119 related articles for article (PubMed ID: 8654784)
21. Investigations of non-NMDA receptor-induced toxicity in serum-free antioxidant-rich primary cultures of murine cerebellar granule cells.
Carroll FY; Cheung NS; Beart PM
Neurochem Int; 1998 Jul; 33(1):23-8. PubMed ID: 9694038
[TBL] [Abstract][Full Text] [Related]
22. Intracellular glutathione levels determine cerebellar granule neuron sensitivity to excitotoxic injury by kainic acid.
Ceccon M; Giusti P; Facci L; Borin G; Imbesi M; Floreani M; Skaper SD
Brain Res; 2000 Apr; 862(1-2):83-9. PubMed ID: 10799672
[TBL] [Abstract][Full Text] [Related]
23. Glycogen synthase kinase-3 inhibitors protect central neurons against excitotoxicity.
Facci L; Stevens DA; Skaper SD
Neuroreport; 2003 Aug; 14(11):1467-70. PubMed ID: 12960765
[TBL] [Abstract][Full Text] [Related]
24. Brevetoxins cause acute excitotoxicity in primary cultures of rat cerebellar granule neurons.
Berman FW; Murray TF
J Pharmacol Exp Ther; 1999 Jul; 290(1):439-44. PubMed ID: 10381810
[TBL] [Abstract][Full Text] [Related]
25. N-methyl-D-aspartate receptor agonists decrease protooncogene bcl-2 mRNA expression in cultured rat cerebellar granule neurons.
Montpied P; Weller M; Paul SM
Biochem Biophys Res Commun; 1993 Sep; 195(2):623-9. PubMed ID: 8103985
[TBL] [Abstract][Full Text] [Related]
26. Attenuation of excitatory amino acid toxicity by metabotropic glutamate receptor agonists and aniracetam in primary cultures of cerebellar granule cells.
Pizzi M; Fallacara C; Arrighi V; Memo M; Spano PF
J Neurochem; 1993 Aug; 61(2):683-9. PubMed ID: 8101561
[TBL] [Abstract][Full Text] [Related]
27. Hemoglobin potentiates excitotoxic injury in cortical cell culture.
Regan RF; Panter SS
J Neurotrauma; 1996 Apr; 13(4):223-31. PubMed ID: 8860203
[TBL] [Abstract][Full Text] [Related]
28. Inhibitory effect of fangchinoline on excitatory amino acids-induced neurotoxicity in cultured rat cerebellar granule cells.
Kim SD; Oh SK; Kim HS; Seong YH
Arch Pharm Res; 2001 Apr; 24(2):164-70. PubMed ID: 11339637
[TBL] [Abstract][Full Text] [Related]
29. Neuronal fodrin proteolysis occurs independently of excitatory amino acid-induced neurotoxicity.
Di Stasi AM; Gallo V; Ceccarini M; Petrucci TC
Neuron; 1991 Mar; 6(3):445-54. PubMed ID: 1848081
[TBL] [Abstract][Full Text] [Related]
30. Media composition modulates excitatory amino acid-induced death of rat cerebellar granule cells.
Wood AM; Tiwari P; Bristow DR
Hum Exp Toxicol; 1997 Jul; 16(7):350-5. PubMed ID: 9257158
[TBL] [Abstract][Full Text] [Related]
31. Medium-dependent dissociation of cytotoxic and GABA-releasing effects of N-methyl-D-aspartate (NMDA), alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionate (AMPA) and kainate in rat cortical cultures.
Erdö SL; Cai N; Lakics V
Neurosci Lett; 1993 Apr; 152(1-2):84-6. PubMed ID: 7685870
[TBL] [Abstract][Full Text] [Related]
32. Characterization of the excitoprotective actions of N-methyl-D-aspartate in cultured cerebellar granule neurons.
Damschroder-Williams P; Irwin RP; Lin SZ; Paul SM
J Neurochem; 1995 Sep; 65(3):1069-76. PubMed ID: 7643085
[TBL] [Abstract][Full Text] [Related]
33. Treatment with excitatory amino acids or high K+ and NMDA receptors in cerebellar granule cells.
Resink A; Boer GJ; Balázs R
Neuroreport; 1992 Sep; 3(9):757-60. PubMed ID: 1421132
[TBL] [Abstract][Full Text] [Related]
34. DL-threo-3-hydroxyaspartate reduces NMDA receptor activation by glutamate in cultured neurons.
Marini A; Novelli A
Eur J Pharmacol; 1991 Feb; 194(1):131-2. PubMed ID: 1676372
[No Abstract] [Full Text] [Related]
35. Metabotropic glutamate receptors and neuronal toxicity.
Aleppo G; Pisani A; Copani A; Bruno V; Aronica E; D'Agata V; Canonico PL; Nicoletti F
Adv Exp Med Biol; 1992; 318():137-45. PubMed ID: 1322025
[No Abstract] [Full Text] [Related]
36. Glufosinate ammonium stimulates nitric oxide production through N-methyl D-aspartate receptors in rat cerebellum.
Nakaki T; Mishima A; Suzuki E; Shintani F; Fujii T
Neurosci Lett; 2000 Sep; 290(3):209-12. PubMed ID: 10963900
[TBL] [Abstract][Full Text] [Related]
37. Domoic acid neurotoxicity in cultured cerebellar granule neurons is controlled preferentially by the NMDA receptor Ca(2+) influx pathway.
Berman FW; LePage KT; Murray TF
Brain Res; 2002 Jan; 924(1):20-9. PubMed ID: 11743991
[TBL] [Abstract][Full Text] [Related]
38. L-2-chloropropionic acid inhibits glutamate and aspartate release from rat cerebellar slices but does not activate cerebellar NMDA receptors: implications for L-2-chloropropionic acid-induced neurotoxicity.
Widdowson PS; Briggs I; BoSmith RE; Sturgess NC; Rosbottom A; Smith JC; Wyatt I
Neurotoxicology; 1997; 18(1):169-77. PubMed ID: 9215999
[TBL] [Abstract][Full Text] [Related]
39. Excitotoxic death induced by released glutamate in depolarized primary cultures of mouse cerebellar granule cells is dependent on GABAA receptors and niflumic acid-sensitive chloride channels.
Babot Z; Cristòfol R; Suñol C
Eur J Neurosci; 2005 Jan; 21(1):103-12. PubMed ID: 15654847
[TBL] [Abstract][Full Text] [Related]
40. N-methyl-D-aspartate receptor-mediated mitochondrial Ca(2+) overload in acute excitotoxic motor neuron death: a mechanism distinct from chronic neurotoxicity after Ca(2+) influx.
Urushitani M; Nakamizo T; Inoue R; Sawada H; Kihara T; Honda K; Akaike A; Shimohama S
J Neurosci Res; 2001 Mar; 63(5):377-87. PubMed ID: 11223912
[TBL] [Abstract][Full Text] [Related]
[Previous] [Next] [New Search]