218 related articles for article (PubMed ID: 8063382)
1. Site-specific immunity to Leishmania major in SWR mice: the site of infection influences susceptibility and expression of the antileishmanial immune response.
Nabors GS; Farrell JP
Infect Immun; 1994 Sep; 62(9):3655-62. PubMed ID: 8063382
[TBL] [Abstract][Full Text] [Related]
2. The influence of the site of parasite inoculation on the development of Th1 and Th2 type immune responses in (BALB/c x C57BL/6) F1 mice infected with Leishmania major.
Nabors GS; Nolan T; Croop W; Li J; Farrell JP
Parasite Immunol; 1995 Nov; 17(11):569-79. PubMed ID: 8817603
[TBL] [Abstract][Full Text] [Related]
3. Early IL-4 production does not predict susceptibility to Leishmania major.
Scott P; Eaton A; Gause WC; di Zhou X; Hondowicz B
Exp Parasitol; 1996 Nov; 84(2):178-87. PubMed ID: 8932767
[TBL] [Abstract][Full Text] [Related]
4. Depletion of interleukin-4 in BALB/c mice with established Leishmania major infections increases the efficacy of antimony therapy and promotes Th1-like responses.
Nabors GS; Farrell JP
Infect Immun; 1994 Dec; 62(12):5498-504. PubMed ID: 7960131
[TBL] [Abstract][Full Text] [Related]
5. Leishmania major: a clone with low virulence for BALB/c mice elicits a Th1 type response and protects against infection with a highly virulent clone.
Li J; Nolan TJ; Farrell JP
Exp Parasitol; 1997 Sep; 87(1):47-57. PubMed ID: 9287957
[TBL] [Abstract][Full Text] [Related]
6. Pregnancy impairs resistance of C57BL/6 mice to Leishmania major infection and causes decreased antigen-specific IFN-gamma response and increased production of T helper 2 cytokines.
Krishnan L; Guilbert LJ; Russell AS; Wegmann TG; Mosmann TR; Belosevic M
J Immunol; 1996 Jan; 156(2):644-52. PubMed ID: 8543816
[TBL] [Abstract][Full Text] [Related]
7. Nonhealing infection despite Th1 polarization produced by a strain of Leishmania major in C57BL/6 mice.
Anderson CF; Mendez S; Sacks DL
J Immunol; 2005 Mar; 174(5):2934-41. PubMed ID: 15728505
[TBL] [Abstract][Full Text] [Related]
8. Genetically resistant mice lacking interleukin-12 are susceptible to infection with Leishmania major and mount a polarized Th2 cell response.
Mattner F; Magram J; Ferrante J; Launois P; Di Padova K; Behin R; Gately MK; Louis JA; Alber G
Eur J Immunol; 1996 Jul; 26(7):1553-9. PubMed ID: 8766560
[TBL] [Abstract][Full Text] [Related]
9. Susceptibility to Leishmania major infection in interleukin-4-deficient mice.
Noben-Trauth N; Kropf P; Müller I
Science; 1996 Feb; 271(5251):987-90. PubMed ID: 8584936
[TBL] [Abstract][Full Text] [Related]
10. Genetically resistant mice lacking MyD88-adapter protein display a high susceptibility to Leishmania major infection associated with a polarized Th2 response.
Muraille E; De Trez C; Brait M; De Baetselier P; Leo O; Carlier Y
J Immunol; 2003 Apr; 170(8):4237-41. PubMed ID: 12682257
[TBL] [Abstract][Full Text] [Related]
11. CD28 costimulation and parasite dose combine to influence the susceptibility of BALB/c mice to infection with Leishmania major.
Compton HL; Farrell JP
J Immunol; 2002 Feb; 168(3):1302-8. PubMed ID: 11801669
[TBL] [Abstract][Full Text] [Related]
12. Differential regulation of IL-9-expression after infection with Leishmania major in susceptible and resistant mice.
Gessner A; Blum H; Röllinghoff M
Immunobiology; 1993 Dec; 189(5):419-35. PubMed ID: 8125519
[TBL] [Abstract][Full Text] [Related]
13. An immunomodulatory function for neutrophils during the induction of a CD4+ Th2 response in BALB/c mice infected with Leishmania major.
Tacchini-Cottier F; Zweifel C; Belkaid Y; Mukankundiye C; Vasei M; Launois P; Milon G; Louis JA
J Immunol; 2000 Sep; 165(5):2628-36. PubMed ID: 10946291
[TBL] [Abstract][Full Text] [Related]
14. Interleukin-4-independent acceleration of cutaneous leishmaniasis in susceptible BALB/c mice following treatment with anti-CTLA4 antibody.
Heinzel FP; Maier RA
Infect Immun; 1999 Dec; 67(12):6454-60. PubMed ID: 10569763
[TBL] [Abstract][Full Text] [Related]
15. Distinct immunological states in murine cutaneous leishmaniasis by immunising with different amounts of antigen: the generation of beneficial, potentially harmful, harmful and potentially extremely harmful states.
Bretscher PA; Ogunremi O; Menon JN
Behring Inst Mitt; 1997 Feb; (98):153-9. PubMed ID: 9382736
[TBL] [Abstract][Full Text] [Related]
16. Transgenic expression of CXCR3 on T cells enhances susceptibility to cutaneous Leishmania major infection by inhibiting monocyte maturation and promoting a Th2 response.
Oghumu S; Stock JC; Varikuti S; Dong R; Terrazas C; Edwards JA; Rappleye CA; Holovatyk A; Sharpe A; Satoskar AR
Infect Immun; 2015 Jan; 83(1):67-76. PubMed ID: 25312956
[TBL] [Abstract][Full Text] [Related]
17. IL-12 is required for natural killer cell activation and subsequent T helper 1 cell development in experimental leishmaniasis.
Scharton-Kersten T; Afonso LC; Wysocka M; Trinchieri G; Scott P
J Immunol; 1995 May; 154(10):5320-30. PubMed ID: 7730635
[TBL] [Abstract][Full Text] [Related]
18. IL-12Rbeta2-deficient mice of a genetically resistant background are susceptible to Leishmania major infection and develop a parasite-specific Th2 immune response.
Chakir H; Campos-Neto A; Mojibian M; Webb JR
Microbes Infect; 2003 Apr; 5(4):241-9. PubMed ID: 12706437
[TBL] [Abstract][Full Text] [Related]
19. Anti-leishmania effector functions of CD4+ Th1 cells and early events instructing Th2 cell development and susceptibility to Leishmania major in BALB/c mice.
Louis JA; Conceiçao-Silva F; Himmelrich H; Tacchini-Cottier F; Launois P
Adv Exp Med Biol; 1998; 452():53-60. PubMed ID: 9889959
[TBL] [Abstract][Full Text] [Related]
20. Deletion of Interleukin-4 Receptor Alpha-Responsive Keratinocytes in BALB/c Mice Does Not Alter Susceptibility to Cutaneous Leishmaniasis.
Govender M; Hurdayal R; Martinez-Salazar B; Gqada K; Pillay S; Gcanga L; Passelli K; Nieuwenhuizen NE; Tacchini-Cottier F; Guler R; Brombacher F
Infect Immun; 2018 Dec; 86(12):. PubMed ID: 30275010
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
