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4. Induced p21WAF1 expression acts to reverse myc myelomonocytic cell transformation. Dolnikov A; Millington M; Sun LQ; Symonds G Cancer Gene Ther; 2000 Nov; 7(11):1491-503. PubMed ID: 11129291 [TBL] [Abstract][Full Text] [Related]
5. Transformation suppressor activity of C3G is independent of its CDC25-homology domain. Guerrero C; Fernandez-Medarde A; Rojas JM; Font de Mora J; Esteban LM; Santos E Oncogene; 1998 Feb; 16(5):613-24. PubMed ID: 9482107 [TBL] [Abstract][Full Text] [Related]
6. BPV-4 E8 transforms NIH3T3 cells, up-regulates cyclin A and cyclin A-associated kinase activity and de-regulates expression of the cdk inhibitor p27Kip1. O'Brien V; Campo MS Oncogene; 1998 Jul; 17(3):293-301. PubMed ID: 9690511 [TBL] [Abstract][Full Text] [Related]
7. Expression of an ATP binding mutant of PKC-delta inhibits Sis-induced transformation of NIH3T3 cells. Li W; Michieli P; Alimandi M; Lorenzi MV; Wu Y; Wang LH; Heidaran MA; Pierce JH Oncogene; 1996 Aug; 13(4):731-7. PubMed ID: 8761294 [TBL] [Abstract][Full Text] [Related]
8. Malolactomycin D, a potent inhibitor of transcription controlled by the Ras responsive element, inhibits Ras-mediated transformation activity with suppression of MMP-1 and MMP-9 in NIH3T3 cells. Futamura M; Kamiya S; Tsukamoto M; Hirano A; Monden Y; Arakawa H; Nishimura S Oncogene; 2001 Oct; 20(46):6724-30. PubMed ID: 11709707 [TBL] [Abstract][Full Text] [Related]
9. Both Sp1 and Sp3 are responsible for p21waf1 promoter activity induced by histone deacetylase inhibitor in NIH3T3 cells. Xiao H; Hasegawa T; Isobe K J Cell Biochem; 1999 Jun; 73(3):291-302. PubMed ID: 10321829 [TBL] [Abstract][Full Text] [Related]
10. Induction of p21WAF1/CIP1 and cyclin D1 expression by the Src oncoprotein in mouse fibroblasts: role of activated STAT3 signaling. Sinibaldi D; Wharton W; Turkson J; Bowman T; Pledger WJ; Jove R Oncogene; 2000 Nov; 19(48):5419-27. PubMed ID: 11114718 [TBL] [Abstract][Full Text] [Related]
12. C3G-mediated suppression of oncogene-induced focus formation in fibroblasts involves inhibition of ERK activation, cyclin A expression and alterations of anchorage-independent growth. Guerrero C; Martín-Encabo S; Fernández-Medarde A; Santos E Oncogene; 2004 Jun; 23(28):4885-93. PubMed ID: 15077165 [TBL] [Abstract][Full Text] [Related]
13. K-ras modulates the cell cycle via both positive and negative regulatory pathways. Fan J; Bertino JR Oncogene; 1997 May; 14(21):2595-607. PubMed ID: 9191059 [TBL] [Abstract][Full Text] [Related]
14. H-ras and raf-1 cooperate in transformation of NIH3T3 fibroblasts. Cuadrado A; Bruder JT; Heidaran MA; App H; Rapp UR; Aaronson SA Oncogene; 1993 Sep; 8(9):2443-8. PubMed ID: 8361757 [TBL] [Abstract][Full Text] [Related]
15. Selective loss of endogenous p21waf1/cip1 induction underlies the G1 checkpoint defect of monomeric p53 proteins. Tarunina M; Grimaldi M; Ruaro E; Pavlenko M; Schneider C; Jenkins JR Oncogene; 1996 Aug; 13(3):589-98. PubMed ID: 8760300 [TBL] [Abstract][Full Text] [Related]
16. The differentiation primary response gene MyD118, related to GADD45, encodes for a nuclear protein which interacts with PCNA and p21WAF1/CIP1. Vairapandi M; Balliet AG; Fornace AJ; Hoffman B; Liebermann DA Oncogene; 1996 Jun; 12(12):2579-94. PubMed ID: 8700517 [TBL] [Abstract][Full Text] [Related]
17. p53 expression overcomes p21WAF1/CIP1-mediated G1 arrest and induces apoptosis in human cancer cells. Kagawa S; Fujiwara T; Hizuta A; Yasuda T; Zhang WW; Roth JA; Tanaka N Oncogene; 1997 Oct; 15(16):1903-9. PubMed ID: 9365236 [TBL] [Abstract][Full Text] [Related]
18. Cell lines containing and expressing the human herpesvirus 6A ts gene are protected from both H-ras and BPV-1 transformation. Araujo JC; Doniger J; Stöppler H; Sadaie MR; Rosenthal LJ Oncogene; 1997 Feb; 14(8):937-43. PubMed ID: 9050993 [TBL] [Abstract][Full Text] [Related]
19. Resistance to apoptosis induced by alkylating agents in v-Ha-ras-transformed cells due to defect in p53 function. Kuo ML; Chou YW; Chau YP; Huang TS Mol Carcinog; 1997 Apr; 18(4):221-31. PubMed ID: 9142217 [TBL] [Abstract][Full Text] [Related]
20. Ras-interacting domain of Ral GDP dissociation stimulator like (RGL) reverses v-Ras-induced transformation and Raf-1 activation in NIH3T3 cells. Okazaki M; Kishida S; Murai H; Hinoi T; Kikuchi A Cancer Res; 1996 May; 56(10):2387-92. PubMed ID: 8625316 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]