177 related articles for article (PubMed ID: 9407116)
1. The Src family kinase Hck interacts with Bcr-Abl by a kinase-independent mechanism and phosphorylates the Grb2-binding site of Bcr.
Warmuth M; Bergmann M; Priess A; Häuslmann K; Emmerich B; Hallek M
J Biol Chem; 1997 Dec; 272(52):33260-70. PubMed ID: 9407116
[TBL] [Abstract][Full Text] [Related]
2. The interaction of the Bcr-Abl tyrosine kinase with the Src kinase Hck is mediated by multiple binding domains.
Stanglmaier M; Warmuth M; Kleinlein I; Reis S; Hallek M
Leukemia; 2003 Feb; 17(2):283-9. PubMed ID: 12592324
[TBL] [Abstract][Full Text] [Related]
3. Tyrosine phosphorylation of p120cbl in BCR/abl transformed hematopoietic cells mediates enhanced association with phosphatidylinositol 3-kinase.
Jain SK; Langdon WY; Varticovski L
Oncogene; 1997 May; 14(18):2217-28. PubMed ID: 9174058
[TBL] [Abstract][Full Text] [Related]
4. The Src family kinase Hck couples BCR/ABL to STAT5 activation in myeloid leukemia cells.
Klejman A; Schreiner SJ; Nieborowska-Skorska M; Slupianek A; Wilson M; Smithgall TE; Skorski T
EMBO J; 2002 Nov; 21(21):5766-74. PubMed ID: 12411494
[TBL] [Abstract][Full Text] [Related]
5. Coupling between p210bcr-abl and Shc and Grb2 adaptor proteins in hematopoietic cells permits growth factor receptor-independent link to ras activation pathway.
Tauchi T; Boswell HS; Leibowitz D; Broxmeyer HE
J Exp Med; 1994 Jan; 179(1):167-75. PubMed ID: 7505797
[TBL] [Abstract][Full Text] [Related]
6. Transformation of myeloid leukemia cells to cytokine independence by Bcr-Abl is suppressed by kinase-defective Hck.
Lionberger JM; Wilson MB; Smithgall TE
J Biol Chem; 2000 Jun; 275(24):18581-5. PubMed ID: 10849448
[TBL] [Abstract][Full Text] [Related]
7. Coexistence of phosphotyrosine-dependent and -independent interactions between Cbl and Bcr-Abl.
Gaston I; Johnson KJ; Oda T; Bhat A; Reis M; Langdon W; Shen L; Deininger MW; Druker BJ
Exp Hematol; 2004 Jan; 32(1):113-21. PubMed ID: 14725908
[TBL] [Abstract][Full Text] [Related]
8. Mutant forms of growth factor-binding protein-2 reverse BCR-ABL-induced transformation.
Gishizky ML; Cortez D; Pendergast AM
Proc Natl Acad Sci U S A; 1995 Nov; 92(24):10889-93. PubMed ID: 7479904
[TBL] [Abstract][Full Text] [Related]
9. Bcr phosphorylated on tyrosine 177 binds Grb2.
Ma G; Lu D; Wu Y; Liu J; Arlinghaus RB
Oncogene; 1997 May; 14(19):2367-72. PubMed ID: 9178913
[TBL] [Abstract][Full Text] [Related]
10. Tyrosine phosphorylation of Grb2 by Bcr/Abl and epidermal growth factor receptor: a novel regulatory mechanism for tyrosine kinase signaling.
Li S; Couvillon AD; Brasher BB; Van Etten RA
EMBO J; 2001 Dec; 20(23):6793-804. PubMed ID: 11726515
[TBL] [Abstract][Full Text] [Related]
11. Structural and signaling requirements for BCR-ABL-mediated transformation and inhibition of apoptosis.
Cortez D; Kadlec L; Pendergast AM
Mol Cell Biol; 1995 Oct; 15(10):5531-41. PubMed ID: 7565705
[TBL] [Abstract][Full Text] [Related]
12. The Grb2 binding site is required for the induction of chronic myeloid leukemia-like disease in mice by the Bcr/Abl tyrosine kinase.
Million RP; Van Etten RA
Blood; 2000 Jul; 96(2):664-70. PubMed ID: 10887132
[TBL] [Abstract][Full Text] [Related]
13. Phosphorylation-dependent interactions between ADAM15 cytoplasmic domain and Src family protein-tyrosine kinases.
Poghosyan Z; Robbins SM; Houslay MD; Webster A; Murphy G; Edwards DR
J Biol Chem; 2002 Feb; 277(7):4999-5007. PubMed ID: 11741929
[TBL] [Abstract][Full Text] [Related]
14. BCR-ABL-induced oncogenesis is mediated by direct interaction with the SH2 domain of the GRB-2 adaptor protein.
Pendergast AM; Quilliam LA; Cripe LD; Bassing CH; Dai Z; Li N; Batzer A; Rabun KM; Der CJ; Schlessinger J
Cell; 1993 Oct; 75(1):175-85. PubMed ID: 8402896
[TBL] [Abstract][Full Text] [Related]
15. Phosphorylation of Cbl following stimulation with interleukin-3 and its association with Grb2, Fyn, and phosphatidylinositol 3-kinase.
Anderson SM; Burton EA; Koch BL
J Biol Chem; 1997 Jan; 272(2):739-45. PubMed ID: 8995358
[TBL] [Abstract][Full Text] [Related]
16. Global phosphoproteomics reveals crosstalk between Bcr-Abl and negative feedback mechanisms controlling Src signaling.
Rubbi L; Titz B; Brown L; Galvan E; Komisopoulou E; Chen SS; Low T; Tahmasian M; Skaggs B; Müschen M; Pellegrini M; Graeber TG
Sci Signal; 2011 Mar; 4(166):ra18. PubMed ID: 21447799
[TBL] [Abstract][Full Text] [Related]
17. An inhibitor-resistant mutant of Hck protects CML cells against the antiproliferative and apoptotic effects of the broad-spectrum Src family kinase inhibitor A-419259.
Pene-Dumitrescu T; Peterson LF; Donato NJ; Smithgall TE
Oncogene; 2008 Nov; 27(56):7055-69. PubMed ID: 18794796
[TBL] [Abstract][Full Text] [Related]
18. Bcr-Abl oncoproteins bind directly to activators of the Ras signalling pathway.
Puil L; Liu J; Gish G; Mbamalu G; Bowtell D; Pelicci PG; Arlinghaus R; Pawson T
EMBO J; 1994 Feb; 13(4):764-73. PubMed ID: 8112292
[TBL] [Abstract][Full Text] [Related]
19. The SH2-containing adapter protein GRB10 interacts with BCR-ABL.
Bai RY; Jahn T; Schrem S; Munzert G; Weidner KM; Wang JY; Duyster J
Oncogene; 1998 Aug; 17(8):941-8. PubMed ID: 9747873
[TBL] [Abstract][Full Text] [Related]
20. Src family kinases mediate cytoplasmic retention of activated STAT5 in BCR-ABL-positive cells.
Chatain N; Ziegler P; Fahrenkamp D; Jost E; Moriggl R; Schmitz-Van de Leur H; Müller-Newen G
Oncogene; 2013 Aug; 32(31):3587-97. PubMed ID: 22926520
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
