468 related articles for article (PubMed ID: 8995390)
1. Tumor necrosis factor-alpha-induced insulin resistance in 3T3-L1 adipocytes is accompanied by a loss of insulin receptor substrate-1 and GLUT4 expression without a loss of insulin receptor-mediated signal transduction.
Stephens JM; Lee J; Pilch PF
J Biol Chem; 1997 Jan; 272(2):971-6. PubMed ID: 8995390
[TBL] [Abstract][Full Text] [Related]
2. Interleukin-6 (IL-6) induces insulin resistance in 3T3-L1 adipocytes and is, like IL-8 and tumor necrosis factor-alpha, overexpressed in human fat cells from insulin-resistant subjects.
Rotter V; Nagaev I; Smith U
J Biol Chem; 2003 Nov; 278(46):45777-84. PubMed ID: 12952969
[TBL] [Abstract][Full Text] [Related]
3. Dexamethasone-induced insulin resistance in 3T3-L1 adipocytes is due to inhibition of glucose transport rather than insulin signal transduction.
Sakoda H; Ogihara T; Anai M; Funaki M; Inukai K; Katagiri H; Fukushima Y; Onishi Y; Ono H; Fujishiro M; Kikuchi M; Oka Y; Asano T
Diabetes; 2000 Oct; 49(10):1700-8. PubMed ID: 11016454
[TBL] [Abstract][Full Text] [Related]
4. Reduced glucose uptake precedes insulin signaling defects in adipocytes from heterozygous GLUT4 knockout mice.
Li J; Houseknecht KL; Stenbit AE; Katz EB; Charron MJ
FASEB J; 2000 Jun; 14(9):1117-25. PubMed ID: 10834933
[TBL] [Abstract][Full Text] [Related]
5. Tumor necrosis factor-alpha suppresses adipocyte-specific genes and activates expression of preadipocyte genes in 3T3-L1 adipocytes: nuclear factor-kappaB activation by TNF-alpha is obligatory.
Ruan H; Hacohen N; Golub TR; Van Parijs L; Lodish HF
Diabetes; 2002 May; 51(5):1319-36. PubMed ID: 11978627
[TBL] [Abstract][Full Text] [Related]
6. Lipid mediators of insulin resistance: ceramide signalling down-regulates GLUT4 gene transcription in 3T3-L1 adipocytes.
Long SD; Pekala PH
Biochem J; 1996 Oct; 319 ( Pt 1)(Pt 1):179-84. PubMed ID: 8870666
[TBL] [Abstract][Full Text] [Related]
7. Effects of cell-permeable ceramides and tumor necrosis factor-alpha on insulin signaling and glucose uptake in 3T3-L1 adipocytes.
Wang CN; O'Brien L; Brindley DN
Diabetes; 1998 Jan; 47(1):24-31. PubMed ID: 9421370
[TBL] [Abstract][Full Text] [Related]
8. Platelet-derived growth factor inhibits insulin stimulation of insulin receptor substrate-1-associated phosphatidylinositol 3-kinase in 3T3-L1 adipocytes without affecting glucose transport.
Staubs PA; Nelson JG; Reichart DR; Olefsky JM
J Biol Chem; 1998 Sep; 273(39):25139-47. PubMed ID: 9737973
[TBL] [Abstract][Full Text] [Related]
9. Engagement of the insulin-sensitive pathway in the stimulation of glucose transport by alpha-lipoic acid in 3T3-L1 adipocytes.
Yaworsky K; Somwar R; Ramlal T; Tritschler HJ; Klip A
Diabetologia; 2000 Mar; 43(3):294-303. PubMed ID: 10768090
[TBL] [Abstract][Full Text] [Related]
10. Tumor necrosis factor (TNF)-α-induced repression of GKAP42 protein levels through cGMP-dependent kinase (cGK)-Iα causes insulin resistance in 3T3-L1 adipocytes.
Ando Y; Shinozawa Y; Iijima Y; Yu BC; Sone M; Ooi Y; Watanaka Y; Chida K; Hakuno F; Takahashi S
J Biol Chem; 2015 Feb; 290(9):5881-92. PubMed ID: 25586176
[TBL] [Abstract][Full Text] [Related]
11. Cross-talk mechanisms in the development of insulin resistance of skeletal muscle cells palmitate rather than tumour necrosis factor inhibits insulin-dependent protein kinase B (PKB)/Akt stimulation and glucose uptake.
Storz P; Döppler H; Wernig A; Pfizenmaier K; Müller G
Eur J Biochem; 1999 Nov; 266(1):17-25. PubMed ID: 10542046
[TBL] [Abstract][Full Text] [Related]
12. Tumor necrosis factor-alpha and ceramides in insulin resistance.
Brindley DN; Wang CN; Mei J; Xu J; Hanna AN
Lipids; 1999; 34 Suppl():S85-8. PubMed ID: 10419100
[TBL] [Abstract][Full Text] [Related]
13. Phosphorylation of Ser24 in the pleckstrin homology domain of insulin receptor substrate-1 by Mouse Pelle-like kinase/interleukin-1 receptor-associated kinase: cross-talk between inflammatory signaling and insulin signaling that may contribute to insulin resistance.
Kim JA; Yeh DC; Ver M; Li Y; Carranza A; Conrads TP; Veenstra TD; Harrington MA; Quon MJ
J Biol Chem; 2005 Jun; 280(24):23173-83. PubMed ID: 15849359
[TBL] [Abstract][Full Text] [Related]
14. Activation of the mammalian target of rapamycin pathway acutely inhibits insulin signaling to Akt and glucose transport in 3T3-L1 and human adipocytes.
Tremblay F; Gagnon A; Veilleux A; Sorisky A; Marette A
Endocrinology; 2005 Mar; 146(3):1328-37. PubMed ID: 15576463
[TBL] [Abstract][Full Text] [Related]
15. Substrates of semicarbazide-sensitive amine oxidase co-operate with vanadate to stimulate tyrosine phosphorylation of insulin-receptor-substrate proteins, phosphoinositide 3-kinase activity and GLUT4 translocation in adipose cells.
Enrique-Tarancón G; Castan I; Morin N; Marti L; Abella A; Camps M; Casamitjana R; Palacín M; Testar X; Degerman E; Carpéné C; Zorzano A
Biochem J; 2000 Aug; 350 Pt 1(Pt 1):171-80. PubMed ID: 10926841
[TBL] [Abstract][Full Text] [Related]
16. Endothelin-1-stimulated glucose uptake is desensitized by tumor necrosis factor-alpha in 3T3-L1 adipocytes.
Rachdaoui N; Nagy LE
Am J Physiol Endocrinol Metab; 2003 Sep; 285(3):E545-51. PubMed ID: 12773307
[TBL] [Abstract][Full Text] [Related]
17. Arsenite stimulated glucose transport in 3T3-L1 adipocytes involves both Glut4 translocation and p38 MAPK activity.
Bazuine M; Ouwens DM; Gomes de Mesquita DS; Maassen JA
Eur J Biochem; 2003 Oct; 270(19):3891-903. PubMed ID: 14511371
[TBL] [Abstract][Full Text] [Related]
18. Chronic endothelin-1 treatment leads to heterologous desensitization of insulin signaling in 3T3-L1 adipocytes.
Ishibashi KI; Imamura T; Sharma PM; Huang J; Ugi S; Olefsky JM
J Clin Invest; 2001 May; 107(9):1193-202. PubMed ID: 11342583
[TBL] [Abstract][Full Text] [Related]
19. Tumor necrosis factor-alpha causes insulin receptor substrate-2-mediated insulin resistance and inhibits insulin-induced adipogenesis in fetal brown adipocytes.
Valverde AM; Teruel T; Navarro P; Benito M; Lorenzo M
Endocrinology; 1998 Mar; 139(3):1229-38. PubMed ID: 9492058
[TBL] [Abstract][Full Text] [Related]
20. Nitric oxide stimulates glucose transport through insulin-independent GLUT4 translocation in 3T3-L1 adipocytes.
Tanaka T; Nakatani K; Morioka K; Urakawa H; Maruyama N; Kitagawa N; Katsuki A; Araki-Sasaki R; Hori Y; Gabazza EC; Yano Y; Wada H; Nobori T; Sumida Y; Adachi Y
Eur J Endocrinol; 2003 Jul; 149(1):61-7. PubMed ID: 12824867
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]