BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

598 related articles for article (PubMed ID: 9111507)

  • 1. Oxidative stress, mutant SOD1, and neurofilament pathology in transgenic mouse models of human motor neuron disease.
    Tu PH; Gurney ME; Julien JP; Lee VM; Trojanowski JQ
    Lab Invest; 1997 Apr; 76(4):441-56. PubMed ID: 9111507
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Amyotrophic lateral sclerosis and Alzheimer disease. Lessons from model systems.
    Price DL; Wong PC; Borchelt DR; Pardo CA; Thinakaran G; Doan AP; Lee MK; Martin LJ; Sisodia SS
    Rev Neurol (Paris); 1997 Sep; 153(8-9):484-95. PubMed ID: 9683997
    [TBL] [Abstract][Full Text] [Related]  

  • 3. [Familial amyotrophic lateral sclerosis and mutations in the Cu/Zn superoxide dismutase gene].
    Nakano R
    Rinsho Shinkeigaku; 1995 Dec; 35(12):1546-8. PubMed ID: 8752459
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Therapeutic benefits of putrescine-modified catalase in a transgenic mouse model of familial amyotrophic lateral sclerosis.
    Reinholz MM; Merkle CM; Poduslo JF
    Exp Neurol; 1999 Sep; 159(1):204-16. PubMed ID: 10486188
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Transgenics, toxicity and therapeutics in rodent models of mutant SOD1-mediated familial ALS.
    Turner BJ; Talbot K
    Prog Neurobiol; 2008 May; 85(1):94-134. PubMed ID: 18282652
    [TBL] [Abstract][Full Text] [Related]  

  • 6. [Amyotrophic lateral sclerosis: recent insights from transgenic animal models with SOD1 mutations].
    Aoki M
    Rinsho Shinkeigaku; 2004 Nov; 44(11):788-91. PubMed ID: 15651292
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Pathogenic mechanisms in familial amyotrophic lateral sclerosis due to mutation of Cu, Zn superoxide dismutase.
    Gurney ME; Cutting FB; Zhai P; Andrus PK; Hall ED
    Pathol Biol (Paris); 1996 Jan; 44(1):51-6. PubMed ID: 8734301
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Superoxide dismutase--applications and relevance to human diseases.
    Noor R; Mittal S; Iqbal J
    Med Sci Monit; 2002 Sep; 8(9):RA210-5. PubMed ID: 12218958
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D; Haasdijk ED; Grashorn JA; Hawkins R; van Duijn W; Verspaget HW; London J; Holstege JC
    Neurobiol Dis; 2000 Dec; 7(6 Pt B):623-43. PubMed ID: 11114261
    [TBL] [Abstract][Full Text] [Related]  

  • 10. SOD1 mutation is associated with accumulation of neurofilaments in amyotrophic lateral sclerosis.
    Rouleau GA; Clark AW; Rooke K; Pramatarova A; Krizus A; Suchowersky O; Julien JP; Figlewicz D
    Ann Neurol; 1996 Jan; 39(1):128-31. PubMed ID: 8572658
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Neurofilaments and orthograde transport are reduced in ventral root axons of transgenic mice that express human SOD1 with a G93A mutation.
    Zhang B; Tu P; Abtahian F; Trojanowski JQ; Lee VM
    J Cell Biol; 1997 Dec; 139(5):1307-15. PubMed ID: 9382875
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Comparison of pathological alterations in ALS and a murine transgenic model: pathogenetic implications.
    Dal Canto MC
    Clin Neurosci; 1995-1996; 3(6):332-7. PubMed ID: 9021254
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Transgenic mice carrying a human mutant superoxide dismutase transgene develop neuronal cytoskeletal pathology resembling human amyotrophic lateral sclerosis lesions.
    Tu PH; Raju P; Robinson KA; Gurney ME; Trojanowski JQ; Lee VM
    Proc Natl Acad Sci U S A; 1996 Apr; 93(7):3155-60. PubMed ID: 8610185
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Familial amyotrophic lateral sclerosis.
    Siddique T; Hentati A
    Clin Neurosci; 1995-1996; 3(6):338-47. PubMed ID: 9021255
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis.
    Rosen DR; Siddique T; Patterson D; Figlewicz DA; Sapp P; Hentati A; Donaldson D; Goto J; O'Regan JP; Deng HX
    Nature; 1993 Mar; 362(6415):59-62. PubMed ID: 8446170
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Sequence variants in human neurofilament proteins: absence of linkage to familial amyotrophic lateral sclerosis.
    Vechio JD; Bruijn LI; Xu Z; Brown RH; Cleveland DW
    Ann Neurol; 1996 Oct; 40(4):603-10. PubMed ID: 8871580
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Defective axonal transport in a transgenic mouse model of amyotrophic lateral sclerosis.
    Collard JF; Côté F; Julien JP
    Nature; 1995 May; 375(6526):61-4. PubMed ID: 7536898
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Animal models of ALS.
    Pioro EP; Mitsumoto H
    Clin Neurosci; 1995-1996; 3(6):375-85. PubMed ID: 9021259
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Neuronal over-expression of chromogranin A accelerates disease onset in a mouse model of ALS.
    Ezzi SA; Larivière R; Urushitani M; Julien JP
    J Neurochem; 2010 Dec; 115(5):1102-11. PubMed ID: 20807312
    [TBL] [Abstract][Full Text] [Related]  

  • 20. The neuronal Golgi apparatus is fragmented in transgenic mice expressing a mutant human SOD1, but not in mice expressing the human NF-H gene.
    Stieber A; Gonatas JO; Collard J; Meier J; Julien J; Schweitzer P; Gonatas NK
    J Neurol Sci; 2000 Feb; 173(1):63-72. PubMed ID: 10675581
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 30.