These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
328 related articles for article (PubMed ID: 8951817)
1. Co-ordinate regulation of Salmonella typhimurium invasion genes by environmental and regulatory factors is mediated by control of hilA expression. Bajaj V; Lucas RL; Hwang C; Lee CA Mol Microbiol; 1996 Nov; 22(4):703-14. PubMed ID: 8951817 [TBL] [Abstract][Full Text] [Related]
2. hilA is a novel ompR/toxR family member that activates the expression of Salmonella typhimurium invasion genes. Bajaj V; Hwang C; Lee CA Mol Microbiol; 1995 Nov; 18(4):715-27. PubMed ID: 8817493 [TBL] [Abstract][Full Text] [Related]
3. Two AraC/XylS family members can independently counteract the effect of repressing sequences upstream of the hilA promoter. Schechter LM; Damrauer SM; Lee CA Mol Microbiol; 1999 May; 32(3):629-42. PubMed ID: 10320584 [TBL] [Abstract][Full Text] [Related]
4. Short-chain volatile fatty acids modulate the expression of the hilA and invF genes of Salmonella typhimurium. Durant JA; Corrier DE; Ricke SC J Food Prot; 2000 May; 63(5):573-8. PubMed ID: 10826713 [TBL] [Abstract][Full Text] [Related]
5. Identification and characterization of mutants with increased expression of hilA, the invasion gene transcriptional activator of Salmonella typhimurium. Fahlen TF; Mathur N; Jones BD FEMS Immunol Med Microbiol; 2000 May; 28(1):25-35. PubMed ID: 10767604 [TBL] [Abstract][Full Text] [Related]
6. Roles of hilC and hilD in regulation of hilA expression in Salmonella enterica serovar Typhimurium. Lucas RL; Lee CA J Bacteriol; 2001 May; 183(9):2733-45. PubMed ID: 11292791 [TBL] [Abstract][Full Text] [Related]
7. AraC/XylS family members, HilD and HilC, directly activate virulence gene expression independently of HilA in Salmonella typhimurium. Akbar S; Schechter LM; Lostroh CP; Lee CA Mol Microbiol; 2003 Feb; 47(3):715-28. PubMed ID: 12535071 [TBL] [Abstract][Full Text] [Related]
8. Hha is a negative modulator of transcription of hilA, the Salmonella enterica serovar Typhimurium invasion gene transcriptional activator. Fahlen TF; Wilson RL; Boddicker JD; Jones BD J Bacteriol; 2001 Nov; 183(22):6620-9. PubMed ID: 11673432 [TBL] [Abstract][Full Text] [Related]
9. HilE interacts with HilD and negatively regulates hilA transcription and expression of the Salmonella enterica serovar Typhimurium invasive phenotype. Baxter MA; Fahlen TF; Wilson RL; Jones BD Infect Immun; 2003 Mar; 71(3):1295-305. PubMed ID: 12595445 [TBL] [Abstract][Full Text] [Related]
10. Coordinate regulation of Salmonella pathogenicity island 1 (SPI1) and SPI4 in Salmonella enterica serovar Typhimurium. Main-Hester KL; Colpitts KM; Thomas GA; Fang FC; Libby SJ Infect Immun; 2008 Mar; 76(3):1024-35. PubMed ID: 18160484 [TBL] [Abstract][Full Text] [Related]
11. The Small RNA PinT Contributes to PhoP-Mediated Regulation of the Kim K; Palmer AD; Vanderpool CK; Slauch JM J Bacteriol; 2019 Oct; 201(19):. PubMed ID: 31262841 [No Abstract] [Full Text] [Related]
12. Integration host factor alleviates H-NS silencing of the Salmonella enterica serovar Typhimurium master regulator of SPI1, hilA. Queiroz MH; Madrid C; Paytubi S; Balsalobre C; Juárez A Microbiology (Reading); 2011 Sep; 157(Pt 9):2504-2514. PubMed ID: 21680637 [TBL] [Abstract][Full Text] [Related]
13. Transcription of the Salmonella invasion gene activator, hilA, requires HilD activation in the absence of negative regulators. Boddicker JD; Knosp BM; Jones BD J Bacteriol; 2003 Jan; 185(2):525-33. PubMed ID: 12511499 [TBL] [Abstract][Full Text] [Related]
14. Lon protease activity causes down-regulation of Salmonella pathogenicity island 1 invasion gene expression after infection of epithelial cells. Boddicker JD; Jones BD Infect Immun; 2004 Apr; 72(4):2002-13. PubMed ID: 15039320 [TBL] [Abstract][Full Text] [Related]
15. The fimYZ genes regulate Salmonella enterica Serovar Typhimurium invasion in addition to type 1 fimbrial expression and bacterial motility. Baxter MA; Jones BD Infect Immun; 2005 Mar; 73(3):1377-85. PubMed ID: 15731035 [TBL] [Abstract][Full Text] [Related]
16. Invasion genes are not required for Salmonella enterica serovar typhimurium to breach the intestinal epithelium: evidence that salmonella pathogenicity island 1 has alternative functions during infection. Murray RA; Lee CA Infect Immun; 2000 Sep; 68(9):5050-5. PubMed ID: 10948124 [TBL] [Abstract][Full Text] [Related]
17. HilD, HilC and RtsA constitute a feed forward loop that controls expression of the SPI1 type three secretion system regulator hilA in Salmonella enterica serovar Typhimurium. Ellermeier CD; Ellermeier JR; Slauch JM Mol Microbiol; 2005 Aug; 57(3):691-705. PubMed ID: 16045614 [TBL] [Abstract][Full Text] [Related]
18. AraC/XylS family members, HilC and HilD, directly bind and derepress the Salmonella typhimurium hilA promoter. Schechter LM; Lee CA Mol Microbiol; 2001 Jun; 40(6):1289-99. PubMed ID: 11442828 [TBL] [Abstract][Full Text] [Related]
19. The ATP-dependent lon protease of Salmonella enterica serovar Typhimurium regulates invasion and expression of genes carried on Salmonella pathogenicity island 1. Takaya A; Tomoyasu T; Tokumitsu A; Morioka M; Yamamoto T J Bacteriol; 2002 Jan; 184(1):224-32. PubMed ID: 11741864 [TBL] [Abstract][Full Text] [Related]
20. InvF is required for expression of genes encoding proteins secreted by the SPI1 type III secretion apparatus in Salmonella typhimurium. Darwin KH; Miller VL J Bacteriol; 1999 Aug; 181(16):4949-54. PubMed ID: 10438766 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]