403 related articles for article (PubMed ID: 9115981)
1. Importance of two adjacent C-terminal sequences of SNAP-25 in exocytosis from intact and permeabilized chromaffin cells revealed by inhibition with botulinum neurotoxins A and E.
Lawrence GW; Foran P; Mohammed N; DasGupta BR; Dolly JO
Biochemistry; 1997 Mar; 36(11):3061-7. PubMed ID: 9115981
[TBL] [Abstract][Full Text] [Related]
2. Distinct exocytotic responses of intact and permeabilised chromaffin cells after cleavage of the 25-kDa synaptosomal-associated protein (SNAP-25) or synaptobrevin by botulinum toxin A or B.
Lawrence GW; Foran P; Dolly JO
Eur J Biochem; 1996 Mar; 236(3):877-86. PubMed ID: 8665909
[TBL] [Abstract][Full Text] [Related]
3. Botulinum neurotoxin C1 cleaves both syntaxin and SNAP-25 in intact and permeabilized chromaffin cells: correlation with its blockade of catecholamine release.
Foran P; Lawrence GW; Shone CC; Foster KA; Dolly JO
Biochemistry; 1996 Feb; 35(8):2630-6. PubMed ID: 8611567
[TBL] [Abstract][Full Text] [Related]
4. Differential contribution of syntaxin 1 and SNAP-25 to secretion in noradrenergic and adrenergic chromaffin cells.
Baltazar G; Tomé A; Carvalho AP; Duarte EP
Eur J Cell Biol; 2000 Dec; 79(12):883-91. PubMed ID: 11152280
[TBL] [Abstract][Full Text] [Related]
5. Dual effects of botulinum neurotoxin A on the secretory stages of chromaffin cells.
Gil A; Viniegra S; Gutiérrez LM
Eur J Neurosci; 1998 Nov; 10(11):3369-78. PubMed ID: 9824450
[TBL] [Abstract][Full Text] [Related]
6. SNAP-25 is required for a late postdocking step in Ca2+-dependent exocytosis.
Banerjee A; Kowalchyk JA; DasGupta BR; Martin TF
J Biol Chem; 1996 Aug; 271(34):20227-30. PubMed ID: 8702751
[TBL] [Abstract][Full Text] [Related]
7. Dissociation of SNAP-25 and VAMP-2 by MgATP in permeabilized adrenal chromaffin cells.
Misonou H; Nishiki T; Sekiguchi M; Takahashi M; Kamata Y; Kozaki S; Ohara-Imaizumi M; Kumakura K
Brain Res; 1996 Oct; 737(1-2):351-5. PubMed ID: 8930391
[TBL] [Abstract][Full Text] [Related]
8. Rescue of exocytosis in botulinum toxin A-poisoned chromaffin cells by expression of cleavage-resistant SNAP-25. Identification of the minimal essential C-terminal residues.
O'Sullivan GA; Mohammed N; Foran PG; Lawrence GW; Oliver Dolly J
J Biol Chem; 1999 Dec; 274(52):36897-904. PubMed ID: 10601242
[TBL] [Abstract][Full Text] [Related]
9. Ca2+-induced changes in SNAREs and synaptotagmin I correlate with triggered exocytosis from chromaffin cells: insights gleaned into the signal transduction using trypsin and botulinum toxins.
Lawrence GW; Dolly JO
J Cell Sci; 2002 Jul; 115(Pt 13):2791-800. PubMed ID: 12077369
[TBL] [Abstract][Full Text] [Related]
10. Novel therapeutics based on recombinant botulinum neurotoxins to normalize the release of transmitters and pain mediators.
Dolly JO; Wang J; Zurawski TH; Meng J
FEBS J; 2011 Dec; 278(23):4454-66. PubMed ID: 21645262
[TBL] [Abstract][Full Text] [Related]
11. Proteolysis of SNAP-25 isoforms by botulinum neurotoxin types A, C, and E: domains and amino acid residues controlling the formation of enzyme-substrate complexes and cleavage.
Vaidyanathan VV; Yoshino K; Jahnz M; Dörries C; Bade S; Nauenburg S; Niemann H; Binz T
J Neurochem; 1999 Jan; 72(1):327-37. PubMed ID: 9886085
[TBL] [Abstract][Full Text] [Related]
12. Uptake of botulinum neurotoxin into cultured neurons.
Keller JE; Cai F; Neale EA
Biochemistry; 2004 Jan; 43(2):526-32. PubMed ID: 14717608
[TBL] [Abstract][Full Text] [Related]
13. The sensitivity of catecholamine release to botulinum toxin C1 and E suggests selective targeting of vesicles set into the readily releasable pool.
Stigliani S; Raiteri L; Fassio A; Bonanno G
J Neurochem; 2003 Apr; 85(2):409-21. PubMed ID: 12675917
[TBL] [Abstract][Full Text] [Related]
14. Fusion of Golgi-derived vesicles mediated by SNAP-25 is essential for sympathetic neuron outgrowth but relatively insensitive to botulinum neurotoxins in vitro.
Lawrence GW; Wang J; Brin MF; Aoki KR; Wheeler L; Dolly JO
FEBS J; 2014 Jul; 281(14):3243-60. PubMed ID: 24863955
[TBL] [Abstract][Full Text] [Related]
15. A peptide that mimics the C-terminal sequence of SNAP-25 inhibits secretory vesicle docking in chromaffin cells.
Gutierrez LM; Viniegra S; Rueda J; Ferrer-Montiel AV; Canaves JM; Montal M
J Biol Chem; 1997 Jan; 272(5):2634-9. PubMed ID: 9006897
[TBL] [Abstract][Full Text] [Related]
16. Truncated SNAP-25 (1-197), like botulinum neurotoxin A, can inhibit insulin secretion from HIT-T15 insulinoma cells.
Huang X; Wheeler MB; Kang YH; Sheu L; Lukacs GL; Trimble WS; Gaisano HY
Mol Endocrinol; 1998 Jul; 12(7):1060-70. PubMed ID: 9658409
[TBL] [Abstract][Full Text] [Related]
17. Presynaptic protein interactions in vivo: evidence from botulinum A, C, D and E action at frog neuromuscular junction.
Raciborska DA; Trimble WS; Charlton MP
Eur J Neurosci; 1998 Aug; 10(8):2617-28. PubMed ID: 9767392
[TBL] [Abstract][Full Text] [Related]
18. Adrenal chromaffin cells contain functionally different SNAP-25 monomers and SNAP-25/syntaxin heterodimers.
Höhne-Zell B; Gratzl M
FEBS Lett; 1996 Sep; 394(2):109-16. PubMed ID: 8843145
[TBL] [Abstract][Full Text] [Related]
19. Enhancement of the endopeptidase activity of purified botulinum neurotoxins A and E by an isolated component of the native neurotoxin associated proteins.
Sharma SK; Singh BR
Biochemistry; 2004 Apr; 43(16):4791-8. PubMed ID: 15096048
[TBL] [Abstract][Full Text] [Related]
20. Persistence of botulinum neurotoxin action in cultured spinal cord cells.
Keller JE; Neale EA; Oyler G; Adler M
FEBS Lett; 1999 Jul; 456(1):137-42. PubMed ID: 10452545
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]