174 related articles for article (PubMed ID: 8102105)
1. ErbA: tumor suppressor turned oncogene?
Damm K
FASEB J; 1993 Jul; 7(10):904-9. PubMed ID: 8102105
[TBL] [Abstract][Full Text] [Related]
2. Modulation of normal erythroid differentiation by the endogenous thyroid hormone and retinoic acid receptors: a possible target for v-erbA oncogene action.
Schroeder C; Gibson L; Zenke M; Beug H
Oncogene; 1992 Feb; 7(2):217-27. PubMed ID: 1347914
[TBL] [Abstract][Full Text] [Related]
3. The v-erbA oncogene requires cooperation with tyrosine kinases to arrest erythroid differentiation induced by ligand-activated endogenous c-erbA and retinoic acid receptor.
Schroeder C; Gibson L; Beug H
Oncogene; 1992 Feb; 7(2):203-16. PubMed ID: 1347913
[TBL] [Abstract][Full Text] [Related]
4. V-erbA requires auxiliary proteins for dominant negative activity.
Hermann T; Hoffmann B; Piedrafita FJ; Zhang XK; Pfahl M
Oncogene; 1993 Jan; 8(1):55-65. PubMed ID: 8093812
[TBL] [Abstract][Full Text] [Related]
5. Thyroid hormone receptor/and v-erbA. A single amino acid difference in the C-terminal region influences dominant negative activity and receptor dimer formation.
Selmi S; Samuels HH
J Biol Chem; 1991 Jun; 266(18):11589-93. PubMed ID: 1675637
[TBL] [Abstract][Full Text] [Related]
6. Myb-Ets fusion oncoprotein inhibits thyroid hormone receptor/c-ErbA and retinoic acid receptor functions: a novel mechanism of action for leukemogenic transformation by E26 avian retrovirus.
Rascle A; Ferrand N; Gandrillon O; Samarut J
Mol Cell Biol; 1996 Nov; 16(11):6338-51. PubMed ID: 8887663
[TBL] [Abstract][Full Text] [Related]
7. Functional domains of the v-erbA protein necessary for oncogenesis are required for transcriptional activation in Saccharomyces cerevisiae.
Smit-McBride Z; Privalsky ML
Oncogene; 1993 Jun; 8(6):1465-75. PubMed ID: 8099219
[TBL] [Abstract][Full Text] [Related]
8. Identification of a domain required for oncogenic activity and transcriptional suppression by v-erbA and thyroid-hormone receptor alpha.
Damm K; Evans RM
Proc Natl Acad Sci U S A; 1993 Nov; 90(22):10668-72. PubMed ID: 7902566
[TBL] [Abstract][Full Text] [Related]
9. V-erbA and c-erbA proteins enhance transcriptional activation by c-jun.
Sharif M; Privalsky ML
Oncogene; 1992 May; 7(5):953-60. PubMed ID: 1349165
[TBL] [Abstract][Full Text] [Related]
10. v-erbA oncogene activation entails the loss of hormone-dependent regulator activity of c-erbA.
Zenke M; Muñoz A; Sap J; Vennström B; Beug H
Cell; 1990 Jun; 61(6):1035-49. PubMed ID: 1972036
[TBL] [Abstract][Full Text] [Related]
11. A novel mechanism of action for v-ErbA: abrogation of the inactivation of transcription factor AP-1 by retinoic acid and thyroid hormone receptors.
Desbois C; Aubert D; Legrand C; Pain B; Samarut J
Cell; 1991 Nov; 67(4):731-40. PubMed ID: 1682056
[TBL] [Abstract][Full Text] [Related]
12. A conserved C-terminal sequence that is deleted in v-ErbA is essential for the biological activities of c-ErbA (the thyroid hormone receptor).
Saatcioglu F; Bartunek P; Deng T; Zenke M; Karin M
Mol Cell Biol; 1993 Jun; 13(6):3675-85. PubMed ID: 8098843
[TBL] [Abstract][Full Text] [Related]
13. c-erbA: protooncogene or growth suppressor gene?
Damm K
Adv Cancer Res; 1992; 59():89-113. PubMed ID: 1355627
[No Abstract] [Full Text] [Related]
14. v-erbA cooperates with bFGF in neuroretina cell transformation.
Garrido C; Li RP; Samarut J; Gospodarowicz D; Saule S
Virology; 1993 Feb; 192(2):578-86. PubMed ID: 7678474
[TBL] [Abstract][Full Text] [Related]
15. v-erbA acts on retinoic acid receptors in immature avian erythroid cells.
Sande S; Sharif M; Chen H; Privalsky M
J Virol; 1993 Feb; 67(2):1067-74. PubMed ID: 8093487
[TBL] [Abstract][Full Text] [Related]
16. Unliganded T3R, but not its oncogenic variant, v-erbA, suppresses RAR-dependent transactivation by titrating out RXR.
Barettino D; Bugge TH; Bartunek P; Vivanco Ruiz MD; Sonntag-Buck V; Beug H; Zenke M; Stunnenberg HG
EMBO J; 1993 Apr; 12(4):1343-54. PubMed ID: 8096810
[TBL] [Abstract][Full Text] [Related]
17. v-erbA oncogene function in neoplasia correlates with its ability to repress retinoic acid receptor action.
Sharif M; Privalsky ML
Cell; 1991 Sep; 66(5):885-93. PubMed ID: 1679679
[TBL] [Abstract][Full Text] [Related]
18. Phosphorylation of the v-erbA protein is required for its function as an oncogene.
Glineur C; Zenke M; Beug H; Ghysdael J
Genes Dev; 1990 Oct; 4(10):1663-76. PubMed ID: 1979040
[TBL] [Abstract][Full Text] [Related]
19. The lack of transcriptional activation of the v-erbA oncogene is in part due to a mutation present in the DNA binding domain of the protein.
de Verneuil H; Metzger D
Nucleic Acids Res; 1990 Aug; 18(15):4489-97. PubMed ID: 1975094
[TBL] [Abstract][Full Text] [Related]
20. The v-erbA oncogene is a thyroid hormone receptor antagonist.
Evans RM
Int J Cancer Suppl; 1989; 4():26-8. PubMed ID: 2572567
[No Abstract] [Full Text] [Related]
[Next] [New Search]
