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23. Introduction of a loss-of-function point mutation from the SH3 region of the Caenorhabditis elegans sem-5 gene activates the transforming ability of c-abl in vivo and abolishes binding of proline-rich ligands in vitro. Van Etten RA; Debnath J; Zhou H; Casasnovas JM Oncogene; 1995 May; 10(10):1977-88. PubMed ID: 7539119 [TBL] [Abstract][Full Text] [Related]
24. v-Rel and c-Rel are differentially affected by mutations at a consensus protein kinase recognition sequence. Mosialos G; Gilmore TD Oncogene; 1993 Mar; 8(3):721-30. PubMed ID: 8437855 [TBL] [Abstract][Full Text] [Related]
25. Expression of a novel isoform of Vav, Vav-T, containing a single Src homology 3 domain in murine testicular germ cells. Okumura K; Kaneko Y; Nonoguchi K; Nishiyama H; Yokoi H; Higuchi T; Itoh K; Yoshida O; Miki T; Fujita J Oncogene; 1997 Feb; 14(6):713-20. PubMed ID: 9038379 [TBL] [Abstract][Full Text] [Related]
26. The amino-terminal Src homology 2 domain of phospholipase C gamma 1 is essential for TCR-induced tyrosine phosphorylation of phospholipase C gamma 1. Stoica B; DeBell KE; Graham L; Rellahan BL; Alava MA; Laborda J; Bonvini E J Immunol; 1998 Feb; 160(3):1059-66. PubMed ID: 9570517 [TBL] [Abstract][Full Text] [Related]
27. The amino-terminal region of pp60c-src has a modulatory role and contains multiple sites of tyrosine phosphorylation. Espino PC; Harvey R; Schweickhardt RL; White GA; Smith AE; Cheng SH Oncogene; 1990 Mar; 5(3):283-93. PubMed ID: 1690377 [TBL] [Abstract][Full Text] [Related]
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29. Mutational analysis of the regulatory function of the c-Abl Src homology 3 domain. Brasher BB; Roumiantsev S; Van Etten RA Oncogene; 2001 Nov; 20(53):7744-52. PubMed ID: 11753652 [TBL] [Abstract][Full Text] [Related]
30. The SH3 and SH2 domains are capable of directing specificity in protein interactions between the non-receptor tyrosine kinases cSrc and cYes. Summy JM; Guappone AC; Sudol M; Flynn DC Oncogene; 2000 Jan; 19(1):155-60. PubMed ID: 10644991 [TBL] [Abstract][Full Text] [Related]
31. Transformation suppressor activity of C3G is independent of its CDC25-homology domain. Guerrero C; Fernandez-Medarde A; Rojas JM; Font de Mora J; Esteban LM; Santos E Oncogene; 1998 Feb; 16(5):613-24. PubMed ID: 9482107 [TBL] [Abstract][Full Text] [Related]
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34. Analysis of structure and activation of some receptor-type tyrosine kinase oncogenes. Shibuya M; Matsushime H; Yamazaki H; Wang LH; Fukui Y; Ueyama Y; Tamaoki N Princess Takamatsu Symp; 1986; 17():195-202. PubMed ID: 3455414 [TBL] [Abstract][Full Text] [Related]
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39. Highly conserved amino acids in the SH2 and catalytic domains of v-src are altered in naturally occurring, transformation-defective alleles. Verderame MF; Varmus HE Oncogene; 1994 Jan; 9(1):175-82. PubMed ID: 8302576 [TBL] [Abstract][Full Text] [Related]
40. Defective antigen receptor-mediated proliferation of B and T cells in the absence of Vav. Tarakhovsky A; Turner M; Schaal S; Mee PJ; Duddy LP; Rajewsky K; Tybulewicz VL Nature; 1995 Mar; 374(6521):467-70. PubMed ID: 7700358 [TBL] [Abstract][Full Text] [Related] [Previous] [Next] [New Search]