These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
113 related articles for article (PubMed ID: 9502590)
1. Allele-specific PCR analysis for detection of the gld Fas-ligand point mutation. Hoek RM; Kortekaas MC; Sedgwick JD J Immunol Methods; 1997 Dec; 210(1):109-12. PubMed ID: 9502590 [TBL] [Abstract][Full Text] [Related]
2. Generalized lymphoproliferative disease in mice, caused by a point mutation in the Fas ligand. Takahashi T; Tanaka M; Brannan CI; Jenkins NA; Copeland NG; Suda T; Nagata S Cell; 1994 Mar; 76(6):969-76. PubMed ID: 7511063 [TBL] [Abstract][Full Text] [Related]
3. Complete loss of Fas ligand gene causes massive lymphoproliferation and early death, indicating a residual activity of gld allele. Karray S; Kress C; Cuvellier S; Hue-Beauvais C; Damotte D; Babinet C; Lévi-Strauss M J Immunol; 2004 Feb; 172(4):2118-25. PubMed ID: 14764677 [TBL] [Abstract][Full Text] [Related]
4. Gld mutation of Fas ligand increases the frequency and up-regulates cell survival genes in CD25+CD4+ TR cells. Mohamood AS; Trujillo CJ; Zheng D; Jie C; Murillo FM; Schneck JP; Hamad AR Int Immunol; 2006 Aug; 18(8):1265-77. PubMed ID: 16769751 [TBL] [Abstract][Full Text] [Related]
5. Amelioration of lymphoid hyperplasia and hypergammaglobulinemia in lupus-prone mice (gld) by Fas-ligand gene transfer. Hong NM; Masuko-Hongo K; Sasakawa H; Kato T; Shirai T; Okumura K; Nishioka K; Kobata T J Autoimmun; 1998 Aug; 11(4):301-7. PubMed ID: 9776707 [TBL] [Abstract][Full Text] [Related]
6. Abnormal IgG galactosylation and arthritis in MRL-Fas(lpr) or MRL-FasL(gld) mice are under the control of the MRL genetic background. Kuroda Y; Nakata M; Nose M; Kojima N; Mizuochi T FEBS Lett; 2001 Oct; 507(2):210-4. PubMed ID: 11684100 [TBL] [Abstract][Full Text] [Related]
7. Apoptosis of nur77/N10-transgenic thymocytes involves the Fas/Fas ligand pathway. Weih F; Ryseck RP; Chen L; Bravo R Proc Natl Acad Sci U S A; 1996 May; 93(11):5533-8. PubMed ID: 8643610 [TBL] [Abstract][Full Text] [Related]
9. The mouse Fas-ligand gene is mutated in gld mice and is part of a TNF family gene cluster. Lynch DH; Watson ML; Alderson MR; Baum PR; Miller RE; Tough T; Gibson M; Davis-Smith T; Smith CA; Hunter K Immunity; 1994 May; 1(2):131-6. PubMed ID: 7889405 [TBL] [Abstract][Full Text] [Related]
10. Lack of Fas and Fas-L mutations in patients with lymphoproliferative disorders associated with Sjögren's syndrome and type II mixed cryoglobulinemia. Bertolo F; De Vita S; Dolcetti R; Carbone A; Ferraccioli GF; Bartoli E; Boiocchi M Clin Exp Rheumatol; 1999; 17(3):339-42. PubMed ID: 10410268 [TBL] [Abstract][Full Text] [Related]
11. Apoptosis mediated by the Fas system. Nagata S Prog Mol Subcell Biol; 1996; 16():87-103. PubMed ID: 8822794 [TBL] [Abstract][Full Text] [Related]
12. Preimplantation maternal stress impairs embryo development by inducing oviductal apoptosis with activation of the Fas system. Zheng LL; Tan XW; Cui XZ; Yuan HJ; Li H; Jiao GZ; Ji CL; Tan JH Mol Hum Reprod; 2016 Nov; 22(11):778-790. PubMed ID: 27475493 [TBL] [Abstract][Full Text] [Related]
13. Fas ligand (gld)- and Fas (lpr)-deficient mice do not show alterations in the extravasation or apoptosis of inflammatory neutrophils. Fecho K; Cohen PL J Leukoc Biol; 1998 Sep; 64(3):373-83. PubMed ID: 9738665 [TBL] [Abstract][Full Text] [Related]
14. Cellular interactions in the lpr and gld models of systemic autoimmunity. Sobel ES Adv Dent Res; 1996 Apr; 10(1):76-80. PubMed ID: 8934931 [TBL] [Abstract][Full Text] [Related]
15. Human autoimmune lymphoproliferative syndrome, a defect in the apoptosis-inducing Fas receptor: a lesson from the mouse model. Nagata S J Hum Genet; 1998; 43(1):2-8. PubMed ID: 9609991 [TBL] [Abstract][Full Text] [Related]
16. Characterization of the non-functional Fas ligand of gld mice. Hahne M; Peitsch MC; Irmler M; Schröter M; Lowin B; Rousseau M; Bron C; Renno T; French L; Tschopp J Int Immunol; 1995 Sep; 7(9):1381-6. PubMed ID: 7495745 [TBL] [Abstract][Full Text] [Related]