153 related articles for article (PubMed ID: 9216088)
1. The toxicity of the Alzheimer's beta-amyloid peptide correlates with a distinct fiber morphology.
Seilheimer B; Bohrmann B; Bondolfi L; Müller F; Stüber D; Döbeli H
J Struct Biol; 1997 Jun; 119(1):59-71. PubMed ID: 9216088
[TBL] [Abstract][Full Text] [Related]
2. Toxicity of non-abeta component of Alzheimer's disease amyloid, and N-terminal fragments thereof, correlates to formation of beta-sheet structure and fibrils.
Bodles AM; Guthrie DJ; Harriott P; Campbell P; Irvine GB
Eur J Biochem; 2000 Apr; 267(8):2186-94. PubMed ID: 10759841
[TBL] [Abstract][Full Text] [Related]
3. Inhibition of toxicity in the beta-amyloid peptide fragment beta -(25-35) using N-methylated derivatives: a general strategy to prevent amyloid formation.
Hughes E; Burke RM; Doig AJ
J Biol Chem; 2000 Aug; 275(33):25109-15. PubMed ID: 10825171
[TBL] [Abstract][Full Text] [Related]
4. N-Methylated peptide inhibitors of beta-amyloid aggregation and toxicity. Optimization of the inhibitor structure.
Kokkoni N; Stott K; Amijee H; Mason JM; Doig AJ
Biochemistry; 2006 Aug; 45(32):9906-18. PubMed ID: 16893191
[TBL] [Abstract][Full Text] [Related]
5. Cytotoxic amyloid peptides inhibit cellular 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT) reduction by enhancing MTT formazan exocytosis.
Liu Y; Schubert D
J Neurochem; 1997 Dec; 69(6):2285-93. PubMed ID: 9375659
[TBL] [Abstract][Full Text] [Related]
6. Beta-amyloid-induced cell toxicity: enhancement of 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide-dependent cell death.
Hertel C; Hauser N; Schubenel R; Seilheimer B; Kemp JA
J Neurochem; 1996 Jul; 67(1):272-6. PubMed ID: 8667002
[TBL] [Abstract][Full Text] [Related]
7. The intracellular component of cellular 3-(4,5-dimethylthiazol-2-yl)-2, 5-diphenyltetrazolium bromide (MTT) reduction is specifically inhibited by beta-amyloid peptides.
Shearman MS; Hawtin SR; Tailor VJ
J Neurochem; 1995 Jul; 65(1):218-27. PubMed ID: 7790863
[TBL] [Abstract][Full Text] [Related]
8. Cellular MTT reduction distinguishes the mechanism of action of beta-amyloid from that of tachykinin receptor peptides.
Shearman MS
Neuropeptides; 1996 Apr; 30(2):125-32. PubMed ID: 8771554
[TBL] [Abstract][Full Text] [Related]
9. Methionine oxidation reduces lag-times for amyloid-β(1-40) fiber formation but generates highly fragmented fibers.
Gu M; Viles JH
Biochim Biophys Acta; 2016 Sep; 1864(9):1260-1269. PubMed ID: 27108954
[TBL] [Abstract][Full Text] [Related]
10. Strong inhibition of beta-amyloid peptide aggregation realized by two-steps evolved peptides.
Ghimire Gautam S; Komatsu M; Nishigaki K
Chem Biol Drug Des; 2015 Mar; 85(3):356-68. PubMed ID: 25082146
[TBL] [Abstract][Full Text] [Related]
11. Inhibitors of V-type ATPases, bafilomycin A1 and concanamycin A, protect against beta-amyloid-mediated effects on 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT) reduction.
Kane MD; Schwarz RD; St Pierre L; Watson MD; Emmerling MR; Boxer PA; Walker GK
J Neurochem; 1999 May; 72(5):1939-47. PubMed ID: 10217271
[TBL] [Abstract][Full Text] [Related]
12. Amyloid-like fibril formation by tachykinin neuropeptides and its relevance to amyloid β-protein aggregation and toxicity.
Singh PK; Maji SK
Cell Biochem Biophys; 2012 Sep; 64(1):29-44. PubMed ID: 22628076
[TBL] [Abstract][Full Text] [Related]
13. A biotechnological method provides access to aggregation competent monomeric Alzheimer's 1-42 residue amyloid peptide.
Döbeli H; Draeger N; Huber G; Jakob P; Schmidt D; Seilheimer B; Stüber D; Wipf B; Zulauf M
Biotechnology (N Y); 1995 Sep; 13(9):988-93. PubMed ID: 9636276
[TBL] [Abstract][Full Text] [Related]
14. C-Terminal Fragment, Aβ32-37, Analogues Protect Against Aβ Aggregation-Induced Toxicity.
Bansal S; Maurya IK; Yadav N; Thota CK; Kumar V; Tikoo K; Chauhan VS; Jain R
ACS Chem Neurosci; 2016 May; 7(5):615-23. PubMed ID: 26835536
[TBL] [Abstract][Full Text] [Related]
15. Identification of the region of non-Abeta component (NAC) of Alzheimer's disease amyloid responsible for its aggregation and toxicity.
Bodles AM; Guthrie DJ; Greer B; Irvine GB
J Neurochem; 2001 Jul; 78(2):384-95. PubMed ID: 11461974
[TBL] [Abstract][Full Text] [Related]
16. Acidic pH promotes the formation of toxic fibrils from beta-amyloid peptide.
Su Y; Chang PT
Brain Res; 2001 Mar; 893(1-2):287-91. PubMed ID: 11223020
[TBL] [Abstract][Full Text] [Related]
17. Inhibition of PC12 cell redox activity is a specific, early indicator of the mechanism of beta-amyloid-mediated cell death.
Shearman MS; Ragan CI; Iversen LL
Proc Natl Acad Sci U S A; 1994 Feb; 91(4):1470-4. PubMed ID: 8108433
[TBL] [Abstract][Full Text] [Related]
18. Beta-amyloid (1-42) affects MTT reduction in astrocytes: implications for vesicular trafficking and cell functionality.
Kerokoski P; Soininen H; Pirttilä T
Neurochem Int; 2001 Feb; 38(2):127-34. PubMed ID: 11137881
[TBL] [Abstract][Full Text] [Related]
19. Amyloid beta-peptide-induced inhibition of MTT reduction in PC12h and C1300 neuroblastoma cells: effect of nitroprusside.
Takenouchi T; Munekata E
Peptides; 1998; 19(2):365-72. PubMed ID: 9493870
[TBL] [Abstract][Full Text] [Related]
20. Amyloid toxicity is independent of polypeptide sequence, length and chirality.
Pastor MT; Kümmerer N; Schubert V; Esteras-Chopo A; Dotti CG; López de la Paz M; Serrano L
J Mol Biol; 2008 Jan; 375(3):695-707. PubMed ID: 18036611
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
