219 related articles for article (PubMed ID: 7902566)
1. Identification of a domain required for oncogenic activity and transcriptional suppression by v-erbA and thyroid-hormone receptor alpha.
Damm K; Evans RM
Proc Natl Acad Sci U S A; 1993 Nov; 90(22):10668-72. PubMed ID: 7902566
[TBL] [Abstract][Full Text] [Related]
2. Thyroid hormone receptor/and v-erbA. A single amino acid difference in the C-terminal region influences dominant negative activity and receptor dimer formation.
Selmi S; Samuels HH
J Biol Chem; 1991 Jun; 266(18):11589-93. PubMed ID: 1675637
[TBL] [Abstract][Full Text] [Related]
3. v-erbA oncogene activation entails the loss of hormone-dependent regulator activity of c-erbA.
Zenke M; Muñoz A; Sap J; Vennström B; Beug H
Cell; 1990 Jun; 61(6):1035-49. PubMed ID: 1972036
[TBL] [Abstract][Full Text] [Related]
4. Phosphorylation of the v-erbA protein is required for its function as an oncogene.
Glineur C; Zenke M; Beug H; Ghysdael J
Genes Dev; 1990 Oct; 4(10):1663-76. PubMed ID: 1979040
[TBL] [Abstract][Full Text] [Related]
5. The lack of transcriptional activation of the v-erbA oncogene is in part due to a mutation present in the DNA binding domain of the protein.
de Verneuil H; Metzger D
Nucleic Acids Res; 1990 Aug; 18(15):4489-97. PubMed ID: 1975094
[TBL] [Abstract][Full Text] [Related]
6. Myb-Ets fusion oncoprotein inhibits thyroid hormone receptor/c-ErbA and retinoic acid receptor functions: a novel mechanism of action for leukemogenic transformation by E26 avian retrovirus.
Rascle A; Ferrand N; Gandrillon O; Samarut J
Mol Cell Biol; 1996 Nov; 16(11):6338-51. PubMed ID: 8887663
[TBL] [Abstract][Full Text] [Related]
7. v-erbA acts on retinoic acid receptors in immature avian erythroid cells.
Sande S; Sharif M; Chen H; Privalsky M
J Virol; 1993 Feb; 67(2):1067-74. PubMed ID: 8093487
[TBL] [Abstract][Full Text] [Related]
8. A conserved C-terminal sequence that is deleted in v-ErbA is essential for the biological activities of c-ErbA (the thyroid hormone receptor).
Saatcioglu F; Bartunek P; Deng T; Zenke M; Karin M
Mol Cell Biol; 1993 Jun; 13(6):3675-85. PubMed ID: 8098843
[TBL] [Abstract][Full Text] [Related]
9. Ontogeny of the v-erbA oncoprotein from the thyroid hormone receptor: an alteration in the DNA binding domain plays a role crucial for v-erbA function.
Bonde BG; Sharif M; Privalsky ML
J Virol; 1991 Apr; 65(4):2037-46. PubMed ID: 1672166
[TBL] [Abstract][Full Text] [Related]
10. c-ErbA, but not v-ErbA, competes with a putative erythroid repressor for binding to the carbonic anhydrase II promoter.
Rascle A; Ghysdael J; Samarut J
Oncogene; 1994 Oct; 9(10):2853-67. PubMed ID: 7916146
[TBL] [Abstract][Full Text] [Related]
11. A transferable silencing domain is present in the thyroid hormone receptor, in the v-erbA oncogene product and in the retinoic acid receptor.
Baniahmad A; Köhne AC; Renkawitz R
EMBO J; 1992 Mar; 11(3):1015-23. PubMed ID: 1347744
[TBL] [Abstract][Full Text] [Related]
12. V-erbA requires auxiliary proteins for dominant negative activity.
Hermann T; Hoffmann B; Piedrafita FJ; Zhang XK; Pfahl M
Oncogene; 1993 Jan; 8(1):55-65. PubMed ID: 8093812
[TBL] [Abstract][Full Text] [Related]
13. Unliganded T3R, but not its oncogenic variant, v-erbA, suppresses RAR-dependent transactivation by titrating out RXR.
Barettino D; Bugge TH; Bartunek P; Vivanco Ruiz MD; Sonntag-Buck V; Beug H; Zenke M; Stunnenberg HG
EMBO J; 1993 Apr; 12(4):1343-54. PubMed ID: 8096810
[TBL] [Abstract][Full Text] [Related]
14. The viral erbA oncogene protein, a constitutive repressor in animal cells, is a hormone-regulated activator in yeast.
Privalsky ML; Sharif M; Yamamoto KR
Cell; 1990 Dec; 63(6):1277-86. PubMed ID: 1979758
[TBL] [Abstract][Full Text] [Related]
15. v-erbA oncogene function in neoplasia correlates with its ability to repress retinoic acid receptor action.
Sharif M; Privalsky ML
Cell; 1991 Sep; 66(5):885-93. PubMed ID: 1679679
[TBL] [Abstract][Full Text] [Related]
16. A subpopulation of the avian erythroblastosis virus v-erbA protein, a member of the nuclear hormone receptor family, is glycosylated.
Privalsky ML
J Virol; 1990 Jan; 64(1):463-6. PubMed ID: 1967151
[TBL] [Abstract][Full Text] [Related]
17. Modulation of normal erythroid differentiation by the endogenous thyroid hormone and retinoic acid receptors: a possible target for v-erbA oncogene action.
Schroeder C; Gibson L; Zenke M; Beug H
Oncogene; 1992 Feb; 7(2):217-27. PubMed ID: 1347914
[TBL] [Abstract][Full Text] [Related]
18. The leukaemia oncogene v-erbA: a dominant negative version of ligand dependent transcription factors that regulates red cell differentiation?
Ghysdael J; Beug H
Cancer Surv; 1992; 14():169-80. PubMed ID: 1358441
[TBL] [Abstract][Full Text] [Related]
19. Requirement for the C-terminal domain of the v-erbA oncogene protein for biological function and transcriptional repression.
Forrest D; Muñoz A; Raynoschek C; Vennström B; Beug H
Oncogene; 1990 Mar; 5(3):309-16. PubMed ID: 1969136
[TBL] [Abstract][Full Text] [Related]
20. v-erbA overexpression is required to extinguish c-erbA function in erythroid cell differentiation and regulation of the erbA target gene CAII.
Disela C; Glineur C; Bugge T; Sap J; Stengl G; Dodgson J; Stunnenberg H; Beug H; Zenke M
Genes Dev; 1991 Nov; 5(11):2033-47. PubMed ID: 1682217
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
