197 related articles for article (PubMed ID: 9060667)
1. Identification of active-site residues in protease 3C of hepatitis A virus by site-directed mutagenesis.
Gosert R; Dollenmaier G; Weitz M
J Virol; 1997 Apr; 71(4):3062-8. PubMed ID: 9060667
[TBL] [Abstract][Full Text] [Related]
2. Identification and site-directed mutagenesis of the primary (2A/2B) cleavage site of the hepatitis A virus polyprotein: functional impact on the infectivity of HAV RNA transcripts.
Martin A; Escriou N; Chao SF; Girard M; Lemon SM; Wychowski C
Virology; 1995 Oct; 213(1):213-22. PubMed ID: 7483265
[TBL] [Abstract][Full Text] [Related]
3. Maturation of the hepatitis A virus capsid protein VP1 is not dependent on processing by the 3Cpro proteinase.
Martin A; Bénichou D; Chao SF; Cohen LM; Lemon SM
J Virol; 1999 Aug; 73(8):6220-7. PubMed ID: 10400711
[TBL] [Abstract][Full Text] [Related]
4. Proteinase 3C of hepatitis A virus (HAV) cleaves the HAV polyprotein P2-P3 at all sites including VP1/2A and 2A/2B.
Schultheiss T; Kusov YY; Gauss-Müller V
Virology; 1994 Jan; 198(1):275-81. PubMed ID: 8259663
[TBL] [Abstract][Full Text] [Related]
5. Polyprotein processing in Southampton virus: identification of 3C-like protease cleavage sites by in vitro mutagenesis.
Liu B; Clarke IN; Lambden PR
J Virol; 1996 Apr; 70(4):2605-10. PubMed ID: 8642693
[TBL] [Abstract][Full Text] [Related]
6. Polyprotein processing in cis and in trans by hepatitis A virus 3C protease cloned and expressed in Escherichia coli.
Harmon SA; Updike W; Jia XY; Summers DF; Ehrenfeld E
J Virol; 1992 Sep; 66(9):5242-7. PubMed ID: 1323691
[TBL] [Abstract][Full Text] [Related]
7. Improving proteolytic cleavage at the 3A/3B site of the hepatitis A virus polyprotein impairs processing and particle formation, and the impairment can be complemented in trans by 3AB and 3ABC.
Kusov Y; Gauss-Müller V
J Virol; 1999 Dec; 73(12):9867-78. PubMed ID: 10559299
[TBL] [Abstract][Full Text] [Related]
8. Identification of essential amino acid residues in the functional activity of poliovirus 2A protease.
Yu SF; Lloyd RE
Virology; 1991 Jun; 182(2):615-25. PubMed ID: 1850921
[TBL] [Abstract][Full Text] [Related]
9. Expression of hepatitis A virus precursor protein P3 in vivo and in vitro: polyprotein processing of the 3CD cleavage site.
Tesar M; Pak I; Jia XY; Richards OC; Summers DF; Ehrenfeld E
Virology; 1994 Feb; 198(2):524-33. PubMed ID: 8291234
[TBL] [Abstract][Full Text] [Related]
10. Site-directed mutagenesis of the putative active site residues of 3C proteinase of coxsackievirus B3: evidence of a functional relationship with trypsin-like serine proteinases.
Miyashita K; Kusumi M; Utsumi R; Katayama S; Noda M; Komano T; Satoh N
Protein Eng; 1993 Feb; 6(2):189-93. PubMed ID: 8386363
[TBL] [Abstract][Full Text] [Related]
11. Cleavage specificity of purified recombinant hepatitis A virus 3C proteinase on natural substrates.
Schultheiss T; Sommergruber W; Kusov Y; Gauss-Müller V
J Virol; 1995 Mar; 69(3):1727-33. PubMed ID: 7853510
[TBL] [Abstract][Full Text] [Related]
12. In vitro RNA binding of the hepatitis A virus proteinase 3C (HAV 3Cpro) to secondary structure elements within the 5' terminus of the HAV genome.
Kusov YY; Gauss-Müller V
RNA; 1997 Mar; 3(3):291-302. PubMed ID: 9056766
[TBL] [Abstract][Full Text] [Related]
13. Substitution mutations at the putative catalytic triad of the poliovirus 3C protease have differential effects on cleavage at different sites.
Kean KM; Howell MT; Grünert S; Girard M; Jackson RJ
Virology; 1993 May; 194(1):360-4. PubMed ID: 8097606
[TBL] [Abstract][Full Text] [Related]
14. Characterization of replication-competent hepatitis A virus constructs containing insertions at the N terminus of the polyprotein.
Zhang Y; Kaplan GG
J Virol; 1998 Jan; 72(1):349-57. PubMed ID: 9420233
[TBL] [Abstract][Full Text] [Related]
15. Poliovirus capsid proteins derived from P1 precursors with glutamine-valine cleavage sites have defects in assembly and RNA encapsidation.
Ansardi DC; Morrow CD
J Virol; 1993 Dec; 67(12):7284-97. PubMed ID: 8230452
[TBL] [Abstract][Full Text] [Related]
16. Proteinase 3C-mediated processing of VP1-2A of two hepatitis A virus strains: in vivo evidence for cleavage at amino acid position 273/274 of VP1.
Probst C; Jecht M; Gauss-Müller V
J Virol; 1997 Apr; 71(4):3288-92. PubMed ID: 9060697
[TBL] [Abstract][Full Text] [Related]
17. Cleavage site mutations in the encephalomyocarditis virus P3 region lethally abrogate the normal processing cascade.
Hall DJ; Palmenberg AC
J Virol; 1996 Sep; 70(9):5954-61. PubMed ID: 8709217
[TBL] [Abstract][Full Text] [Related]
18. Proteolytic activity of hepatitis A virus 3C protein.
Jia XY; Ehrenfeld E; Summers DF
J Virol; 1991 May; 65(5):2595-600. PubMed ID: 1850033
[TBL] [Abstract][Full Text] [Related]
19. Identification of hepatitis A virus non-structural protein 2B and its release by the major virus protease 3C.
Gosert R; Cassinotti P; Siegl G; Weitz M
J Gen Virol; 1996 Feb; 77 ( Pt 2 )():247-55. PubMed ID: 8627228
[TBL] [Abstract][Full Text] [Related]
20. Translation of hepatitis A virus RNA in vitro: aberrant internal initiations influenced by 5' noncoding region.
Jia XY; Scheper G; Brown D; Updike W; Harmon S; Richards O; Summers D; Ehrenfeld E
Virology; 1991 Jun; 182(2):712-22. PubMed ID: 1850924
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
