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2. Mechanisms of Host IFI16, PML, and Daxx Protein Restriction of Herpes Simplex Virus 1 Replication. Merkl PE; Orzalli MH; Knipe DM J Virol; 2018 May; 92(10):. PubMed ID: 29491153 [TBL] [Abstract][Full Text] [Related]
3. ND10 protein PML is recruited to herpes simplex virus type 1 prereplicative sites and replication compartments in the presence of viral DNA polymerase. Burkham J; Coen DM; Weller SK J Virol; 1998 Dec; 72(12):10100-7. PubMed ID: 9811750 [TBL] [Abstract][Full Text] [Related]
4. ND10 components relocate to sites associated with herpes simplex virus type 1 nucleoprotein complexes during virus infection. Everett RD; Murray J J Virol; 2005 Apr; 79(8):5078-89. PubMed ID: 15795293 [TBL] [Abstract][Full Text] [Related]
5. Modification of discrete nuclear domains induced by herpes simplex virus type 1 immediate early gene 1 product (ICP0). Maul GG; Guldner HH; Spivack JG J Gen Virol; 1993 Dec; 74 ( Pt 12)():2679-90. PubMed ID: 8277273 [TBL] [Abstract][Full Text] [Related]
6. Identification of three redundant segments responsible for herpes simplex virus 1 ICP0 to fuse with ND10 nuclear bodies. Zheng Y; Gu H J Virol; 2015 Apr; 89(8):4214-26. PubMed ID: 25631093 [TBL] [Abstract][Full Text] [Related]
7. Overexpression of promyelocytic leukemia protein precludes the dispersal of ND10 structures and has no effect on accumulation of infectious herpes simplex virus 1 or its proteins. Lopez P; Jacob RJ; Roizman B J Virol; 2002 Sep; 76(18):9355-67. PubMed ID: 12186918 [TBL] [Abstract][Full Text] [Related]
8. The nuclear location of PML, a cellular member of the C3HC4 zinc-binding domain protein family, is rearranged during herpes simplex virus infection by the C3HC4 viral protein ICP0. Maul GG; Everett RD J Gen Virol; 1994 Jun; 75 ( Pt 6)():1223-33. PubMed ID: 8207389 [TBL] [Abstract][Full Text] [Related]
9. The periphery of nuclear domain 10 (ND10) as site of DNA virus deposition. Ishov AM; Maul GG J Cell Biol; 1996 Aug; 134(4):815-26. PubMed ID: 8769408 [TBL] [Abstract][Full Text] [Related]
10. Formation of nuclear foci of the herpes simplex virus type 1 regulatory protein ICP4 at early times of infection: localization, dynamics, recruitment of ICP27, and evidence for the de novo induction of ND10-like complexes. Everett RD; Sourvinos G; Leiper C; Clements JB; Orr A J Virol; 2004 Feb; 78(4):1903-17. PubMed ID: 14747555 [TBL] [Abstract][Full Text] [Related]
11. Components of promyelocytic leukemia nuclear bodies (ND10) act cooperatively to repress herpesvirus infection. Glass M; Everett RD J Virol; 2013 Feb; 87(4):2174-85. PubMed ID: 23221561 [TBL] [Abstract][Full Text] [Related]
12. Recruitment of human cytomegalovirus immediate-early 2 protein onto parental viral genomes in association with ND10 in live-infected cells. Sourvinos G; Tavalai N; Berndt A; Spandidos DA; Stamminger T J Virol; 2007 Sep; 81(18):10123-36. PubMed ID: 17626080 [TBL] [Abstract][Full Text] [Related]
13. Two overlapping regions within the N-terminal half of the herpes simplex virus 1 E3 ubiquitin ligase ICP0 facilitate the degradation and dissociation of PML and dissociation of Sp100 from ND10. Perusina Lanfranca M; Mostafa HH; Davido DJ J Virol; 2013 Dec; 87(24):13287-96. PubMed ID: 24089549 [TBL] [Abstract][Full Text] [Related]
14. Recruitment of herpes simplex virus type 1 transcriptional regulatory protein ICP4 into foci juxtaposed to ND10 in live, infected cells. Everett RD; Sourvinos G; Orr A J Virol; 2003 Mar; 77(6):3680-9. PubMed ID: 12610143 [TBL] [Abstract][Full Text] [Related]
15. Evidence for a role of the cellular ND10 protein PML in mediating intrinsic immunity against human cytomegalovirus infections. Tavalai N; Papior P; Rechter S; Leis M; Stamminger T J Virol; 2006 Aug; 80(16):8006-18. PubMed ID: 16873257 [TBL] [Abstract][Full Text] [Related]
16. PML contributes to a cellular mechanism of repression of herpes simplex virus type 1 infection that is inactivated by ICP0. Everett RD; Rechter S; Papior P; Tavalai N; Stamminger T; Orr A J Virol; 2006 Aug; 80(16):7995-8005. PubMed ID: 16873256 [TBL] [Abstract][Full Text] [Related]
17. Replication of ICP0-null mutant herpes simplex virus type 1 is restricted by both PML and Sp100. Everett RD; Parada C; Gripon P; Sirma H; Orr A J Virol; 2008 Mar; 82(6):2661-72. PubMed ID: 18160441 [TBL] [Abstract][Full Text] [Related]
18. Stimulation of the Replication of ICP0-Null Mutant Herpes Simplex Virus 1 and pp71-Deficient Human Cytomegalovirus by Epstein-Barr Virus Tegument Protein BNRF1. Lu Y; Orr A; Everett RD J Virol; 2016 Nov; 90(21):9664-9673. PubMed ID: 27535048 [TBL] [Abstract][Full Text] [Related]
19. Effect of SUMO-SIM Interaction on the ICP0-Mediated Degradation of PML Isoform II and Its Associated Proteins in Herpes Simplex Virus 1 Infection. Jan Fada B; Kaadi E; Samrat SK; Zheng Y; Gu H J Virol; 2020 Jun; 94(12):. PubMed ID: 32295906 [TBL] [Abstract][Full Text] [Related]