These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
218 related articles for article (PubMed ID: 9628896)
1. OCI-5/GPC3, a glypican encoded by a gene that is mutated in the Simpson-Golabi-Behmel overgrowth syndrome, induces apoptosis in a cell line-specific manner. Gonzalez AD; Kaya M; Shi W; Song H; Testa JR; Penn LZ; Filmus J J Cell Biol; 1998 Jun; 141(6):1407-14. PubMed ID: 9628896 [TBL] [Abstract][Full Text] [Related]
2. Glypican-3-deficient mice exhibit developmental overgrowth and some of the abnormalities typical of Simpson-Golabi-Behmel syndrome. Cano-Gauci DF; Song HH; Yang H; McKerlie C; Choo B; Shi W; Pullano R; Piscione TD; Grisaru S; Soon S; Sedlackova L; Tanswell AK; Mak TW; Yeger H; Lockwood GA; Rosenblum ND; Filmus J J Cell Biol; 1999 Jul; 146(1):255-64. PubMed ID: 10402475 [TBL] [Abstract][Full Text] [Related]
3. Mutational analysis of the GPC3/GPC4 glypican gene cluster on Xq26 in patients with Simpson-Golabi-Behmel syndrome: identification of loss-of-function mutations in the GPC3 gene. Veugelers M; Cat BD; Muyldermans SY; Reekmans G; Delande N; Frints S; Legius E; Fryns JP; Schrander-Stumpel C; Weidle B; Magdalena N; David G Hum Mol Genet; 2000 May; 9(9):1321-8. PubMed ID: 10814714 [TBL] [Abstract][Full Text] [Related]
4. GPC3 mutation analysis in a spectrum of patients with overgrowth expands the phenotype of Simpson-Golabi-Behmel syndrome. Li M; Shuman C; Fei YL; Cutiongco E; Bender HA; Stevens C; Wilkins-Haug L; Day-Salvatore D; Yong SL; Geraghty MT; Squire J; Weksberg R Am J Med Genet; 2001 Aug; 102(2):161-8. PubMed ID: 11477610 [TBL] [Abstract][Full Text] [Related]
5. GPC4, the gene for human K-glypican, flanks GPC3 on xq26: deletion of the GPC3-GPC4 gene cluster in one family with Simpson-Golabi-Behmel syndrome. Veugelers M; Vermeesch J; Watanabe K; Yamaguchi Y; Marynen P; David G Genomics; 1998 Oct; 53(1):1-11. PubMed ID: 9787072 [TBL] [Abstract][Full Text] [Related]
6. OCI-5/rat glypican-3 binds to fibroblast growth factor-2 but not to insulin-like growth factor-2. Song HH; Shi W; Filmus J J Biol Chem; 1997 Mar; 272(12):7574-7. PubMed ID: 9065409 [TBL] [Abstract][Full Text] [Related]
7. The Simpson-Golabi-Behmel syndrome causative glypican-3, binds to and inhibits the dipeptidyl peptidase activity of CD26. Davoodi J; Kelly J; Gendron NH; MacKenzie AE Proteomics; 2007 Jun; 7(13):2300-10. PubMed ID: 17549790 [TBL] [Abstract][Full Text] [Related]
8. Blocking endogenous glypican-3 expression releases Hep 3B cells from G1 arrest. Farooq M; Hwang SY; Park MK; Kim JC; Kim MK; Sung YK Mol Cells; 2003 Jun; 15(3):356-60. PubMed ID: 12872992 [TBL] [Abstract][Full Text] [Related]
9. Mutations in GPC3, a glypican gene, cause the Simpson-Golabi-Behmel overgrowth syndrome. Pilia G; Hughes-Benzie RM; MacKenzie A; Baybayan P; Chen EY; Huber R; Neri G; Cao A; Forabosco A; Schlessinger D Nat Genet; 1996 Mar; 12(3):241-7. PubMed ID: 8589713 [TBL] [Abstract][Full Text] [Related]
10. Glypican-3 expression is silenced in human breast cancer. Xiang YY; Ladeda V; Filmus J Oncogene; 2001 Nov; 20(50):7408-12. PubMed ID: 11704870 [TBL] [Abstract][Full Text] [Related]
11. The role of glypican-3 in the regulation of body size and cancer. Filmus J; Capurro M Cell Cycle; 2008 Sep; 7(18):2787-90. PubMed ID: 18787398 [TBL] [Abstract][Full Text] [Related]
12. Simpson Golabi Behmel syndrome: progress toward understanding the molecular basis for overgrowth, malformation, and cancer predisposition. DeBaun MR; Ess J; Saunders S Mol Genet Metab; 2001 Apr; 72(4):279-86. PubMed ID: 11286501 [TBL] [Abstract][Full Text] [Related]
13. Expression of OCI-5/glypican 3 during intestinal morphogenesis: regulation by cell shape in intestinal epithelial cells. Li M; Choo B; Wong ZM; Filmus J; Buick RN Exp Cell Res; 1997 Aug; 235(1):3-12. PubMed ID: 9281346 [TBL] [Abstract][Full Text] [Related]
14. Glypicans in growth control and cancer. Filmus J Glycobiology; 2001 Mar; 11(3):19R-23R. PubMed ID: 11320054 [TBL] [Abstract][Full Text] [Related]
15. Overgrowth of a mouse model of the Simpson-Golabi-Behmel syndrome is independent of IGF signaling. Chiao E; Fisher P; Crisponi L; Deiana M; Dragatsis I; Schlessinger D; Pilia G; Efstratiadis A Dev Biol; 2002 Mar; 243(1):185-206. PubMed ID: 11846487 [TBL] [Abstract][Full Text] [Related]
16. Hedgehog pathway modulation by glypican 3-conjugated heparan sulfate. Liu YC; Wierbowski BM; Salic A J Cell Sci; 2022 Mar; 135(6):. PubMed ID: 35142364 [TBL] [Abstract][Full Text] [Related]
17. Expression of GPC3, an X-linked recessive overgrowth gene, is silenced in malignant mesothelioma. Murthy SS; Shen T; De Rienzo A; Lee WC; Ferriola PC; Jhanwar SC; Mossman BT; Filmus J; Testa JR Oncogene; 2000 Jan; 19(3):410-6. PubMed ID: 10656689 [TBL] [Abstract][Full Text] [Related]
18. Genotype/phenotype correlations of males affected by Simpson-Golabi-Behmel syndrome with GPC3 gene mutations: patient report and review of the literature. Mariani S; Iughetti L; Bertorelli R; Coviello D; Pellegrini M; Forabosco A; Bernasconi S J Pediatr Endocrinol Metab; 2003 Feb; 16(2):225-32. PubMed ID: 12713262 [TBL] [Abstract][Full Text] [Related]
19. The Simpson-Golabi-Behmel gene, GPC3, is not involved in sporadic Wilms tumorigenesis. Gillan TL; Hughes R; Godbout R; Grundy PE Am J Med Genet A; 2003 Sep; 122A(1):30-6. PubMed ID: 12949968 [TBL] [Abstract][Full Text] [Related]
20. Glypicans and the biology of renal malformations. Grisaru S; Rosenblum ND Pediatr Nephrol; 2001 Mar; 16(3):302-6. PubMed ID: 11322381 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]