103 related articles for article (PubMed ID: 9400614)
1. A point mutation in the Sendai virus accessory C proteins attenuates virulence for mice, but not virus growth in cell culture.
Garcin D; Itoh M; Kolakofsky D
Virology; 1997 Nov; 238(2):424-31. PubMed ID: 9400614
[TBL] [Abstract][Full Text] [Related]
2. Functional significance of alternate phosphorylation in Sendai virus P protein.
Hu Cj; Gupta KC
Virology; 2000 Mar; 268(2):517-32. PubMed ID: 10704359
[TBL] [Abstract][Full Text] [Related]
3. Sendai viruses with altered P, V, and W protein expression.
Delenda C; Taylor G; Hausmann S; Garcin D; Kolakofsky D
Virology; 1998 Mar; 242(2):327-37. PubMed ID: 9514977
[TBL] [Abstract][Full Text] [Related]
4. Role of primary constitutive phosphorylation of Sendai virus P and V proteins in viral replication and pathogenesis.
Hu CJ; Kato A; Bowman MC; Kiyotani K; Yoshida T; Moyer SA; Nagai Y; Gupta KC
Virology; 1999 Oct; 263(1):195-208. PubMed ID: 10544094
[TBL] [Abstract][Full Text] [Related]
5. Mutations in conserved domain II of the large (L) subunit of the Sendai virus RNA polymerase abolish RNA synthesis.
Smallwood S; Easson CD; Feller JA; Horikami SM; Moyer SA
Virology; 1999 Sep; 262(2):375-83. PubMed ID: 10502516
[TBL] [Abstract][Full Text] [Related]
6. Sendai virus C proteins are categorically nonessential gene products but silencing their expression severely impairs viral replication and pathogenesis.
Kurotani A; Kiyotani K; Kato A; Shioda T; Sakai Y; Mizumoto K; Yoshida T; Nagai Y
Genes Cells; 1998 Feb; 3(2):111-24. PubMed ID: 9605405
[TBL] [Abstract][Full Text] [Related]
7. V and C proteins of measles virus function as virulence factors in vivo.
Patterson JB; Thomas D; Lewicki H; Billeter MA; Oldstone MB
Virology; 2000 Feb; 267(1):80-9. PubMed ID: 10648185
[TBL] [Abstract][Full Text] [Related]
8. The paramyxovirus, Sendai virus, V protein encodes a luxury function required for viral pathogenesis.
Kato A; Kiyotani K; Sakai Y; Yoshida T; Nagai Y
EMBO J; 1997 Feb; 16(3):578-87. PubMed ID: 9034340
[TBL] [Abstract][Full Text] [Related]
9. Phosphorylation of the Sendai virus M protein is not essential for virus replication either in vitro or in vivo.
Sakaguchi T; Kiyotani K; Kato A; Asakawa M; Fujii Y; Nagai Y; Yoshida T
Virology; 1997 Sep; 235(2):360-6. PubMed ID: 9281516
[TBL] [Abstract][Full Text] [Related]
10. Mutations in domain V of the Sendai virus L polymerase protein uncouple transcription and replication and differentially affect replication in vitro and in vivo.
Cortese CK; Feller JA; Moyer SA
Virology; 2000 Nov; 277(2):387-96. PubMed ID: 11080486
[TBL] [Abstract][Full Text] [Related]
11. Sendai virus wild-type and mutant C proteins show a direct correlation between L polymerase binding and inhibition of viral RNA synthesis.
Grogan CC; Moyer SA
Virology; 2001 Sep; 288(1):96-108. PubMed ID: 11543662
[TBL] [Abstract][Full Text] [Related]
12. Paramyxovirus Sendai virus V protein counteracts innate virus clearance through IRF-3 activation, but not via interferon, in mice.
Kiyotani K; Sakaguchi T; Kato A; Nagai Y; Yoshida T
Virology; 2007 Mar; 359(1):82-91. PubMed ID: 17027894
[TBL] [Abstract][Full Text] [Related]
13. The YLDL sequence within Sendai virus M protein is critical for budding of virus-like particles and interacts with Alix/AIP1 independently of C protein.
Irie T; Shimazu Y; Yoshida T; Sakaguchi T
J Virol; 2007 Mar; 81(5):2263-73. PubMed ID: 17166905
[TBL] [Abstract][Full Text] [Related]
14. The L polymerase protein of parainfluenza virus 3 forms an oligomer and can interact with the heterologous Sendai virus L, P and C proteins.
Smallwood S; Moyer SA
Virology; 2004 Jan; 318(1):439-50. PubMed ID: 14972569
[TBL] [Abstract][Full Text] [Related]
15. Mutations in the C, D, and V open reading frames of human parainfluenza virus type 3 attenuate replication in rodents and primates.
Durbin AP; McAuliffe JM; Collins PL; Murphy BR
Virology; 1999 Sep; 261(2):319-30. PubMed ID: 10497117
[TBL] [Abstract][Full Text] [Related]
16. A herpes simplex virus type 1 mutant deleted for gamma34.5 and LAT kills glioma cells in vitro and is inhibited for in vivo reactivation.
Samoto K; Perng GC; Ehtesham M; Liu Y; Wechsler SL; Nesburn AB; Black KL; Yu JS
Cancer Gene Ther; 2001 Apr; 8(4):269-77. PubMed ID: 11393279
[TBL] [Abstract][Full Text] [Related]
17. A single nucleotide substitution in the transcription start signal of the M2 gene of respiratory syncytial virus vaccine candidate cpts248/404 is the major determinant of the temperature-sensitive and attenuation phenotypes.
Whitehead SS; Firestone CY; Collins PL; Murphy BR
Virology; 1998 Aug; 247(2):232-9. PubMed ID: 9705916
[TBL] [Abstract][Full Text] [Related]
18. Attenuation of a field Sendai virus isolate through egg-passages is associated with an impediment of viral genome replication in mouse respiratory cells.
Kiyotani K; Sakaguchi T; Fujii Y; Yoshida T
Arch Virol; 2001; 146(5):893-908. PubMed ID: 11448028
[TBL] [Abstract][Full Text] [Related]
19. Deletion and substitution analysis defines regions and residues within the phosphoprotein of bovine respiratory syncytial virus that affect transcription, RNA replication, and interaction with the nucleoprotein.
Khattar SK; Yunus AS; Collins PL; Samal SK
Virology; 2001 Jul; 285(2):253-69. PubMed ID: 11437660
[TBL] [Abstract][Full Text] [Related]
20. Recombinant Sendai viruses with L1618V mutation in their L polymerase protein establish persistent infection, but not temperature sensitivity.
Nishio M; Nagata A; Tsurudome M; Ito M; Kawano M; Komada H; Ito Y
Virology; 2004 Nov; 329(2):289-301. PubMed ID: 15518809
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
