BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

234 related articles for article (PubMed ID: 9840938)

  • 1. Prevention of mammalian DNA reduplication, following the release from the mitotic spindle checkpoint, requires p53 protein, but not p53-mediated transcriptional activity.
    Notterman D; Young S; Wainger B; Levine AJ
    Oncogene; 1998 Nov; 17(21):2743-51. PubMed ID: 9840938
    [TBL] [Abstract][Full Text] [Related]  

  • 2. p53 and pRb prevent rereplication in response to microtubule inhibitors by mediating a reversible G1 arrest.
    Khan SH; Wahl GM
    Cancer Res; 1998 Feb; 58(3):396-401. PubMed ID: 9458079
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Crosstalk of the mitotic spindle assembly checkpoint with p53 to prevent polyploidy.
    Vogel C; Kienitz A; Hofmann I; Müller R; Bastians H
    Oncogene; 2004 Sep; 23(41):6845-53. PubMed ID: 15286707
    [TBL] [Abstract][Full Text] [Related]  

  • 4. p53/p21(CIP1) cooperate in enforcing rapamycin-induced G(1) arrest and determine the cellular response to rapamycin.
    Huang S; Liu LN; Hosoi H; Dilling MB; Shikata T; Houghton PJ
    Cancer Res; 2001 Apr; 61(8):3373-81. PubMed ID: 11309295
    [TBL] [Abstract][Full Text] [Related]  

  • 5. The role of p53 in the response to mitotic spindle damage.
    Meek DW
    Pathol Biol (Paris); 2000 Apr; 48(3):246-54. PubMed ID: 10858957
    [TBL] [Abstract][Full Text] [Related]  

  • 6. IC261, a specific inhibitor of the protein kinases casein kinase 1-delta and -epsilon, triggers the mitotic checkpoint and induces p53-dependent postmitotic effects.
    Behrend L; Milne DM; Stöter M; Deppert W; Campbell LE; Meek DW; Knippschild U
    Oncogene; 2000 Nov; 19(47):5303-13. PubMed ID: 11103931
    [TBL] [Abstract][Full Text] [Related]  

  • 7. The farnesyltransferase inhibitor, FTI-2153, inhibits bipolar spindle formation during mitosis independently of transformation and Ras and p53 mutation status.
    Crespo NC; Delarue F; Ohkanda J; Carrico D; Hamilton AD; Sebti SM
    Cell Death Differ; 2002 Jul; 9(7):702-9. PubMed ID: 12058275
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Mechanism of hyperploid cell formation induced by microtubule inhibiting drug in glioma cell lines.
    Tsuiki H; Nitta M; Tada M; Inagaki M; Ushio Y; Saya H
    Oncogene; 2001 Jan; 20(4):420-9. PubMed ID: 11313973
    [TBL] [Abstract][Full Text] [Related]  

  • 9. p53 activation in response to mitotic spindle damage requires signaling via BubR1-mediated phosphorylation.
    Ha GH; Baek KH; Kim HS; Jeong SJ; Kim CM; McKeon F; Lee CW
    Cancer Res; 2007 Aug; 67(15):7155-64. PubMed ID: 17671183
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Requirement of a functional spindle checkpoint for arsenite-induced apoptosis.
    Wu YC; Yen WY; Yih LH
    J Cell Biochem; 2008 Oct; 105(3):678-87. PubMed ID: 18668508
    [TBL] [Abstract][Full Text] [Related]  

  • 11. p53 protein accumulation in addition to the transactivation activity is required for p53-dependent cell cycle arrest after treatment of cells with camptothecin.
    Jaks V; Jõers A; Kristjuhan A; Maimets T
    Oncogene; 2001 Mar; 20(10):1212-9. PubMed ID: 11313865
    [TBL] [Abstract][Full Text] [Related]  

  • 12. G1 tetraploidy checkpoint and the suppression of tumorigenesis.
    Margolis RL; Lohez OD; Andreassen PR
    J Cell Biochem; 2003 Mar; 88(4):673-83. PubMed ID: 12577301
    [TBL] [Abstract][Full Text] [Related]  

  • 13. High and low fluences of alpha-particles induce a G1 checkpoint in human diploid fibroblasts.
    Azzam EI; de Toledo SM; Waker AJ; Little JB
    Cancer Res; 2000 May; 60(10):2623-31. PubMed ID: 10825133
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Multiple centrosomes arise from tetraploidy checkpoint failure and mitotic centrosome clusters in p53 and RB pocket protein-compromised cells.
    Borel F; Lohez OD; Lacroix FB; Margolis RL
    Proc Natl Acad Sci U S A; 2002 Jul; 99(15):9819-24. PubMed ID: 12119403
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Cell cycle-dependent regulation of nuclear p53 traffic occurs in one subclass of human tumor cells and in untransformed cells.
    David-Pfeuty T; Chakrani F; Ory K; Nouvian-Dooghe Y
    Cell Growth Differ; 1996 Sep; 7(9):1211-25. PubMed ID: 8877102
    [TBL] [Abstract][Full Text] [Related]  

  • 16. p130/p107/p105Rb-dependent transcriptional repression during DNA-damage-induced cell-cycle exit at G2.
    Jackson MW; Agarwal MK; Yang J; Bruss P; Uchiumi T; Agarwal ML; Stark GR; Taylor WR
    J Cell Sci; 2005 May; 118(Pt 9):1821-32. PubMed ID: 15827088
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Role of retinoblastoma gene product in p53-mediated DNA damage response.
    Smith ML; Zhan Q; Bae I; Fornace AJ
    Exp Cell Res; 1994 Dec; 215(2):386-9. PubMed ID: 7982477
    [TBL] [Abstract][Full Text] [Related]  

  • 18. A transcriptional activation function of p53 is dispensable for and inhibitory of its apoptotic function.
    Kokontis JM; Wagner AJ; O'Leary M; Liao S; Hay N
    Oncogene; 2001 Feb; 20(6):659-68. PubMed ID: 11313999
    [TBL] [Abstract][Full Text] [Related]  

  • 19. p53-independent apoptosis and p53-dependent block of DNA rereplication following mitotic spindle inhibition in human cells.
    Casenghi M; Mangiacasale R; Tuynder M; Caillet-Fauquet P; Elhajouji A; Lavia P; Mousset S; Kirsch-Volders M; Cundari E
    Exp Cell Res; 1999 Aug; 250(2):339-50. PubMed ID: 10413588
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Human tumor-derived p53 proteins exhibit binding site selectivity and temperature sensitivity for transactivation in a yeast-based assay.
    Di Como CJ; Prives C
    Oncogene; 1998 May; 16(19):2527-39. PubMed ID: 9627118
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 12.