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2. Cyclin D1 is dispensable for G1 control in retinoblastoma gene-deficient cells independently of cdk4 activity. Lukas J; Bartkova J; Rohde M; Strauss M; Bartek J Mol Cell Biol; 1995 May; 15(5):2600-11. PubMed ID: 7739541 [TBL] [Abstract][Full Text] [Related]
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8. Induction of cell cycle regulatory proteins in anti-immunoglobulin-stimulated mature B lymphocytes. Solvason N; Wu WW; Kabra N; Wu X; Lees E; Howard MC J Exp Med; 1996 Aug; 184(2):407-17. PubMed ID: 8760794 [TBL] [Abstract][Full Text] [Related]
9. Identification of G1 kinase activity for cdk6, a novel cyclin D partner. Meyerson M; Harlow E Mol Cell Biol; 1994 Mar; 14(3):2077-86. PubMed ID: 8114739 [TBL] [Abstract][Full Text] [Related]
10. Expression of cyclin E renders cyclin D-CDK4 dispensable for inactivation of the retinoblastoma tumor suppressor protein, activation of E2F, and G1-S phase progression. Keenan SM; Lents NH; Baldassare JJ J Biol Chem; 2004 Feb; 279(7):5387-96. PubMed ID: 14645251 [TBL] [Abstract][Full Text] [Related]
11. Mutual requirement of CDK4 and Myc in malignant transformation: evidence for cyclin D1/CDK4 and p16INK4A as upstream regulators of Myc. Haas K; Staller P; Geisen C; Bartek J; Eilers M; Möröy T Oncogene; 1997 Jul; 15(2):179-92. PubMed ID: 9244353 [TBL] [Abstract][Full Text] [Related]
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