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2. Elevated levels of wild-type p53 induced by radiolabeling of cells leads to apoptosis or sustained growth arrest. Yeargin J; Haas M Curr Biol; 1995 Apr; 5(4):423-31. PubMed ID: 7627557 [TBL] [Abstract][Full Text] [Related]
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5. Two-dimensional gel analysis of p53-mediated changes in protein expression. Maxwell SA Anticancer Res; 1994; 14(6B):2549-56. PubMed ID: 7872680 [TBL] [Abstract][Full Text] [Related]
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9. Apoptosis in erythroid progenitors deprived of erythropoietin occurs during the G1 and S phases of the cell cycle without growth arrest or stabilization of wild-type p53. Kelley LL; Green WF; Hicks GG; Bondurant MC; Koury MJ; Ruley HE Mol Cell Biol; 1994 Jun; 14(6):4183-92. PubMed ID: 8196656 [TBL] [Abstract][Full Text] [Related]
10. Participation of p53 protein in the cellular response to DNA damage. Kastan MB; Onyekwere O; Sidransky D; Vogelstein B; Craig RW Cancer Res; 1991 Dec; 51(23 Pt 1):6304-11. PubMed ID: 1933891 [TBL] [Abstract][Full Text] [Related]
11. Role of retinoblastoma gene product in p53-mediated DNA damage response. Smith ML; Zhan Q; Bae I; Fornace AJ Exp Cell Res; 1994 Dec; 215(2):386-9. PubMed ID: 7982477 [TBL] [Abstract][Full Text] [Related]
12. ES cells do not activate p53-dependent stress responses and undergo p53-independent apoptosis in response to DNA damage. Aladjem MI; Spike BT; Rodewald LW; Hope TJ; Klemm M; Jaenisch R; Wahl GM Curr Biol; 1998 Jan; 8(3):145-55. PubMed ID: 9443911 [TBL] [Abstract][Full Text] [Related]
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