112 related articles for article (PubMed ID: 9464806)
1. Antigen presentation by T cells: T cell receptor ligation promotes antigen acquisition from professional antigen-presenting cells.
Arnold PY; Davidian DK; Mannie MD
Eur J Immunol; 1997 Dec; 27(12):3198-205. PubMed ID: 9464806
[TBL] [Abstract][Full Text] [Related]
2. Class II MHC/peptide complexes are released from APC and are acquired by T cell responders during specific antigen recognition.
Patel DM; Arnold PY; White GA; Nardella JP; Mannie MD
J Immunol; 1999 Nov; 163(10):5201-10. PubMed ID: 10553040
[TBL] [Abstract][Full Text] [Related]
3. Class II MHC/peptide complexes on T cell antigen-presenting cells: agonistic antigen recognition inhibits subsequent antigen presentation.
Mannie MD; Nardella JP; White GA; Arnold PY; Davidian DK
Cell Immunol; 1998 Jun; 186(2):111-20. PubMed ID: 9665753
[TBL] [Abstract][Full Text] [Related]
4. Anergy-associated T cell antigen presentation. A mechanism of infectious tolerance in experimental autoimmune encephalomyelitis.
Mannie MD; Rendall SK; Arnold PY; Nardella JP; White GA
J Immunol; 1996 Aug; 157(3):1062-70. PubMed ID: 8757610
[TBL] [Abstract][Full Text] [Related]
5. MHC class-II-restricted antigen presentation by myelin basic protein-specific CD4+ T cells causes prolonged desensitization and outgrowth of CD4- responders.
Mannie MD; Norris MS
Cell Immunol; 2001 Aug; 212(1):51-62. PubMed ID: 11716529
[TBL] [Abstract][Full Text] [Related]
6. Competition between foreign and self proteins in antigen presentation. Ovalbumin can inhibit activation of myelin basic protein-specific T cells.
Gautam AM; Glynn P
J Immunol; 1990 Feb; 144(4):1177-80. PubMed ID: 1689345
[TBL] [Abstract][Full Text] [Related]
7. Genetic control of the development of experimental allergic encephalomyelitis in rats. Separation of MHC and non-MHC gene effects.
Happ MP; Wettstein P; Dietzschold B; Heber-Katz E
J Immunol; 1988 Sep; 141(5):1489-94. PubMed ID: 2457618
[TBL] [Abstract][Full Text] [Related]
8. Differentiation of encephalitogenic T cells confers resistance to an inhibitory anti-CD4 monoclonal antibody.
Mannie MD; Morrison-Plummer J; McConnell TJ
J Immunol; 1993 Dec; 151(12):7293-306. PubMed ID: 7505025
[TBL] [Abstract][Full Text] [Related]
9. Feedback activation of T-cell antigen-presenting cells during interactions with T-cell responders.
Mannie MD; Walker MR
J Leukoc Biol; 2001 Aug; 70(2):252-60. PubMed ID: 11493617
[TBL] [Abstract][Full Text] [Related]
10. Identification and characterization of the antigen presenting cell in rat autoimmune myocarditis: evidence of bone marrow derivation and non-requirement for MHC class I compatibility with pathogenic T cells.
Ratcliffe NR; Wegmann KW; Zhao RW; Hickey WF
J Autoimmun; 2000 Nov; 15(3):369-79. PubMed ID: 11040077
[TBL] [Abstract][Full Text] [Related]
11. Lewis rat T cells can reutilize, process, and present myelin basic protein to antigen-specific T cell lines.
St Louis JM; Pasick JM; Stein C; Freeman D; Singh B; Dales S; Strejan GH
Cell Immunol; 1994 Jun; 156(1):36-53. PubMed ID: 7515332
[TBL] [Abstract][Full Text] [Related]
12. A monoclonal antibody specific for a cytochrome c T cell stimulatory peptide inhibits T cell responses and affects the way the peptide associates with antigen-presenting cells.
Jemmerson R; Johnson JG; Burrell E; Taylor PS; Jenkins MK
Eur J Immunol; 1991 Jan; 21(1):143-51. PubMed ID: 1846813
[TBL] [Abstract][Full Text] [Related]
13. Activation of specific T cell lines by the antigens avidin and myelin basic protein in the absence of antigen-presenting cells.
Altmann DM; Lider O; Douek DC; Cohen IR
Eur J Immunol; 1987 Nov; 17(11):1635-40. PubMed ID: 2445586
[TBL] [Abstract][Full Text] [Related]
14. MHC class II biosynthesis by activated rat CD4+ T cells: development of repression in vitro and modulation by APC-derived signals.
Mannie MD; Dawkins JG; Walker MR; Clayson BA; Patel DM
Cell Immunol; 2004 Jul; 230(1):33-43. PubMed ID: 15541717
[TBL] [Abstract][Full Text] [Related]
15. A cross-reactive idiotope on T cells from PL/J mice and Lewis rats that recognizes different myelin basic protein encephalitogenic epitopes but is restricted by TCR V beta 8.2.
Zhou SR; Whitaker JN; Han Q; Maier C; Blalock JE
J Immunol; 1994 Sep; 153(5):2340-51. PubMed ID: 7519648
[TBL] [Abstract][Full Text] [Related]
16. Activation of an encephalitogenic T lymphocyte line with a cellfree supernatant containing basic protein and I region gene products.
Vandenbark AA; Teal P; Offner H
J Immunol; 1987 Jan; 138(2):452-9. PubMed ID: 2432125
[TBL] [Abstract][Full Text] [Related]
17. Parallel costimulatory pathways promote myelin basic protein-stimulated proliferation of encephalitogenic rat T cells.
Mannie MD; Morrison-Plummer J; Torres-Garcia B; Hannaway C; Jones C; Smith AM
Cell Immunol; 1994 Feb; 153(2):312-28. PubMed ID: 7509724
[TBL] [Abstract][Full Text] [Related]
18. Analysis of the T cell repertoire for myelin basic protein in thymus-grafted and other types of chimera: evidence that major histocompatibility complex molecules on accessory cells rather than T cell specificity mainly regulate susceptibility to autoimmune encephalomyelitis.
Matsumoto Y; Kawai K; Fujiwara M
Eur J Immunol; 1990 Sep; 20(9):2119-26. PubMed ID: 1698640
[TBL] [Abstract][Full Text] [Related]
19. Superantigens induce primary T cell responses to soluble autoantigens by a non-V beta-specific mechanism of bystander activation.
Rott O; Mignon-Godefroy K; Fleischer B; Charreire J; Cash E
Cell Immunol; 1995 Apr; 161(2):158-65. PubMed ID: 7535195
[TBL] [Abstract][Full Text] [Related]
20. Autologous rat myelin basic protein is a partial agonist that is converted into a full antagonist upon blockade of CD4. Evidence for the integration of efficacious and nonefficacious signals during T cell antigen recognition.
Mannie MD; Rosser JM; White GA
J Immunol; 1995 Mar; 154(6):2642-54. PubMed ID: 7533179
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]