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6. Impairment of lagging strand synthesis triggers the formation of a RuvABC substrate at replication forks. Flores MJ; Bierne H; Ehrlich SD; Michel B EMBO J; 2001 Feb; 20(3):619-29. PubMed ID: 11157768 [TBL] [Abstract][Full Text] [Related]
7. Regression of replication forks stalled by leading-strand template damage: I. Both RecG and RuvAB catalyze regression, but RuvC cleaves the holliday junctions formed by RecG preferentially. Gupta S; Yeeles JT; Marians KJ J Biol Chem; 2014 Oct; 289(41):28376-87. PubMed ID: 25138216 [TBL] [Abstract][Full Text] [Related]
8. Cleavage of holliday junctions by the Escherichia coli RuvABC complex. Eggleston AK; West SC J Biol Chem; 2000 Aug; 275(34):26467-76. PubMed ID: 10851230 [TBL] [Abstract][Full Text] [Related]
9. Resolution of holliday junctions by RuvABC prevents dimer formation in rep mutants and UV-irradiated cells. Michel B; Recchia GD; Penel-Colin M; Ehrlich SD; Sherratt DJ Mol Microbiol; 2000 Jul; 37(1):180-91. PubMed ID: 10931315 [TBL] [Abstract][Full Text] [Related]
10. Replication fork reversal in DNA polymerase III mutants of Escherichia coli: a role for the beta clamp. Grompone G; Seigneur M; Ehrlich SD; Michel B Mol Microbiol; 2002 Jun; 44(5):1331-9. PubMed ID: 12028381 [TBL] [Abstract][Full Text] [Related]
11. Resolution of Holliday intermediates in recombination and DNA repair: indirect suppression of ruvA, ruvB, and ruvC mutations. Mandal TN; Mahdi AA; Sharples GJ; Lloyd RG J Bacteriol; 1993 Jul; 175(14):4325-34. PubMed ID: 8331065 [TBL] [Abstract][Full Text] [Related]
12. Cells defective for replication restart undergo replication fork reversal. Grompone G; Ehrlich D; Michel B EMBO Rep; 2004 Jun; 5(6):607-12. PubMed ID: 15167889 [TBL] [Abstract][Full Text] [Related]
13. Interactions between RuvA and RuvC at Holliday junctions: inhibition of junction cleavage and formation of a RuvA-RuvC-DNA complex. Whitby MC; Bolt EL; Chan SN; Lloyd RG J Mol Biol; 1996 Dec; 264(5):878-90. PubMed ID: 9000618 [TBL] [Abstract][Full Text] [Related]
14. Modulation of RNA polymerase by (p)ppGpp reveals a RecG-dependent mechanism for replication fork progression. McGlynn P; Lloyd RG Cell; 2000 Mar; 101(1):35-45. PubMed ID: 10778854 [TBL] [Abstract][Full Text] [Related]
15. RecA4142 causes SOS constitutive expression by loading onto reversed replication forks in Escherichia coli K-12. Long JE; Massoni SC; Sandler SJ J Bacteriol; 2010 May; 192(10):2575-82. PubMed ID: 20304994 [TBL] [Abstract][Full Text] [Related]
16. Loss of lambda prophage recombinogenicity in UV-irradiated Escherichia coli: the role of host genes ruvA, ruvB, ruvC, and recG. Zahradka K; Zahradka D; Petranović M Res Microbiol; 2001 Dec; 152(10):873-81. PubMed ID: 11766962 [TBL] [Abstract][Full Text] [Related]
17. Tandem repeat recombination induced by replication fork defects in Escherichia coli requires a novel factor, RadC. Saveson CJ; Lovett ST Genetics; 1999 May; 152(1):5-13. PubMed ID: 10224240 [TBL] [Abstract][Full Text] [Related]
18. Direct rescue of stalled DNA replication forks via the combined action of PriA and RecG helicase activities. Gregg AV; McGlynn P; Jaktaji RP; Lloyd RG Mol Cell; 2002 Feb; 9(2):241-51. PubMed ID: 11864599 [TBL] [Abstract][Full Text] [Related]
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20. RuvAB is essential for replication forks reversal in certain replication mutants. Baharoglu Z; Petranovic M; Flores MJ; Michel B EMBO J; 2006 Feb; 25(3):596-604. PubMed ID: 16424908 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]