These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

183 related articles for article (PubMed ID: 9133255)

  • 1. Lens lipid peroxides and glutathione concentrations in diabetic cataract.
    Ozmen D; Mutaf I; Ozmen B; Mentes J; Bayindir O
    Ann Clin Biochem; 1997 Mar; 34 ( Pt 2)():190-2. PubMed ID: 9133255
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Blood and lens lipid peroxidation and antioxidant status in normal individuals, senile and diabetic cataractous patients.
    Donma O; Yorulmaz E; Pekel H; Suyugül N
    Curr Eye Res; 2002 Jul; 25(1):9-16. PubMed ID: 12518238
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Lipid peroxide and reactive oxygen species generating systems of the crystalline lens.
    Babizhayev MA; Costa EB
    Biochim Biophys Acta; 1994 Feb; 1225(3):326-37. PubMed ID: 8312381
    [TBL] [Abstract][Full Text] [Related]  

  • 4. [The oxidative stress in the cataract formation].
    Obara Y
    Nippon Ganka Gakkai Zasshi; 1995 Dec; 99(12):1303-41. PubMed ID: 8571853
    [TBL] [Abstract][Full Text] [Related]  

  • 5. [Parameters of oxidative stress in the lens, aqueous humor and blood in patients with diabetes and senile cataracts].
    Zorić L
    Srp Arh Celok Lek; 2003; 131(3-4):137-42. PubMed ID: 14608877
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Potentiation of intraocular absorption and drug metabolism of N-acetylcarnosine lubricant eye drops: drug interaction with sight threatening lipid peroxides in the treatment for age-related eye diseases.
    Babizhayev MA
    Drug Metabol Drug Interact; 2009; 24(2-4):275-323. PubMed ID: 20408504
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Mitochondria induce oxidative stress, generation of reactive oxygen species and redox state unbalance of the eye lens leading to human cataract formation: disruption of redox lens organization by phospholipid hydroperoxides as a common basis for cataract disease.
    Babizhayev MA
    Cell Biochem Funct; 2011 Apr; 29(3):183-206. PubMed ID: 21381059
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Ultrasonic and biochemical evaluation of human diabetic lens.
    Raitelaitiene R; Paunksnis A; Ivanov L; Kurapkiene S
    Medicina (Kaunas); 2005; 41(8):641-8. PubMed ID: 16160411
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Failure to withstand oxidative stress induced by phospholipid hydroperoxides as a possible cause of the lens opacities in systemic diseases and ageing.
    Babizhayev MA
    Biochim Biophys Acta; 1996 Mar; 1315(2):87-99. PubMed ID: 8608175
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Reduced glutathione levels in senile cataractous lens epithelial cells.
    Sueno T; Bando M; Obazawa H
    Tokai J Exp Clin Med; 1992 May; 17(1):5-9. PubMed ID: 1523693
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Accumulation of the hydroxyl free radical markers meta-, ortho-tyrosine and DOPA in cataractous lenses is accompanied by a lower protein and phenylalanine content of the water-soluble phase.
    Molnár GA; Nemes V; Biró Z; Ludány A; Wagner Z; Wittmann I
    Free Radic Res; 2005 Dec; 39(12):1359-66. PubMed ID: 16298866
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Lens superoxide dismutase and catalase activities in diabetic cataract.
    Ozmen B; Ozmen D; Erkin E; Güner I; Habif S; Bayindir O
    Clin Biochem; 2002 Feb; 35(1):69-72. PubMed ID: 11937081
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Human diabetic cataract: role of lipid peroxidation.
    Altomare E; Vendemiale G; Grattagliano I; Angelini P; Micelli-Ferrari T; Cardia L
    Diabete Metab; 1995 Jun; 21(3):173-9. PubMed ID: 7556808
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Antioxidant markers in human senile and diabetic cataractous lenses.
    Hashim Z; Zarina S
    J Coll Physicians Surg Pak; 2006 Oct; 16(10):637-40. PubMed ID: 17007750
    [TBL] [Abstract][Full Text] [Related]  

  • 15. The decrease of free epsilon-amino groups in senile and diabetic cataracts.
    Simonelli F; Cotticelli L; Iura A; Manna C; Nesti A; Rinaldi E; Auricchio G
    Ophthalmic Res; 1990; 22(3):160-5. PubMed ID: 2385432
    [TBL] [Abstract][Full Text] [Related]  

  • 16. [Accumulation of lipid peroxidation products in cataractous lenses].
    Babizhaev MA; Shvedova AA; Arkhipenko IuV; Kagan VE
    Biull Eksp Biol Med; 1985 Sep; 100(9):299-301. PubMed ID: 4041596
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Beneficial influence of fungal metabolite nigerloxin on eye lens abnormalities in experimental diabetes.
    Suresha BS; Sattur AP; Srinivasan K
    Can J Physiol Pharmacol; 2012 Apr; 90(4):387-94. PubMed ID: 22423974
    [TBL] [Abstract][Full Text] [Related]  

  • 18. [Reduced glutathione level and GSH-dependent enzyme activities in corticonuclear blocks of lenses in patients with senile cataract].
    Kisić B; Mirić D; Žorić L; Ilić A; Dragojević I
    Srp Arh Celok Lek; 2012; 140(9-10):563-70. PubMed ID: 23289270
    [TBL] [Abstract][Full Text] [Related]  

  • 19. H2O2-dependent NADH oxidation activity in senile cataractous human lens: its relation to glutathione redox cycle.
    Bando M; Obazawa H
    Jpn J Ophthalmol; 1990; 34(2):188-95. PubMed ID: 2214362
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Changes in the lens epithelium with respect to cataractogenesis: light microscopic and Scheimpflug densitometric analysis of the cataractous and the clear lens of diabetics and non-diabetics.
    Tkachov SI; Lautenschläger C; Ehrich D; Struck HG
    Graefes Arch Clin Exp Ophthalmol; 2006 May; 244(5):596-602. PubMed ID: 16175371
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 10.