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9. Suppression of insulitis in non-obese diabetic (NOD) mice by oral insulin administration is associated with selective expression of interleukin-4 and -10, transforming growth factor-beta, and prostaglandin-E. Hancock WW; Polanski M; Zhang J; Blogg N; Weiner HL Am J Pathol; 1995 Nov; 147(5):1193-9. PubMed ID: 7485382 [TBL] [Abstract][Full Text] [Related]
10. Induction of diabetes in nonobese diabetic mice by Th2 T cell clones from a TCR transgenic mouse. Poulin M; Haskins K J Immunol; 2000 Mar; 164(6):3072-8. PubMed ID: 10706696 [TBL] [Abstract][Full Text] [Related]
11. Differential expression of ICAM-1 and LFA-1 versus L-selectin and VCAM-1 in autoimmune insulitis of NOD mice and association with both Th1- and Th2-type infiltrates. Martin S; Hibino T; Faust A; Kleemann R; Kolb H J Autoimmun; 1996 Oct; 9(5):637-43. PubMed ID: 8933279 [TBL] [Abstract][Full Text] [Related]
12. IL-18 inhibits diabetes development in nonobese diabetic mice by counterregulation of Th1-dependent destructive insulitis. Rothe H; Hausmann A; Casteels K; Okamura H; Kurimoto M; Burkart V; Mathieu C; Kolb H J Immunol; 1999 Aug; 163(3):1230-6. PubMed ID: 10415018 [TBL] [Abstract][Full Text] [Related]
13. Local expression of transgene encoded TNF alpha in islets prevents autoimmune diabetes in nonobese diabetic (NOD) mice by preventing the development of auto-reactive islet-specific T cells. Grewal IS; Grewal KD; Wong FS; Picarella DE; Janeway CA; Flavell RA J Exp Med; 1996 Nov; 184(5):1963-74. PubMed ID: 8920883 [TBL] [Abstract][Full Text] [Related]
14. Islet-specific Th1, but not Th2, cells secrete multiple chemokines and promote rapid induction of autoimmune diabetes. Bradley LM; Asensio VC; Schioetz LK; Harbertson J; Krahl T; Patstone G; Woolf N; Campbell IL; Sarvetnick N J Immunol; 1999 Mar; 162(5):2511-20. PubMed ID: 10072490 [TBL] [Abstract][Full Text] [Related]
15. Contribution of T cells to the development of autoimmune diabetes in the NOD mouse model. Toyoda H; Formby B Bioessays; 1998 Sep; 20(9):750-7. PubMed ID: 9819564 [TBL] [Abstract][Full Text] [Related]
16. Prevention of adoptively transferred diabetes in nonobese diabetic mice with IL-10-transduced islet-specific Th1 lymphocytes. A gene therapy model for autoimmune diabetes. Moritani M; Yoshimoto K; Ii S; Kondo M; Iwahana H; Yamaoka T; Sano T; Nakano N; Kikutani H; Itakura M J Clin Invest; 1996 Oct; 98(8):1851-9. PubMed ID: 8878437 [TBL] [Abstract][Full Text] [Related]
17. Production of interleukin 10 by islet cells accelerates immune-mediated destruction of beta cells in nonobese diabetic mice. Wogensen L; Lee MS; Sarvetnick N J Exp Med; 1994 Apr; 179(4):1379-84. PubMed ID: 8145050 [TBL] [Abstract][Full Text] [Related]
18. IL-1alpha, IL-1beta, and IFN-gamma mark beta cells for Fas-dependent destruction by diabetogenic CD4(+) T lymphocytes. Amrani A; Verdaguer J; Thiessen S; Bou S; Santamaria P J Clin Invest; 2000 Feb; 105(4):459-68. PubMed ID: 10683375 [TBL] [Abstract][Full Text] [Related]
19. Absence of significant Th2 response in diabetes-prone non-obese diabetic (NOD) mice. Hartemann-Heurtier A; Richard MF; Boitard C Clin Exp Immunol; 1999 May; 116(2):225-30. PubMed ID: 10337011 [TBL] [Abstract][Full Text] [Related]
20. Abrogation of autoimmune diabetes in nonobese diabetic mice and protection against effector lymphocytes by transgenic paracrine TGF-beta1. Moritani M; Yoshimoto K; Wong SF; Tanaka C; Yamaoka T; Sano T; Komagata Y; Miyazaki J; Kikutani H; Itakura M J Clin Invest; 1998 Aug; 102(3):499-506. PubMed ID: 9691086 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]