BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

102 related articles for article (PubMed ID: 7996600)

  • 1. Macrophage suppression of granulocyte and macrophage growth following burn wound infection.
    Gamelli RL; He LK; Liu H
    J Trauma; 1994 Dec; 37(6):888-92. PubMed ID: 7996600
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Recombinant human granulocyte colony-stimulating factor treatment improves macrophage suppression of granulocyte and macrophage growth after burn and burn wound infection.
    Gamelli RL; He LK; Liu H
    J Trauma; 1995 Dec; 39(6):1141-6; discussion 1146-7. PubMed ID: 7500409
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Macrophage mediated suppression of granulocyte and macrophage growth after burn wound infection reversal by means of anti-PGE2.
    Gamelli RL; He LK; Liu LH
    J Burn Care Rehabil; 2000; 21(1 Pt 1):64-9. PubMed ID: 10661541
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Marrow granulocyte-macrophage progenitor cell response to burn injury as modified by endotoxin and indomethacin.
    Gamelli RL; He LK; Liu H
    J Trauma; 1994 Sep; 37(3):339-46. PubMed ID: 8083891
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Burn wound infection-induced myeloid suppression: the role of prostaglandin E2, elevated adenylate cyclase, and cyclic adenosine monophosphate.
    Gamelli RL; He LK; Liu H; Ricken JD
    J Trauma; 1998 Mar; 44(3):469-74. PubMed ID: 9529173
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Effect of burn injury on granulocyte and macrophage production.
    Gamelli RL; Hebert JC; Foster RS
    J Trauma; 1985 Jul; 25(7):615-9. PubMed ID: 4009767
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Mechanism of increased tumor necrosis factor production after thermal injury. Altered sensitivity to PGE2 and immunomodulation with indomethacin.
    Molloy RG; O'Riordain M; Holzheimer R; Nestor M; Collins K; Mannick JA; Rodrick ML
    J Immunol; 1993 Aug; 151(4):2142-9. PubMed ID: 8345198
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Macrophage activation by granulocyte/macrophage colony-stimulating factor. Priming for enhanced release of tumor necrosis factor-alpha and prostaglandin E2.
    Heidenreich S; Gong JH; Schmidt A; Nain M; Gemsa D
    J Immunol; 1989 Aug; 143(4):1198-205. PubMed ID: 2473121
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Bone marrow norepinephrine mediates development of functionally different macrophages after thermal injury and sepsis.
    Cohen MJ; Shankar R; Stevenson J; Fernandez R; Gamelli RL; Jones SB
    Ann Surg; 2004 Jul; 240(1):132-41. PubMed ID: 15213629
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Myelopoiesis in the infected burn.
    McEuen DD; Ogawa M; Eurenius K
    J Lab Clin Med; 1977 Mar; 89(3):540-3. PubMed ID: 839111
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Clonal analysis of progenitor cell commitment of granulocyte or macrophage production.
    Metcalf D; Burgess AW
    J Cell Physiol; 1982 Jun; 111(3):275-83. PubMed ID: 6980223
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Functional characterization of mouse granulocytes and macrophages produced in vitro from bone marrow progenitors stimulated with interleukin 3 (IL-3) or granulocyte-macrophage colony-stimulating factor (GM-CSF).
    Bender JG; Unverzagt KL; Maples PB; Mehrotra Y; Mellon J; Van Epps DE; Stewart CC
    Exp Hematol; 1992 Oct; 20(9):1135-40. PubMed ID: 1361455
    [TBL] [Abstract][Full Text] [Related]  

  • 13. IL-4 promotes macrophage development by rapidly stimulating lineage restriction of bipotent granulocyte-macrophage colony-forming cells.
    Nicholls SE; Heyworth CM; Dexter TM; Lord JM; Johnson GD; Whetton AD
    J Immunol; 1995 Jul; 155(2):845-53. PubMed ID: 7608562
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Tumor growth changes the contribution of granulocyte-macrophage colony-stimulating factor during macrophage-mediated suppression of allorecognition.
    Walker TM; Yurochko AD; Burger CJ; Elgert KD
    Immunobiology; 1992 Sep; 185(5):427-39. PubMed ID: 1452214
    [TBL] [Abstract][Full Text] [Related]  

  • 15. The role of NO in macrophage dysfunction at early stage after burn injury.
    Luo G; Peng D; Zheng J; Chen X; Wu J; Elster E; Tadaki D
    Burns; 2005 Mar; 31(2):138-44. PubMed ID: 15683683
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Severity of burn injury and sepsis determines the cytokine responses of bone marrow progenitor-derived macrophages.
    Cohen MJ; Carroll C; He LK; Muthu K; Gamelli RL; Jones SB; Shankar R
    J Trauma; 2007 Apr; 62(4):858-67. PubMed ID: 17426540
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Interleukin 2 inhibits in vitro granulocyte-macrophage colony formation.
    Naldini A; Fleischmann WR; Ballas ZK; Klimpel KD; Klimpel GR
    J Immunol; 1987 Sep; 139(6):1880-4. PubMed ID: 2442258
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Blockade of prostaglandin biosynthesis in intact mice dramatically augments the expansion of committed myeloid progenitor cells (colony-forming units-granulocyte, macrophage) after acute administration of recombinant human IL-1 alpha.
    Pelus LM
    J Immunol; 1989 Dec; 143(12):4171-9. PubMed ID: 2592770
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Association between colony forming units-granulocyte macrophage expression of Ia-like (HLA-DR) antigen and control of granulocyte and macrophage production. A new role for prostaglandin E.
    Pelus LM
    J Clin Invest; 1982 Sep; 70(3):568-78. PubMed ID: 6286727
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Enhanced expression of cyclooxygenase-2 and prostaglandin E2 in response to endotoxin after trauma is dependent on MAPK and NF-kappaB mechanisms.
    Yan Z; Stapleton PP; Freeman TA; Fuortes M; Daly JM
    Cell Immunol; 2004; 232(1-2):116-26. PubMed ID: 15890324
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 6.