BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

703 related articles for article (PubMed ID: 8918887)

  • 1. BID: a novel BH3 domain-only death agonist.
    Wang K; Yin XM; Chao DT; Milliman CL; Korsmeyer SJ
    Genes Dev; 1996 Nov; 10(22):2859-69. PubMed ID: 8918887
    [TBL] [Abstract][Full Text] [Related]  

  • 2. MAP-1, a novel proapoptotic protein containing a BH3-like motif that associates with Bax through its Bcl-2 homology domains.
    Tan KO; Tan KM; Chan SL; Yee KS; Bevort M; Ang KC; Yu VC
    J Biol Chem; 2001 Jan; 276(4):2802-7. PubMed ID: 11060313
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Proapoptotic protein Bax heterodimerizes with Bcl-2 and homodimerizes with Bax via a novel domain (BH3) distinct from BH1 and BH2.
    Zha H; Aimé-Sempé C; Sato T; Reed JC
    J Biol Chem; 1996 Mar; 271(13):7440-4. PubMed ID: 8631771
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Characterization of the anti-apoptotic mechanism of Bcl-B.
    Zhai D; Ke N; Zhang H; Ladror U; Joseph M; Eichinger A; Godzik A; Ng SC; Reed JC
    Biochem J; 2003 Nov; 376(Pt 1):229-36. PubMed ID: 12921534
    [TBL] [Abstract][Full Text] [Related]  

  • 5. The C. elegans orthologue ceBNIP3 interacts with CED-9 and CED-3 but kills through a BH3- and caspase-independent mechanism.
    Cizeau J; Ray R; Chen G; Gietz RD; Greenberg AH
    Oncogene; 2000 Nov; 19(48):5453-63. PubMed ID: 11114722
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Bcl-X(L) and calyculin A prevent translocation of Bax to mitochondria during apoptosis.
    Ganju N; Eastman A
    Biochem Biophys Res Commun; 2002 Mar; 291(5):1258-64. PubMed ID: 11883953
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Direct addition of BimL to mitochondria does not lead to cytochrome c release.
    Terradillos O; Montessuit S; Huang DC; Martinou JC
    FEBS Lett; 2002 Jul; 522(1-3):29-34. PubMed ID: 12095614
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Identification of novel isoforms of the BH3 domain protein Bim which directly activate Bax to trigger apoptosis.
    Marani M; Tenev T; Hancock D; Downward J; Lemoine NR
    Mol Cell Biol; 2002 Jun; 22(11):3577-89. PubMed ID: 11997495
    [TBL] [Abstract][Full Text] [Related]  

  • 9. BH3 domains of BH3-only proteins differentially regulate Bax-mediated mitochondrial membrane permeabilization both directly and indirectly.
    Kuwana T; Bouchier-Hayes L; Chipuk JE; Bonzon C; Sullivan BA; Green DR; Newmeyer DD
    Mol Cell; 2005 Feb; 17(4):525-35. PubMed ID: 15721256
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis.
    Desagher S; Osen-Sand A; Nichols A; Eskes R; Montessuit S; Lauper S; Maundrell K; Antonsson B; Martinou JC
    J Cell Biol; 1999 Mar; 144(5):891-901. PubMed ID: 10085289
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Bid chimeras indicate that most BH3-only proteins can directly activate Bak and Bax, and show no preference for Bak versus Bax.
    Hockings C; Anwari K; Ninnis RL; Brouwer J; O'Hely M; Evangelista M; Hinds MG; Czabotar PE; Lee EF; Fairlie WD; Dewson G; Kluck RM
    Cell Death Dis; 2015 Apr; 6(4):e1735. PubMed ID: 25906158
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Induction of apoptosis in prostate carcinoma cells by BH3 peptides which inhibit Bak/Bcl-2 interactions.
    Finnegan NM; Curtin JF; Prevost G; Morgan B; Cotter TG
    Br J Cancer; 2001 Jul; 85(1):115-21. PubMed ID: 11437412
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Membrane-insertion fragments of Bcl-xL, Bax, and Bid.
    García-Sáez AJ; Mingarro I; Pérez-Payá E; Salgado J
    Biochemistry; 2004 Aug; 43(34):10930-43. PubMed ID: 15323553
    [TBL] [Abstract][Full Text] [Related]  

  • 14. harakiri, a novel regulator of cell death, encodes a protein that activates apoptosis and interacts selectively with survival-promoting proteins Bcl-2 and Bcl-X(L).
    Inohara N; Ding L; Chen S; Núñez G
    EMBO J; 1997 Apr; 16(7):1686-94. PubMed ID: 9130713
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Functional identification of the apoptosis effector BH3 domain in cellular protein BNIP1.
    Yasuda M; Chinnadurai G
    Oncogene; 2000 May; 19(19):2363-7. PubMed ID: 10822388
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Bid-induced cytochrome c release is mediated by a pathway independent of mitochondrial permeability transition pore and Bax.
    Kim TH; Zhao Y; Barber MJ; Kuharsky DK; Yin XM
    J Biol Chem; 2000 Dec; 275(50):39474-81. PubMed ID: 10982793
    [TBL] [Abstract][Full Text] [Related]  

  • 17. The conserved N-terminal BH4 domain of Bcl-2 homologues is essential for inhibition of apoptosis and interaction with CED-4.
    Huang DC; Adams JM; Cory S
    EMBO J; 1998 Feb; 17(4):1029-39. PubMed ID: 9463381
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Trail-induced apoptosis in Type I leukemic cells is not enhanced by overexpression of bax.
    Jia L; Patwari Y; Kelsey SM; Newland AC
    Biochem Biophys Res Commun; 2001 May; 283(5):1037-45. PubMed ID: 11355877
    [TBL] [Abstract][Full Text] [Related]  

  • 19. The first alpha helix of Bax plays a necessary role in its ligand-induced activation by the BH3-only proteins Bid and PUMA.
    Cartron PF; Gallenne T; Bougras G; Gautier F; Manero F; Vusio P; Meflah K; Vallette FM; Juin P
    Mol Cell; 2004 Dec; 16(5):807-18. PubMed ID: 15574335
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Mutagenesis of the BH3 domain of BAX identifies residues critical for dimerization and killing.
    Wang K; Gross A; Waksman G; Korsmeyer SJ
    Mol Cell Biol; 1998 Oct; 18(10):6083-9. PubMed ID: 9742125
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 36.