152 related articles for article (PubMed ID: 8663212)
21. Inhibition of neurofibromin and p120 GTPase activating protein (GAP) by dietary fatty acids.
Lee JH; Harwalkar JA; Bryant SS; Sundaram V; Jove R; Golubic M
Adv Exp Med Biol; 1999; 469():391-8. PubMed ID: 10667358
[TBL] [Abstract][Full Text] [Related]
22. Mutations in the GAP-related domain impair the ability of neurofibromin to associate with microtubules.
Xu H; Gutmann DH
Brain Res; 1997 Jun; 759(1):149-52. PubMed ID: 9219873
[TBL] [Abstract][Full Text] [Related]
23. A C-terminal domain of GAP is sufficient to stimulate ras p21 GTPase activity.
Marshall MS; Hill WS; Ng AS; Vogel US; Schaber MD; Scolnick EM; Dixon RA; Sigal IS; Gibbs JB
EMBO J; 1989 Apr; 8(4):1105-10. PubMed ID: 2545441
[TBL] [Abstract][Full Text] [Related]
24. Identification of neurofibromatosis type I gene product as an insoluble GTPase-activating protein toward ras p21.
Hattori S; Maekawa M; Nakamura S
Oncogene; 1992 Mar; 7(3):481-5. PubMed ID: 1549362
[TBL] [Abstract][Full Text] [Related]
25. Identification of neurofibromin mutants that exhibit allele specificity or increased Ras affinity resulting in suppression of activated ras alleles.
Morcos P; Thapar N; Tusneem N; Stacey D; Tamanoi F
Mol Cell Biol; 1996 May; 16(5):2496-503. PubMed ID: 8628317
[TBL] [Abstract][Full Text] [Related]
26. Differential regulation of cellular activities by GTPase-activating protein and NF1.
al-Alawi N; Xu G; White R; Clark R; McCormick F; Feramisco JR
Mol Cell Biol; 1993 Apr; 13(4):2497-503. PubMed ID: 8455625
[TBL] [Abstract][Full Text] [Related]
27. The GAP-related domain of the neurofibromatosis type 1 gene product interacts with ras p21.
Martin GA; Viskochil D; Bollag G; McCabe PC; Crosier WJ; Haubruck H; Conroy L; Clark R; O'Connell P; Cawthon RM
Cell; 1990 Nov; 63(4):843-9. PubMed ID: 2121370
[TBL] [Abstract][Full Text] [Related]
28. Mutation-deletion analysis of a Ca(2+)-dependent phospholipid binding (CaLB) domain within p120 GAP, a GTPase-activating protein for p21 ras.
Gawler DJ; Zhang LJ; Moran MF
Biochem J; 1995 Apr; 307 ( Pt 2)(Pt 2):487-91. PubMed ID: 7733887
[TBL] [Abstract][Full Text] [Related]
29. Selective disactivation of neurofibromin GAP activity in neurofibromatosis type 1.
Klose A; Ahmadian MR; Schuelke M; Scheffzek K; Hoffmeyer S; Gewies A; Schmitz F; Kaufmann D; Peters H; Wittinghofer A; Nürnberg P
Hum Mol Genet; 1998 Aug; 7(8):1261-8. PubMed ID: 9668168
[TBL] [Abstract][Full Text] [Related]
30. Mutational and kinetic analyses of the GTPase-activating protein (GAP)-p21 interaction: the C-terminal domain of GAP is not sufficient for full activity.
Gideon P; John J; Frech M; Lautwein A; Clark R; Scheffler JE; Wittinghofer A
Mol Cell Biol; 1992 May; 12(5):2050-6. PubMed ID: 1569940
[TBL] [Abstract][Full Text] [Related]
31. The arginine finger loop of yeast and human GAP is a determinant for the specificity toward Ras GTPase.
te Biesebeke R; Krab IM; Parmeggiani A
Biochemistry; 2001 Jun; 40(25):7474-9. PubMed ID: 11412100
[TBL] [Abstract][Full Text] [Related]
32. Formation of a transition-state analog of the Ras GTPase reaction by Ras-GDP, tetrafluoroaluminate, and GTPase-activating proteins.
Mittal R; Ahmadian MR; Goody RS; Wittinghofer A
Science; 1996 Jul; 273(5271):115-7. PubMed ID: 8658179
[TBL] [Abstract][Full Text] [Related]
33. Ras-GTP regulation is not altered in cultured melanocytes with reduced levels of neurofibromin derived from patients with neurofibromatosis 1 (NF1).
Griesser J; Kaufmann D; Eisenbarth I; Bäuerle C; Krone W
Biol Chem Hoppe Seyler; 1995 Feb; 376(2):91-101. PubMed ID: 7794530
[TBL] [Abstract][Full Text] [Related]
34. Guanine-nucleotide binding activity, interaction with GTPase-activating protein and solution conformation of the human c-Ha-Ras protein catalytic domain are retained upon deletion of C-terminal 18 amino acid residues.
Fujita-Yoshigaki J; Ito Y; Yamasaki K; Muto Y; Miyazawa T; Nishimura S; Yokoyama S
J Protein Chem; 1992 Dec; 11(6):731-9. PubMed ID: 1466766
[TBL] [Abstract][Full Text] [Related]
35. Regulation of Ras-GAP and the neurofibromatosis-1 gene product by eicosanoids.
Han JW; McCormick F; Macara IG
Science; 1991 Apr; 252(5005):576-9. PubMed ID: 1902323
[TBL] [Abstract][Full Text] [Related]
36. p120 Ras GTPase-activating protein interacts with Ras-GTP through specific conserved residues.
Miao W; Eichelberger L; Baker L; Marshall MS
J Biol Chem; 1996 Jun; 271(26):15322-9. PubMed ID: 8663024
[TBL] [Abstract][Full Text] [Related]
37. The differential effects of the Gly-60 to Ala mutation on the interaction of H-Ras p21 with different downstream targets.
Hwang MC; Sung YJ; Hwang YW
J Biol Chem; 1996 Apr; 271(14):8196-202. PubMed ID: 8626511
[TBL] [Abstract][Full Text] [Related]
38. Crosslinking of the surface immunoglobulin receptor in B lymphocytes induces a redistribution of neurofibromin but not p120-GAP.
Boyer MJ; Gutmann DH; Collins FS; Bar-Sagi D
Oncogene; 1994 Feb; 9(2):349-57. PubMed ID: 8290249
[TBL] [Abstract][Full Text] [Related]
39. Characterization of full-length neurofibromin: tubulin inhibits Ras GAP activity.
Bollag G; McCormick F; Clark R
EMBO J; 1993 May; 12(5):1923-7. PubMed ID: 8491185
[TBL] [Abstract][Full Text] [Related]
40. Determinants of Ras proteins specifying the sensitivity to yeast Ira2p and human p120-GAP.
Parrini MC; Bernardi A; Parmeggiani A
EMBO J; 1996 Mar; 15(5):1107-11. PubMed ID: 8605880
[TBL] [Abstract][Full Text] [Related]
[Previous] [Next] [New Search]
