223 related articles for article (PubMed ID: 7916146)
1. c-ErbA, but not v-ErbA, competes with a putative erythroid repressor for binding to the carbonic anhydrase II promoter.
Rascle A; Ghysdael J; Samarut J
Oncogene; 1994 Oct; 9(10):2853-67. PubMed ID: 7916146
[TBL] [Abstract][Full Text] [Related]
2. The carbonic anhydrase II gene, a gene regulated by thyroid hormone and erythropoietin, is repressed by the v-erbA oncogene in erythrocytic cells.
Pain B; Melet F; Jurdic P; Samarut J
New Biol; 1990 Mar; 2(3):284-94. PubMed ID: 2126201
[TBL] [Abstract][Full Text] [Related]
3. Leukemic transformation by the v-ErbA oncoprotein entails constitutive binding to and repression of an erythroid enhancer in vivo.
Ciana P; Braliou GG; Demay FG; von Lindern M; Barettino D; Beug H; Stunnenberg HG
EMBO J; 1998 Dec; 17(24):7382-94. PubMed ID: 9857194
[TBL] [Abstract][Full Text] [Related]
4. The v-ErbA oncoprotein quenches the activity of an erythroid-specific enhancer.
Braliou GG; Ciana P; Klaassen W; Gandrillon O; Stunnenberg HG
Oncogene; 2001 Feb; 20(7):775-87. PubMed ID: 11314012
[TBL] [Abstract][Full Text] [Related]
5. The oncoprotein P75gag-v-erbA represses thyroid hormone induced transcription only via response elements containing palindromic half-sites.
Wahlström GM; Harbers M; Vennström B
Oncogene; 1996 Aug; 13(4):843-52. PubMed ID: 8761306
[TBL] [Abstract][Full Text] [Related]
6. V-erbA requires auxiliary proteins for dominant negative activity.
Hermann T; Hoffmann B; Piedrafita FJ; Zhang XK; Pfahl M
Oncogene; 1993 Jan; 8(1):55-65. PubMed ID: 8093812
[TBL] [Abstract][Full Text] [Related]
7. V-erbA and c-erbA proteins enhance transcriptional activation by c-jun.
Sharif M; Privalsky ML
Oncogene; 1992 May; 7(5):953-60. PubMed ID: 1349165
[TBL] [Abstract][Full Text] [Related]
8. Modulation of normal erythroid differentiation by the endogenous thyroid hormone and retinoic acid receptors: a possible target for v-erbA oncogene action.
Schroeder C; Gibson L; Zenke M; Beug H
Oncogene; 1992 Feb; 7(2):217-27. PubMed ID: 1347914
[TBL] [Abstract][Full Text] [Related]
9. The v-erbA oncogene requires cooperation with tyrosine kinases to arrest erythroid differentiation induced by ligand-activated endogenous c-erbA and retinoic acid receptor.
Schroeder C; Gibson L; Beug H
Oncogene; 1992 Feb; 7(2):203-16. PubMed ID: 1347913
[TBL] [Abstract][Full Text] [Related]
10. v-erbA overexpression is required to extinguish c-erbA function in erythroid cell differentiation and regulation of the erbA target gene CAII.
Disela C; Glineur C; Bugge T; Sap J; Stengl G; Dodgson J; Stunnenberg H; Beug H; Zenke M
Genes Dev; 1991 Nov; 5(11):2033-47. PubMed ID: 1682217
[TBL] [Abstract][Full Text] [Related]
11. c-erbA alpha/T3R and RARs control commitment of hematopoietic self-renewing progenitor cells to apoptosis or differentiation and are antagonized by the v-erbA oncogene.
Gandrillon O; Ferrand N; Michaille JJ; Roze L; Zile MH; Samarut J
Oncogene; 1994 Mar; 9(3):749-58. PubMed ID: 7906409
[TBL] [Abstract][Full Text] [Related]
12. Requirement for the C-terminal domain of the v-erbA oncogene protein for biological function and transcriptional repression.
Forrest D; Muñoz A; Raynoschek C; Vennström B; Beug H
Oncogene; 1990 Mar; 5(3):309-16. PubMed ID: 1969136
[TBL] [Abstract][Full Text] [Related]
13. The leukaemia oncogene v-erbA: a dominant negative version of ligand dependent transcription factors that regulates red cell differentiation?
Ghysdael J; Beug H
Cancer Surv; 1992; 14():169-80. PubMed ID: 1358441
[TBL] [Abstract][Full Text] [Related]
14. Mechanism of transformation by v-ErbA: substitution for steroid hormone receptor function in self renewal induction.
Bauer A; Ulrich E; Andersson M; Beug H; von Lindern M
Oncogene; 1997 Aug; 15(6):701-15. PubMed ID: 9264411
[TBL] [Abstract][Full Text] [Related]
15. The c-erbA alpha-encoded thyroid hormone receptor is phosphorylated in its amino terminal domain by casein kinase II.
Glineur C; Bailly M; Ghysdael J
Oncogene; 1989 Oct; 4(10):1247-54. PubMed ID: 2552374
[TBL] [Abstract][Full Text] [Related]
16. NeP1. A ubiquitous transcription factor synergizes with v-ERBA in transcriptional silencing.
Köhne AC; Baniahmad A; Renkawitz R
J Mol Biol; 1993 Aug; 232(3):747-55. PubMed ID: 8102652
[TBL] [Abstract][Full Text] [Related]
17. Ligand-independent repression by the thyroid hormone receptor mediated by a nuclear receptor co-repressor.
Hörlein AJ; Näär AM; Heinzel T; Torchia J; Gloss B; Kurokawa R; Ryan A; Kamei Y; Söderström M; Glass CK
Nature; 1995 Oct; 377(6548):397-404. PubMed ID: 7566114
[TBL] [Abstract][Full Text] [Related]
18. Functional interaction between the two zinc finger domains of the v-erb A oncoprotein.
Hall BL; Bonde BG; Judelson C; Privalsky ML
Cell Growth Differ; 1992 Apr; 3(4):207-16. PubMed ID: 1355356
[TBL] [Abstract][Full Text] [Related]
19. The thyroid hormone receptor functions as a ligand-operated developmental switch between proliferation and differentiation of erythroid progenitors.
Bauer A; Mikulits W; Lagger G; Stengl G; Brosch G; Beug H
EMBO J; 1998 Aug; 17(15):4291-303. PubMed ID: 9687498
[TBL] [Abstract][Full Text] [Related]
20. Unliganded T3R, but not its oncogenic variant, v-erbA, suppresses RAR-dependent transactivation by titrating out RXR.
Barettino D; Bugge TH; Bartunek P; Vivanco Ruiz MD; Sonntag-Buck V; Beug H; Zenke M; Stunnenberg HG
EMBO J; 1993 Apr; 12(4):1343-54. PubMed ID: 8096810
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
