BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

390 related articles for article (PubMed ID: 8649810)

  • 21. Cell-cycle progression is not essential for c-Myc to block differentiation.
    Ryan KM; Birnie GD
    Oncogene; 1997 Jun; 14(23):2835-43. PubMed ID: 9190900
    [TBL] [Abstract][Full Text] [Related]  

  • 22. Repression of transcription of the p27(Kip1) cyclin-dependent kinase inhibitor gene by c-Myc.
    Yang W; Shen J; Wu M; Arsura M; FitzGerald M; Suldan Z; Kim DW; Hofmann CS; Pianetti S; Romieu-Mourez R; Freedman LP; Sonenshein GE
    Oncogene; 2001 Mar; 20(14):1688-702. PubMed ID: 11313917
    [TBL] [Abstract][Full Text] [Related]  

  • 23. Sequential expression of the MAD family of transcriptional repressors during differentiation and development.
    Quéva C; Hurlin PJ; Foley KP; Eisenman RN
    Oncogene; 1998 Feb; 16(8):967-77. PubMed ID: 9519870
    [TBL] [Abstract][Full Text] [Related]  

  • 24. c-Myc does not require max for transcriptional activity in PC-12 cells.
    Ribon V; Leff T; Saltiel AR
    Mol Cell Neurosci; 1994 Jun; 5(3):277-82. PubMed ID: 8087425
    [TBL] [Abstract][Full Text] [Related]  

  • 25. Structure, function, and dynamics of the dimerization and DNA-binding domain of oncogenic transcription factor v-Myc.
    Fieber W; Schneider ML; Matt T; Kräutler B; Konrat R; Bister K
    J Mol Biol; 2001 Apr; 307(5):1395-410. PubMed ID: 11292350
    [TBL] [Abstract][Full Text] [Related]  

  • 26. Differential effects by Mad and Max on transformation by cellular and viral oncoproteins.
    Cerni C; Bousset K; Seelos C; Burkhardt H; Henriksson M; Lüscher B
    Oncogene; 1995 Aug; 11(3):587-96. PubMed ID: 7630643
    [TBL] [Abstract][Full Text] [Related]  

  • 27. Mapping of two genes encoding members of a distinct subfamily of MAX interacting proteins: MAD to human chromosome 2 and mouse chromosome 6, and MXI1 to human chromosome 10 and mouse chromosome 19.
    Edelhoff S; Ayer DE; Zervos AS; Steingrímsson E; Jenkins NA; Copeland NG; Eisenman RN; Brent R; Disteche CM
    Oncogene; 1994 Feb; 9(2):665-8. PubMed ID: 8290278
    [TBL] [Abstract][Full Text] [Related]  

  • 28. Decreased tumorigenicity of c-Myc-transformed fibroblasts expressing active USF2.
    Choe C; Chen N; Sawadogo M
    Exp Cell Res; 2005 Jan; 302(1):1-10. PubMed ID: 15541720
    [TBL] [Abstract][Full Text] [Related]  

  • 29. New Myc-interacting proteins: a second Myc network emerges.
    Sakamuro D; Prendergast GC
    Oncogene; 1999 May; 18(19):2942-54. PubMed ID: 10378691
    [TBL] [Abstract][Full Text] [Related]  

  • 30. Kinetics of myc-max-mad gene expression during hepatocyte proliferation in vivo: Differential regulation of mad family and stress-mediated induction of c-myc.
    Mauleon I; Lombard MN; Muñoz-Alonso MJ; Cañelles M; Leon J
    Mol Carcinog; 2004 Feb; 39(2):85-90. PubMed ID: 14750213
    [TBL] [Abstract][Full Text] [Related]  

  • 31. DNA binding by N- and L-Myc proteins.
    Ma A; Moroy T; Collum R; Weintraub H; Alt FW; Blackwell TK
    Oncogene; 1993 Apr; 8(4):1093-8. PubMed ID: 8455937
    [TBL] [Abstract][Full Text] [Related]  

  • 32. Expression of the hTERT gene is regulated at the level of transcriptional initiation and repressed by Mad1.
    Günes C; Lichtsteiner S; Vasserot AP; Englert C
    Cancer Res; 2000 Apr; 60(8):2116-21. PubMed ID: 10786671
    [TBL] [Abstract][Full Text] [Related]  

  • 33. Functional analysis of the carboxy-terminal transforming region of v-Myc: binding to Max is necessary, but not sufficient, for cellular transformation.
    Min S; Mascarenhas NT; Taparowsky EJ
    Oncogene; 1993 Oct; 8(10):2691-701. PubMed ID: 8378081
    [TBL] [Abstract][Full Text] [Related]  

  • 34. c-Myc creates an activation loop by transcriptionally repressing its own functional inhibitor, hMad4, in young fibroblasts, a loop lost in replicatively senescent fibroblasts.
    Marcotte R; Chen JM; Huard S; Wang E
    J Cell Biochem; 2005 Dec; 96(5):1071-85. PubMed ID: 16167342
    [TBL] [Abstract][Full Text] [Related]  

  • 35. Suppression of Myc, but not E1a, transformation activity by Max-associated proteins, Mad and Mxi1.
    Lahoz EG; Xu L; Schreiber-Agus N; DePinho RA
    Proc Natl Acad Sci U S A; 1994 Jun; 91(12):5503-7. PubMed ID: 8202517
    [TBL] [Abstract][Full Text] [Related]  

  • 36. Sin3 corepressor function in Myc-induced transcription and transformation.
    Harper SE; Qiu Y; Sharp PA
    Proc Natl Acad Sci U S A; 1996 Aug; 93(16):8536-40. PubMed ID: 8710905
    [TBL] [Abstract][Full Text] [Related]  

  • 37. Role for N-CoR and histone deacetylase in Sin3-mediated transcriptional repression.
    Alland L; Muhle R; Hou H; Potes J; Chin L; Schreiber-Agus N; DePinho RA
    Nature; 1997 May; 387(6628):49-55. PubMed ID: 9139821
    [TBL] [Abstract][Full Text] [Related]  

  • 38. The C. elegans MDL-1 and MXL-1 proteins can functionally substitute for vertebrate MAD and MAX.
    Yuan J; Tirabassi RS; Bush AB; Cole MD
    Oncogene; 1998 Sep; 17(9):1109-18. PubMed ID: 9764821
    [TBL] [Abstract][Full Text] [Related]  

  • 39. Both the helix-loop-helix and the leucine zipper motifs of c-Myc contribute to its dimerization specificity with Max.
    Davis LJ; Halazonetis TD
    Oncogene; 1993 Jan; 8(1):125-32. PubMed ID: 8423990
    [TBL] [Abstract][Full Text] [Related]  

  • 40. Differential effects of Mxi1-SRalpha and Mxi1-SRbeta in Myc antagonism.
    Dugast-Darzacq C; Grange T; Schreiber-Agus NB
    FEBS J; 2007 Sep; 274(17):4643-53. PubMed ID: 17697116
    [TBL] [Abstract][Full Text] [Related]  

    [Previous]   [Next]    [New Search]
    of 20.