206 related articles for article (PubMed ID: 8761294)
1. Expression of an ATP binding mutant of PKC-delta inhibits Sis-induced transformation of NIH3T3 cells.
Li W; Michieli P; Alimandi M; Lorenzi MV; Wu Y; Wang LH; Heidaran MA; Pierce JH
Oncogene; 1996 Aug; 13(4):731-7. PubMed ID: 8761294
[TBL] [Abstract][Full Text] [Related]
2. Transformation suppressor activity of C3G is independent of its CDC25-homology domain.
Guerrero C; Fernandez-Medarde A; Rojas JM; Font de Mora J; Esteban LM; Santos E
Oncogene; 1998 Feb; 16(5):613-24. PubMed ID: 9482107
[TBL] [Abstract][Full Text] [Related]
3. Characterization of a protein kinase C-delta (PKC-delta) ATP binding mutant. An inactive enzyme that competitively inhibits wild type PKC-delta enzymatic activity.
Li W; Yu JC; Shin DY; Pierce JH
J Biol Chem; 1995 Apr; 270(14):8311-8. PubMed ID: 7713939
[TBL] [Abstract][Full Text] [Related]
4. Inhibition of oncogene-mediated transformation by ectopic expression of p21Waf1 in NIH3T3 cells.
Michieli P; Li W; Lorenzi MV; Miki T; Zakut R; Givol D; Pierce JH
Oncogene; 1996 Feb; 12(4):775-84. PubMed ID: 8632899
[TBL] [Abstract][Full Text] [Related]
5. H-ras and raf-1 cooperate in transformation of NIH3T3 fibroblasts.
Cuadrado A; Bruder JT; Heidaran MA; App H; Rapp UR; Aaronson SA
Oncogene; 1993 Sep; 8(9):2443-8. PubMed ID: 8361757
[TBL] [Abstract][Full Text] [Related]
6. Mechanisms of inhibition by heparin of PDGF stimulated MAP kinase activation in vascular smooth muscle cells.
Pukac LA; Carter JE; Ottlinger ME; Karnovsky MJ
J Cell Physiol; 1997 Jul; 172(1):69-78. PubMed ID: 9207927
[TBL] [Abstract][Full Text] [Related]
7. Angiotensin II type 1 receptor signals through Raf-1 by a protein kinase C-dependent, Ras-independent mechanism.
Arai H; Escobedo JA
Mol Pharmacol; 1996 Sep; 50(3):522-8. PubMed ID: 8794890
[TBL] [Abstract][Full Text] [Related]
8. Protein kinase C-epsilon associates with the Raf-1 kinase and induces the production of growth factors that stimulate Raf-1 activity.
Ueffing M; Lovrić J; Philipp A; Mischak H; Kolch W
Oncogene; 1997 Dec; 15(24):2921-7. PubMed ID: 9416835
[TBL] [Abstract][Full Text] [Related]
9. smg/rap1/Krev-1 p21s inhibit the signal pathway to the c-fos promoter/enhancer from c-Ki-ras p21 but not from c-raf-1 kinase in NIH3T3 cells.
Sakoda T; Kaibuchi K; Kishi K; Kishida S; Doi K; Hoshino M; Hattori S; Takai Y
Oncogene; 1992 Sep; 7(9):1705-11. PubMed ID: 1323817
[TBL] [Abstract][Full Text] [Related]
10. Endothelial exposure to hypoxia induces Egr-1 expression involving PKCalpha-mediated Ras/Raf-1/ERK1/2 pathway.
Lo LW; Cheng JJ; Chiu JJ; Wung BS; Liu YC; Wang DL
J Cell Physiol; 2001 Sep; 188(3):304-12. PubMed ID: 11473356
[TBL] [Abstract][Full Text] [Related]
11. Differential regulation of the p72-74 RAF-1 kinase in 3T3 fibroblasts expressing ras or src oncogenes.
Reed JC; Yum S; Cuddy MP; Turner BC; Rapp UR
Cell Growth Differ; 1991 May; 2(5):235-43. PubMed ID: 1888699
[TBL] [Abstract][Full Text] [Related]
12. Selective platelet-derived growth factor receptor kinase blockers reverse sis-transformation.
Kovalenko M; Gazit A; Böhmer A; Rorsman C; Rönnstrand L; Heldin CH; Waltenberger J; Böhmer FD; Levitzki A
Cancer Res; 1994 Dec; 54(23):6106-14. PubMed ID: 7954456
[TBL] [Abstract][Full Text] [Related]
13. Protein kinase C-stimulated formation of ethanolamine from phosphatidylethanolamine involves a protein phosphorylation mechanism: negative regulation by p21 Ras protein.
Deli E; Kiss Z
Arch Biochem Biophys; 2000 May; 377(1):171-7. PubMed ID: 10775457
[TBL] [Abstract][Full Text] [Related]
14. V-sis induces Egr-1 expression by a pathway mediated by c-Ha-Ras.
Huang RP; Ngo L; Okamura D; Tucker M; Adamson ED
J Cell Biochem; 1994 Dec; 56(4):469-79. PubMed ID: 7890805
[TBL] [Abstract][Full Text] [Related]
15. Protein kinase C alpha activates RAF-1 by direct phosphorylation.
Kolch W; Heidecker G; Kochs G; Hummel R; Vahidi H; Mischak H; Finkenzeller G; Marmé D; Rapp UR
Nature; 1993 Jul; 364(6434):249-52. PubMed ID: 8321321
[TBL] [Abstract][Full Text] [Related]
16. Rit, a non-lipid-modified Ras-related protein, transforms NIH3T3 cells without activating the ERK, JNK, p38 MAPK or PI3K/Akt pathways.
Rusyn EV; Reynolds ER; Shao H; Grana TM; Chan TO; Andres DA; Cox AD
Oncogene; 2000 Sep; 19(41):4685-94. PubMed ID: 11032018
[TBL] [Abstract][Full Text] [Related]
17. Truncated, desensitization-defective neurokinin receptors mediate sustained MAP kinase activation, cell growth and transformation by a Ras-independent mechanism.
Alblas J; van Etten I; Moolenaar WH
EMBO J; 1996 Jul; 15(13):3351-60. PubMed ID: 8670836
[TBL] [Abstract][Full Text] [Related]
18. Identification of AKT-regulated genes in inducible MERAkt cells.
Kuhn I; Bartholdi MF; Salamon H; Feldman RI; Roth RA; Johnson PH
Physiol Genomics; 2001 Dec; 7(2):105-14. PubMed ID: 11773597
[TBL] [Abstract][Full Text] [Related]
19. Calmodulin binds to K-Ras, but not to H- or N-Ras, and modulates its downstream signaling.
Villalonga P; López-Alcalá C; Bosch M; Chiloeches A; Rocamora N; Gil J; Marais R; Marshall CJ; Bachs O; Agell N
Mol Cell Biol; 2001 Nov; 21(21):7345-54. PubMed ID: 11585916
[TBL] [Abstract][Full Text] [Related]
20. C3G-mediated suppression of oncogene-induced focus formation in fibroblasts involves inhibition of ERK activation, cyclin A expression and alterations of anchorage-independent growth.
Guerrero C; Martín-Encabo S; Fernández-Medarde A; Santos E
Oncogene; 2004 Jun; 23(28):4885-93. PubMed ID: 15077165
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
