183 related articles for article (PubMed ID: 9712895)
1. N-linked glycosylation of the thyroid Na+/I- symporter (NIS). Implications for its secondary structure model.
Levy O; De la Vieja A; Ginter CS; Riedel C; Dai G; Carrasco N
J Biol Chem; 1998 Aug; 273(35):22657-63. PubMed ID: 9712895
[TBL] [Abstract][Full Text] [Related]
2. Characterization of the thyroid Na+/I- symporter with an anti-COOH terminus antibody.
Levy O; Dai G; Riedel C; Ginter CS; Paul EM; Lebowitz AN; Carrasco N
Proc Natl Acad Sci U S A; 1997 May; 94(11):5568-73. PubMed ID: 9159113
[TBL] [Abstract][Full Text] [Related]
3. The Q267E mutation in the sodium/iodide symporter (NIS) causes congenital iodide transport defect (ITD) by decreasing the NIS turnover number.
De La Vieja A; Ginter CS; Carrasco N
J Cell Sci; 2004 Feb; 117(Pt 5):677-87. PubMed ID: 14734652
[TBL] [Abstract][Full Text] [Related]
4. Post-transcriptional regulation of the sodium/iodide symporter by thyrotropin.
Riedel C; Levy O; Carrasco N
J Biol Chem; 2001 Jun; 276(24):21458-63. PubMed ID: 11290744
[TBL] [Abstract][Full Text] [Related]
5. Role of N-linked glycosylation in rat renal Na/Pi-cotransport.
Hayes G; Busch A; Lötscher M; Waldegger S; Lang F; Verrey F; Biber J; Murer H
J Biol Chem; 1994 Sep; 269(39):24143-9. PubMed ID: 7929070
[TBL] [Abstract][Full Text] [Related]
6. Identification of a structural requirement for thyroid Na+/I- symporter (NIS) function from analysis of a mutation that causes human congenital hypothyroidism.
Levy O; Ginter CS; De la Vieja A; Levy D; Carrasco N
FEBS Lett; 1998 Jun; 429(1):36-40. PubMed ID: 9657379
[TBL] [Abstract][Full Text] [Related]
7. The Na+/I- symporter (NIS): recent advances.
Levy O; De la Vieja A; Carrasco N
J Bioenerg Biomembr; 1998 Apr; 30(2):195-206. PubMed ID: 9672241
[TBL] [Abstract][Full Text] [Related]
8. Na(+)/I(-) symporter activity requires a small and uncharged amino acid residue at position 395.
Dohán O; Gavrielides MV; Ginter C; Amzel LM; Carrasco N
Mol Endocrinol; 2002 Aug; 16(8):1893-902. PubMed ID: 12145342
[TBL] [Abstract][Full Text] [Related]
9. The iodide-transport-defect-causing mutation R124H: a δ-amino group at position 124 is critical for maturation and trafficking of the Na+/I- symporter.
Paroder V; Nicola JP; Ginter CS; Carrasco N
J Cell Sci; 2013 Aug; 126(Pt 15):3305-13. PubMed ID: 23690546
[TBL] [Abstract][Full Text] [Related]
10. Characterization of gastric Na+/I- symporter of the rat.
Kotani T; Ogata Y; Yamamoto I; Aratake Y; Kawano JI; Suganuma T; Ohtaki S
Clin Immunol Immunopathol; 1998 Dec; 89(3):271-8. PubMed ID: 9837697
[TBL] [Abstract][Full Text] [Related]
11. Molecular characterization of V59E NIS, a Na+/I- symporter mutant that causes congenital I- transport defect.
Reed-Tsur MD; De la Vieja A; Ginter CS; Carrasco N
Endocrinology; 2008 Jun; 149(6):3077-84. PubMed ID: 18339708
[TBL] [Abstract][Full Text] [Related]
12. The Iodide Transport Defect-Causing Y348D Mutation in the Na
Reyna-Neyra A; Jung L; Chakrabarti M; Suárez MX; Amzel LM; Carrasco N
Thyroid; 2021 Aug; 31(8):1272-1281. PubMed ID: 33779310
[No Abstract] [Full Text] [Related]
13. Asn441 plays a key role in folding and function of the Na+/I- symporter (NIS).
Li W; Nicola JP; Amzel LM; Carrasco N
FASEB J; 2013 Aug; 27(8):3229-38. PubMed ID: 23650190
[TBL] [Abstract][Full Text] [Related]
14. Transforming growth factor-beta1 suppresses thyrotropin-induced Na+/I- symporter messenger RNA and protein levels in FRTL-5 rat thyroid cells.
Kawaguchi A; Ikeda M; Endo T; Kogai T; Miyazaki A; Onaya T
Thyroid; 1997 Oct; 7(5):789-94. PubMed ID: 9349586
[TBL] [Abstract][Full Text] [Related]
15. Molecular analysis of a congenital iodide transport defect: G543E impairs maturation and trafficking of the Na+/I- symporter.
De la Vieja A; Ginter CS; Carrasco N
Mol Endocrinol; 2005 Nov; 19(11):2847-58. PubMed ID: 15976004
[TBL] [Abstract][Full Text] [Related]
16. Molecular analysis of the sodium/iodide symporter: impact on thyroid and extrathyroid pathophysiology.
De La Vieja A; Dohan O; Levy O; Carrasco N
Physiol Rev; 2000 Jul; 80(3):1083-105. PubMed ID: 10893432
[TBL] [Abstract][Full Text] [Related]
17. A Carboxy-Terminal Monoleucine-Based Motif Participates in the Basolateral Targeting of the Na+/I- Symporter.
Martín M; Modenutti CP; Peyret V; Geysels RC; Darrouzet E; Pourcher T; Masini-Repiso AM; Martí MA; Carrasco N; Nicola JP
Endocrinology; 2019 Jan; 160(1):156-168. PubMed ID: 30496374
[TBL] [Abstract][Full Text] [Related]
18. Inability to N-glycosylate the human norepinephrine transporter reduces protein stability, surface trafficking, and transport activity but not ligand recognition.
Melikian HE; Ramamoorthy S; Tate CG; Blakely RD
Mol Pharmacol; 1996 Aug; 50(2):266-76. PubMed ID: 8700133
[TBL] [Abstract][Full Text] [Related]
19. Implications of the molecular characterization of the sodium-iodide symporter (NIS).
Schmutzler C; Köhrle J
Exp Clin Endocrinol Diabetes; 1998; 106 Suppl 3():S1-10. PubMed ID: 9865544
[TBL] [Abstract][Full Text] [Related]
20. Cell surface targeting accounts for the difference in iodide uptake activity between human Na+/I- symporter and rat Na+/I- symporter.
Zhang Z; Liu YY; Jhiang SM
J Clin Endocrinol Metab; 2005 Nov; 90(11):6131-40. PubMed ID: 16105966
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
