251 related articles for article (PubMed ID: 9076742)
1. SopA, the outer membrane protease responsible for polar localization of IcsA in Shigella flexneri.
Egile C; d'Hauteville H; Parsot C; Sansonetti PJ
Mol Microbiol; 1997 Mar; 23(5):1063-73. PubMed ID: 9076742
[TBL] [Abstract][Full Text] [Related]
2. The unipolar Shigella surface protein IcsA is targeted directly to the bacterial old pole: IcsP cleavage of IcsA occurs over the entire bacterial surface.
Steinhauer J; Agha R; Pham T; Varga AW; Goldberg MB
Mol Microbiol; 1999 Apr; 32(2):367-77. PubMed ID: 10231492
[TBL] [Abstract][Full Text] [Related]
3. The making of a gradient: IcsA (VirG) polarity in Shigella flexneri.
Robbins JR; Monack D; McCallum SJ; Vegas A; Pham E; Goldberg MB; Theriot JA
Mol Microbiol; 2001 Aug; 41(4):861-72. PubMed ID: 11532149
[TBL] [Abstract][Full Text] [Related]
4. Disruption of IcsP, the major Shigella protease that cleaves IcsA, accelerates actin-based motility.
Shere KD; Sallustio S; Manessis A; D'Aversa TG; Goldberg MB
Mol Microbiol; 1997 Aug; 25(3):451-62. PubMed ID: 9302008
[TBL] [Abstract][Full Text] [Related]
5. Establishment of unipolar localization of IcsA in Shigella flexneri 2a is not dependent on virulence plasmid determinants.
Sandlin RC; Maurelli AT
Infect Immun; 1999 Jan; 67(1):350-6. PubMed ID: 9864236
[TBL] [Abstract][Full Text] [Related]
6. The actin-based motility defect of a Shigella flexneri rmlD rough LPS mutant is not due to loss of IcsA polarity.
Van den Bosch L; Morona R
Microb Pathog; 2003 Jul; 35(1):11-8. PubMed ID: 12860454
[TBL] [Abstract][Full Text] [Related]
7. Actin-based motility is sufficient for bacterial membrane protrusion formation and host cell uptake.
Monack DM; Theriot JA
Cell Microbiol; 2001 Sep; 3(9):633-47. PubMed ID: 11553015
[TBL] [Abstract][Full Text] [Related]
8. Polar targeting of Shigella virulence factor IcsA in Enterobacteriacae and Vibrio.
Charles M; Pérez M; Kobil JH; Goldberg MB
Proc Natl Acad Sci U S A; 2001 Aug; 98(17):9871-6. PubMed ID: 11481451
[TBL] [Abstract][Full Text] [Related]
9. Genetic modulation of Shigella flexneri 2a lipopolysaccharide O antigen modal chain length reveals that it has been optimized for virulence.
Morona R; Daniels C; Van Den Bosch L
Microbiology (Reading); 2003 Apr; 149(Pt 4):925-939. PubMed ID: 12686635
[TBL] [Abstract][Full Text] [Related]
10. Outer membrane protein A (OmpA): a new player in shigella flexneri protrusion formation and inter-cellular spreading.
Ambrosi C; Pompili M; Scribano D; Zagaglia C; Ripa S; Nicoletti M
PLoS One; 2012; 7(11):e49625. PubMed ID: 23166731
[TBL] [Abstract][Full Text] [Related]
11. Effect of O side-chain length and composition on the virulence of Shigella flexneri 2a.
Sandlin RC; Goldberg MB; Maurelli AT
Mol Microbiol; 1996 Oct; 22(1):63-73. PubMed ID: 8899709
[TBL] [Abstract][Full Text] [Related]
12. Functional analysis of a rickettsial OmpA homology domain of Shigella flexneri icsA.
Charles M; Magdalena J; Theriot JA; Goldberg MB
J Bacteriol; 1999 Feb; 181(3):869-78. PubMed ID: 9922250
[TBL] [Abstract][Full Text] [Related]
13. Cardiolipin Synthesis and Outer Membrane Localization Are Required for
Rossi RM; Yum L; Agaisse H; Payne SM
mBio; 2017 Aug; 8(4):. PubMed ID: 28851846
[TBL] [Abstract][Full Text] [Related]
14. Lack of cleavage of IcsA in Shigella flexneri causes aberrant movement and allows demonstration of a cross-reactive eukaryotic protein.
d'Hauteville H; Dufourcq Lagelouse R; Nato F; Sansonetti PJ
Infect Immun; 1996 Feb; 64(2):511-7. PubMed ID: 8550200
[TBL] [Abstract][Full Text] [Related]
15. Polar localization of PhoN2, a periplasmic virulence-associated factor of Shigella flexneri, is required for proper IcsA exposition at the old bacterial pole.
Scribano D; Petrucca A; Pompili M; Ambrosi C; Bruni E; Zagaglia C; Prosseda G; Nencioni L; Casalino M; Polticelli F; Nicoletti M
PLoS One; 2014; 9(2):e90230. PubMed ID: 24587292
[TBL] [Abstract][Full Text] [Related]
16. Avirulence of rough mutants of Shigella flexneri: requirement of O antigen for correct unipolar localization of IcsA in the bacterial outer membrane.
Sandlin RC; Lampel KA; Keasler SP; Goldberg MB; Stolzer AL; Maurelli AT
Infect Immun; 1995 Jan; 63(1):229-37. PubMed ID: 7528731
[TBL] [Abstract][Full Text] [Related]
17. Cleavage of Shigella surface protein VirG occurs at a specific site, but the secretion is not essential for intracellular spreading.
Fukuda I; Suzuki T; Munakata H; Hayashi N; Katayama E; Yoshikawa M; Sasakawa C
J Bacteriol; 1995 Apr; 177(7):1719-26. PubMed ID: 7896693
[TBL] [Abstract][Full Text] [Related]
18. Mutagenesis of the Shigella flexneri autotransporter IcsA reveals novel functional regions involved in IcsA biogenesis and recruitment of host neural Wiscott-Aldrich syndrome protein.
May KL; Morona R
J Bacteriol; 2008 Jul; 190(13):4666-76. PubMed ID: 18456802
[TBL] [Abstract][Full Text] [Related]
19. Activation of the CDC42 effector N-WASP by the Shigella flexneri IcsA protein promotes actin nucleation by Arp2/3 complex and bacterial actin-based motility.
Egile C; Loisel TP; Laurent V; Li R; Pantaloni D; Sansonetti PJ; Carlier MF
J Cell Biol; 1999 Sep; 146(6):1319-32. PubMed ID: 10491394
[TBL] [Abstract][Full Text] [Related]
20. The unrelated surface proteins ActA of Listeria monocytogenes and IcsA of Shigella flexneri are sufficient to confer actin-based motility on Listeria innocua and Escherichia coli respectively.
Kocks C; Marchand JB; Gouin E; d'Hauteville H; Sansonetti PJ; Carlier MF; Cossart P
Mol Microbiol; 1995 Nov; 18(3):413-23. PubMed ID: 8748026
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
