mediates degeneration: alphaB-crystallin modulates aggregation.
    Wang J; Xu G; Li H; Gonzales V; Fromholt D; Karch C; Copeland NG; Jenkins NA; Borchelt DR
    Hum Mol Genet; 2005 Aug; 14(16):2335-47. PubMed ID: 16000321
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D; Haasdijk ED; Grashorn JA; Hawkins R; van Duijn W; Verspaget HW; London J; Holstege JC
    Neurobiol Dis; 2000 Dec; 7(6 Pt B):623-43. PubMed ID: 11114261
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Axonal transport of mutant superoxide dismutase 1 and focal axonal abnormalities in the proximal axons of transgenic mice.
    Borchelt DR; Wong PC; Becher MW; Pardo CA; Lee MK; Xu ZS; Thinakaran G; Jenkins NA; Copeland NG; Sisodia SS; Cleveland DW; Price DL; Hoffman PN
    Neurobiol Dis; 1998 Jul; 5(1):27-35. PubMed ID: 9702785
    [TBL] [Abstract][Full Text] [Related]  

  • 5. An adverse property of a familial ALS-linked SOD1 mutation causes motor neuron disease characterized by vacuolar degeneration of mitochondria.
    Wong PC; Pardo CA; Borchelt DR; Lee MK; Copeland NG; Jenkins NA; Sisodia SS; Cleveland DW; Price DL
    Neuron; 1995 Jun; 14(6):1105-16. PubMed ID: 7605627
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Galectin-1 deficiency improves axonal swelling of motor neurones in SOD1(G93A) transgenic mice.
    Kobayakawa Y; Sakumi K; Kajitani K; Kadoya T; Horie H; Kira J; Nakabeppu Y
    Neuropathol Appl Neurobiol; 2015 Feb; 41(2):227-44. PubMed ID: 24707896
    [TBL] [Abstract][Full Text] [Related]  

  • 7. [Familial amyotrophic lateral sclerosis and mutations in the Cu/Zn superoxide dismutase gene].
    Nakano R
    Rinsho Shinkeigaku; 1995 Dec; 35(12):1546-8. PubMed ID: 8752459
    [TBL] [Abstract][Full Text] [Related]  

  • 8. TDP-43 immunoreactivity in neuronal inclusions in familial amyotrophic lateral sclerosis with or without SOD1 gene mutation.
    Tan CF; Eguchi H; Tagawa A; Onodera O; Iwasaki T; Tsujino A; Nishizawa M; Kakita A; Takahashi H
    Acta Neuropathol; 2007 May; 113(5):535-42. PubMed ID: 17333220
    [TBL] [Abstract][Full Text] [Related]  

  • 9. The copper chaperone CCS is abundant in neurons and astrocytes in human and rodent brain.
    Rothstein JD; Dykes-Hoberg M; Corson LB; Becker M; Cleveland DW; Price DL; Culotta VC; Wong PC
    J Neurochem; 1999 Jan; 72(1):422-9. PubMed ID: 9886096
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Immunodetection of disease-associated conformers of mutant cu/zn superoxide dismutase 1 selectively expressed in degenerating neurons in amyotrophic lateral sclerosis.
    Sábado J; Casanovas A; Hernández S; Piedrafita L; Hereu M; Esquerda JE
    J Neuropathol Exp Neurol; 2013 Jul; 72(7):646-61. PubMed ID: 23771221
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Mouse motor neuron disease caused by truncated SOD1 with or without C-terminal modification.
    Watanabe Y; Yasui K; Nakano T; Doi K; Fukada Y; Kitayama M; Ishimoto M; Kurihara S; Kawashima M; Fukuda H; Adachi Y; Inoue T; Nakashima K
    Brain Res Mol Brain Res; 2005 Apr; 135(1-2):12-20. PubMed ID: 15857664
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Restricted expression of mutant SOD1 in spinal motor neurons and interneurons induces motor neuron pathology.
    Wang L; Sharma K; Deng HX; Siddique T; Grisotti G; Liu E; Roos RP
    Neurobiol Dis; 2008 Mar; 29(3):400-8. PubMed ID: 18054242
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Wild-type nonneuronal cells extend survival of SOD1 mutant motor neurons in ALS mice.
    Clement AM; Nguyen MD; Roberts EA; Garcia ML; Boillée S; Rule M; McMahon AP; Doucette W; Siwek D; Ferrante RJ; Brown RH; Julien JP; Goldstein LS; Cleveland DW
    Science; 2003 Oct; 302(5642):113-7. PubMed ID: 14526083
    [TBL] [Abstract][Full Text] [Related]  

  • 14. The Golgi apparatus of spinal cord motor neurons in transgenic mice expressing mutant Cu,Zn superoxide dismutase becomes fragmented in early, preclinical stages of the disease.
    Mourelatos Z; Gonatas NK; Stieber A; Gurney ME; Dal Canto MC
    Proc Natl Acad Sci U S A; 1996 May; 93(11):5472-7. PubMed ID: 8643599
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Oxidative stress, mutant SOD1, and neurofilament pathology in transgenic mouse models of human motor neuron disease.
    Tu PH; Gurney ME; Julien JP; Lee VM; Trojanowski JQ
    Lab Invest; 1997 Apr; 76(4):441-56. PubMed ID: 9111507
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Relationship of oxygen radical-induced lipid peroxidative damage to disease onset and progression in a transgenic model of familial ALS.
    Hall ED; Andrus PK; Oostveen JA; Fleck TJ; Gurney ME
    J Neurosci Res; 1998 Jul; 53(1):66-77. PubMed ID: 9670993
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Recent advances in research on neuropathological aspects of familial amyotrophic lateral sclerosis with superoxide dismutase 1 gene mutations: neuronal Lewy body-like hyaline inclusions and astrocytic hyaline inclusions.
    Kato S; Saito M; Hirano A; Ohama E
    Histol Histopathol; 1999 Jul; 14(3):973-89. PubMed ID: 10425565
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Inhibition of p38 mitogen activated protein kinase activation and mutant SOD1(G93A)-induced motor neuron death.
    Dewil M; dela Cruz VF; Van Den Bosch L; Robberecht W
    Neurobiol Dis; 2007 May; 26(2):332-41. PubMed ID: 17346981
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Therapeutic benefits of putrescine-modified catalase in a transgenic mouse model of familial amyotrophic lateral sclerosis.
    Reinholz MM; Merkle CM; Poduslo JF
    Exp Neurol; 1999 Sep; 159(1):204-16. PubMed ID: 10486188
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Assessment of metal concentrations in the SOD1
    Enge TG; Ecroyd H; Jolley DF; Yerbury JJ; Kalmar B; Dosseto A
    Mol Cell Neurosci; 2018 Apr; 88():319-329. PubMed ID: 29524628
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 10.