144 related articles for article (PubMed ID: 9447234)
1. A difference in the pattern of repair in a large genomic region in UV-irradiated normal human and Cockayne syndrome cells.
Shanower GA; Kantor GJ
Mutat Res; 1997 Nov; 385(2):127-37. PubMed ID: 9447234
[TBL] [Abstract][Full Text] [Related]
2. Repair of some active genes in Cockayne syndrome cells is at the genome overall rate.
Kantor GJ; Bastin SA
Mutat Res; 1995 May; 336(3):223-33. PubMed ID: 7739610
[TBL] [Abstract][Full Text] [Related]
3. Ultraviolet-sensitive syndrome cells are defective in transcription-coupled repair of cyclobutane pyrimidine dimers.
Spivak G; Itoh T; Matsunaga T; Nikaido O; Hanawalt P; Yamaizumi M
DNA Repair (Amst); 2002 Aug; 1(8):629-43. PubMed ID: 12509286
[TBL] [Abstract][Full Text] [Related]
4. Selective repair of specific chromatin domains in UV-irradiated cells from xeroderma pigmentosum complementation group C.
Kantor GJ; Barsalou LS; Hanawalt PC
Mutat Res; 1990 May; 235(3):171-80. PubMed ID: 2342504
[TBL] [Abstract][Full Text] [Related]
5. Preferential repair of ionizing radiation-induced damage in the transcribed strand of an active human gene is defective in Cockayne syndrome.
Leadon SA; Cooper PK
Proc Natl Acad Sci U S A; 1993 Nov; 90(22):10499-503. PubMed ID: 8248136
[TBL] [Abstract][Full Text] [Related]
6. Evidence for defective repair of cyclobutane pyrimidine dimers with normal repair of other DNA photoproducts in a transcriptionally active gene transfected into Cockayne syndrome cells.
Barrett SF; Robbins JH; Tarone RE; Kraemer KH
Mutat Res; 1991 Nov; 255(3):281-91. PubMed ID: 1719400
[TBL] [Abstract][Full Text] [Related]
7. DNA repair in the genomic region containing the beta-actin gene in xeroderma pigmentosum complementation group C and normal human cells.
Barsalou LS; Kantor GJ; Deiss DM; Hall CE
Mutat Res; 1994 Jul; 315(1):43-54. PubMed ID: 7517010
[TBL] [Abstract][Full Text] [Related]
8. Cells from XP-D and XP-D-CS patients exhibit equally inefficient repair of UV-induced damage in transcribed genes but different capacity to recover UV-inhibited transcription.
van Hoffen A; Kalle WH; de Jong-Versteeg A; Lehmann AR; van Zeeland AA; Mullenders LH
Nucleic Acids Res; 1999 Jul; 27(14):2898-904. PubMed ID: 10390531
[TBL] [Abstract][Full Text] [Related]
9. Nuclear matrix associated DNA is preferentially repaired in normal human fibroblasts, exposed to a low dose of ultraviolet light but not in Cockayne's syndrome fibroblasts.
Mullenders LH; van Kesteren van Leeuwen AC; van Zeeland AA; Natarajan AT
Nucleic Acids Res; 1988 Nov; 16(22):10607-22. PubMed ID: 3205718
[TBL] [Abstract][Full Text] [Related]
10. Transient expression of a plasmid gene, a tool to study DNA repair in human cells: defect of DNA repair in Cockayne syndrome; one thymine cyclobutane dimer is sufficient to block transcription.
Klocker H; Schneider R; Burtscher HJ; Auer B; Hirsch-Kauffmann M; Schweiger M
Eur J Cell Biol; 1986 Jan; 39(2):346-51. PubMed ID: 3956512
[TBL] [Abstract][Full Text] [Related]
11. The genetic defect in Cockayne syndrome is associated with a defect in repair of UV-induced DNA damage in transcriptionally active DNA.
Venema J; Mullenders LH; Natarajan AT; van Zeeland AA; Mayne LV
Proc Natl Acad Sci U S A; 1990 Jun; 87(12):4707-11. PubMed ID: 2352945
[TBL] [Abstract][Full Text] [Related]
12. Xeroderma pigmentosum complementation group C cells remove pyrimidine dimers selectively from the transcribed strand of active genes.
Venema J; van Hoffen A; Karcagi V; Natarajan AT; van Zeeland AA; Mullenders LH
Mol Cell Biol; 1991 Aug; 11(8):4128-34. PubMed ID: 1649389
[TBL] [Abstract][Full Text] [Related]
13. UV-enhanced reactivation of a UV-damaged reporter gene suggests transcription-coupled repair is UV-inducible in human cells.
Francis MA; Rainbow AJ
Carcinogenesis; 1999 Jan; 20(1):19-26. PubMed ID: 9934845
[TBL] [Abstract][Full Text] [Related]
14. Transcription-coupled repair removes both cyclobutane pyrimidine dimers and 6-4 photoproducts with equal efficiency and in a sequential way from transcribed DNA in xeroderma pigmentosum group C fibroblasts.
van Hoffen A; Venema J; Meschini R; van Zeeland AA; Mullenders LH
EMBO J; 1995 Jan; 14(2):360-7. PubMed ID: 7835346
[TBL] [Abstract][Full Text] [Related]
15. Clustered sites of DNA repair synthesis during early nucleotide excision repair in ultraviolet light-irradiated quiescent human fibroblasts.
Svetlova M; Solovjeva L; Pleskach N; Yartseva N; Yakovleva T; Tomilin N; Hanawalt P
Exp Cell Res; 2002 Jun; 276(2):284-95. PubMed ID: 12027458
[TBL] [Abstract][Full Text] [Related]
16. Sequence-specific and domain-specific DNA repair in xeroderma pigmentosum and Cockayne syndrome cells.
Tu Y; Bates S; Pfeifer GP
J Biol Chem; 1997 Aug; 272(33):20747-55. PubMed ID: 9252397
[TBL] [Abstract][Full Text] [Related]
17. Increased expression of p53 enhances transcription-coupled repair and global genomic repair of a UVC-damaged reporter gene in human cells.
Dregoesc D; Rybak AP; Rainbow AJ
DNA Repair (Amst); 2007 May; 6(5):588-601. PubMed ID: 17196445
[TBL] [Abstract][Full Text] [Related]
18. The human CSB (ERCC6) gene corrects the transcription-coupled repair defect in the CHO cell mutant UV61.
Orren DK; Dianov GL; Bohr VA
Nucleic Acids Res; 1996 Sep; 24(17):3317-22. PubMed ID: 8811084
[TBL] [Abstract][Full Text] [Related]
19. Gene-specific DNA repair of UV-induced cyclobutane pyrimidine dimers in some cancer-prone and premature-aging human syndromes.
Evans MK; Bohr VA
Mutat Res; 1994 May; 314(3):221-31. PubMed ID: 7513055
[TBL] [Abstract][Full Text] [Related]
20. Identification of a large genomic region in UV-irradiated human cells which has fewer cyclobutane pyrimidine dimers than most genomic regions.
Kantor GJ; Deiss-Tolbert DM
Photochem Photobiol; 1995 Aug; 62(2):263-70. PubMed ID: 7480136
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]