108 related articles for article (PubMed ID: 8840965)
1. Expression of p21 is not required for senescence of human fibroblasts.
Medcalf AS; Klein-Szanto AJ; Cristofalo VJ
Cancer Res; 1996 Oct; 56(20):4582-5. PubMed ID: 8840965
[TBL] [Abstract][Full Text] [Related]
2. Single-cell analysis of p16(INK4a) and p21(WAF1) expression suggests distinct mechanisms of senescence in normal human and Li-Fraumeni Syndrome fibroblasts.
Mirzayans R; Andrais B; Scott A; Paterson MC; Murray D
J Cell Physiol; 2010 Apr; 223(1):57-67. PubMed ID: 20039273
[TBL] [Abstract][Full Text] [Related]
3. Murine fibroblasts lacking p21 undergo senescence and are resistant to transformation by oncogenic Ras.
Pantoja C; Serrano M
Oncogene; 1999 Sep; 18(35):4974-82. PubMed ID: 10490832
[TBL] [Abstract][Full Text] [Related]
4. Possible involvement of p21 but not of p16 or p53 in keratinocyte senescence.
Sayama K; Shirakata Y; Midorikawa K; Hanakawa Y; Hashimoto K
J Cell Physiol; 1999 Apr; 179(1):40-4. PubMed ID: 10082130
[TBL] [Abstract][Full Text] [Related]
5. Aberrant p21WAF1-dependent growth arrest as the possible mechanism of abnormal resistance to ultraviolet light cytotoxicity in Li-Fraumeni syndrome fibroblast strains heterozygous for TP53 mutations.
Barley RD; Enns L; Paterson MC; Mirzayans R
Oncogene; 1998 Aug; 17(5):533-43. PubMed ID: 9704919
[TBL] [Abstract][Full Text] [Related]
6. Replicative senescence of normal human oral keratinocytes is associated with the loss of telomerase activity without shortening of telomeres.
Kang MK; Guo W; Park NH
Cell Growth Differ; 1998 Jan; 9(1):85-95. PubMed ID: 9438392
[TBL] [Abstract][Full Text] [Related]
7. Independent induction of senescence by p16INK4a and p21CIP1 in spontaneously immortalized human fibroblasts.
Vogt M; Haggblom C; Yeargin J; Christiansen-Weber T; Haas M
Cell Growth Differ; 1998 Feb; 9(2):139-46. PubMed ID: 9486850
[TBL] [Abstract][Full Text] [Related]
8. Regulation by ionizing radiation of CDC2, cyclin A, cyclin B, thymidine kinase, topoisomerase IIalpha, and RAD51 expression in normal human diploid fibroblasts is dependent on p53/p21Waf1.
de Toledo SM; Azzam EI; Keng P; Laffrenier S; Little JB
Cell Growth Differ; 1998 Nov; 9(11):887-96. PubMed ID: 9831241
[TBL] [Abstract][Full Text] [Related]
9. Detection of both mutant and wild-type p53 protein in normal skin fibroblasts and demonstration of a shared 'second hit' on p53 in diverse tumors from a cancer-prone family with Li-Fraumeni syndrome.
Srivastava S; Tong YA; Devadas K; Zou ZQ; Sykes VW; Chen Y; Blattner WA; Pirollo K; Chang EH
Oncogene; 1992 May; 7(5):987-91. PubMed ID: 1373881
[TBL] [Abstract][Full Text] [Related]
10. Investigation of the signaling pathways involved in the proliferative life span barriers in werner syndrome fibroblasts.
Davis T; Faragher RG; Jones CJ; Kipling D
Ann N Y Acad Sci; 2004 Jun; 1019():274-7. PubMed ID: 15247028
[TBL] [Abstract][Full Text] [Related]
11. Immortalization in a normal foreskin fibroblast culture following transduction of cyclin A2 or cdk1 genes in retroviral vectors.
Luo P; Tresini M; Cristofalo V; Chen X; Saulewicz A; Gray MD; Banker DE; Klingelhutz AL; Ohtsubo M; Takihara Y; Norwood TH
Exp Cell Res; 2004 Apr; 294(2):406-19. PubMed ID: 15023530
[TBL] [Abstract][Full Text] [Related]
12. Role of IFI 16 in cellular senescence of human fibroblasts.
Xin H; Pereira-Smith OM; Choubey D
Oncogene; 2004 Aug; 23(37):6209-17. PubMed ID: 15208661
[TBL] [Abstract][Full Text] [Related]
13. Role of the alternative INK4A proteins in human keratinocyte senescence: evidence for the specific inactivation of p16INK4A upon immortalization.
Munro J; Stott FJ; Vousden KH; Peters G; Parkinson EK
Cancer Res; 1999 Jun; 59(11):2516-21. PubMed ID: 10363964
[TBL] [Abstract][Full Text] [Related]
14. Role of p53 and p21waf1/cip1 in senescence-like terminal proliferation arrest induced in human tumor cells by chemotherapeutic drugs.
Chang BD; Xuan Y; Broude EV; Zhu H; Schott B; Fang J; Roninson IB
Oncogene; 1999 Aug; 18(34):4808-18. PubMed ID: 10490814
[TBL] [Abstract][Full Text] [Related]
15. Klotho RNAi induces premature senescence of human cells via a p53/p21 dependent pathway.
de Oliveira RM
FEBS Lett; 2006 Oct; 580(24):5753-8. PubMed ID: 17014852
[TBL] [Abstract][Full Text] [Related]
16. p53 is preferentially recruited to the promoters of growth arrest genes p21 and GADD45 during replicative senescence of normal human fibroblasts.
Jackson JG; Pereira-Smith OM
Cancer Res; 2006 Sep; 66(17):8356-60. PubMed ID: 16951143
[TBL] [Abstract][Full Text] [Related]
17. Histone deacetylase inhibitors induce a senescence-like state in human cells by a p16-dependent mechanism that is independent of a mitotic clock.
Munro J; Barr NI; Ireland H; Morrison V; Parkinson EK
Exp Cell Res; 2004 May; 295(2):525-38. PubMed ID: 15093749
[TBL] [Abstract][Full Text] [Related]
18. A senescence rescue screen identifies BCL6 as an inhibitor of anti-proliferative p19(ARF)-p53 signaling.
Shvarts A; Brummelkamp TR; Scheeren F; Koh E; Daley GQ; Spits H; Bernards R
Genes Dev; 2002 Mar; 16(6):681-6. PubMed ID: 11914273
[TBL] [Abstract][Full Text] [Related]
19. Cellular senescence induced by p53-ras cooperation is independent of p21waf1 in murine embryo fibroblasts.
Castro ME; del Valle Guijarro M; Moneo V; Carnero A
J Cell Biochem; 2004 Jun; 92(3):514-24. PubMed ID: 15156563
[TBL] [Abstract][Full Text] [Related]
20. DNA topoisomerase inhibitors induce reversible senescence in normal human fibroblasts.
Michishita E; Nakabayashi K; Ogino H; Suzuki T; Fujii M; Ayusawa D
Biochem Biophys Res Commun; 1998 Dec; 253(3):667-71. PubMed ID: 9918785
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
