These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
171 related articles for article (PubMed ID: 9853964)
1. Gpc3 expression correlates with the phenotype of the Simpson-Golabi-Behmel syndrome. Pellegrini M; Pilia G; Pantano S; Lucchini F; Uda M; Fumi M; Cao A; Schlessinger D; Forabosco A Dev Dyn; 1998 Dec; 213(4):431-9. PubMed ID: 9853964 [TBL] [Abstract][Full Text] [Related]
2. A clinical and molecular study of a patient with Simpson-Golabi-Behmel syndrome. Okamoto N; Yagi M; Imura K; Wada Y J Hum Genet; 1999; 44(5):327-9. PubMed ID: 10496077 [TBL] [Abstract][Full Text] [Related]
3. Glypican-3-deficient mice exhibit developmental overgrowth and some of the abnormalities typical of Simpson-Golabi-Behmel syndrome. Cano-Gauci DF; Song HH; Yang H; McKerlie C; Choo B; Shi W; Pullano R; Piscione TD; Grisaru S; Soon S; Sedlackova L; Tanswell AK; Mak TW; Yeger H; Lockwood GA; Rosenblum ND; Filmus J J Cell Biol; 1999 Jul; 146(1):255-64. PubMed ID: 10402475 [TBL] [Abstract][Full Text] [Related]
4. Glypican 3 and glypican 4 are juxtaposed in Xq26.1. Huber R; Mazzarella R; Chen CN; Chen E; Ireland M; Lindsay S; Pilia G; Crisponi L Gene; 1998 Dec; 225(1-2):9-16. PubMed ID: 9931407 [TBL] [Abstract][Full Text] [Related]
5. Mutations in GPC3, a glypican gene, cause the Simpson-Golabi-Behmel overgrowth syndrome. Pilia G; Hughes-Benzie RM; MacKenzie A; Baybayan P; Chen EY; Huber R; Neri G; Cao A; Forabosco A; Schlessinger D Nat Genet; 1996 Mar; 12(3):241-7. PubMed ID: 8589713 [TBL] [Abstract][Full Text] [Related]
6. GPC4, the gene for human K-glypican, flanks GPC3 on xq26: deletion of the GPC3-GPC4 gene cluster in one family with Simpson-Golabi-Behmel syndrome. Veugelers M; Vermeesch J; Watanabe K; Yamaguchi Y; Marynen P; David G Genomics; 1998 Oct; 53(1):1-11. PubMed ID: 9787072 [TBL] [Abstract][Full Text] [Related]
8. Large scale deletions in the GPC3 gene may account for a minority of cases of Simpson-Golabi-Behmel syndrome. Lindsay S; Ireland M; O'Brien O; Clayton-Smith J; Hurst JA; Mann J; Cole T; Sampson J; Slaney S; Schlessinger D; Burn J; Pilia G J Med Genet; 1997 Jun; 34(6):480-3. PubMed ID: 9192268 [TBL] [Abstract][Full Text] [Related]
9. Mapping of the Simpson-Golabi-Behmel overgrowth syndrome gene (GPC3) to chromosome X in human and rat by fluorescence in situ hybridization. Shen T; Sonoda G; Hamid J; Li M; Filmus J; Buick RN; Testa JR Mamm Genome; 1997 Jan; 8(1):72. PubMed ID: 9021160 [No Abstract] [Full Text] [Related]
10. A small interstitial deletion in the GPC3 gene causes Simpson-Golabi-Behmel syndrome in a Dutch-Canadian family. Xuan JY; Hughes-Benzie RM; MacKenzie AE J Med Genet; 1999 Jan; 36(1):57-8. PubMed ID: 9950367 [TBL] [Abstract][Full Text] [Related]
11. Frequent silencing of the GPC3 gene in ovarian cancer cell lines. Lin H; Huber R; Schlessinger D; Morin PJ Cancer Res; 1999 Feb; 59(4):807-10. PubMed ID: 10029067 [TBL] [Abstract][Full Text] [Related]
12. Mutational analysis of the GPC3/GPC4 glypican gene cluster on Xq26 in patients with Simpson-Golabi-Behmel syndrome: identification of loss-of-function mutations in the GPC3 gene. Veugelers M; Cat BD; Muyldermans SY; Reekmans G; Delande N; Frints S; Legius E; Fryns JP; Schrander-Stumpel C; Weidle B; Magdalena N; David G Hum Mol Genet; 2000 May; 9(9):1321-8. PubMed ID: 10814714 [TBL] [Abstract][Full Text] [Related]
13. Expression of the cell surface proteoglycan glypican-5 is developmentally regulated in kidney, limb, and brain. Saunders S; Paine-Saunders S; Lander AD Dev Biol; 1997 Oct; 190(1):78-93. PubMed ID: 9331333 [TBL] [Abstract][Full Text] [Related]
14. Multiple Sp1 sites efficiently drive transcription of the TATA-less promoter of the human glypican 3 (GPC3) gene. Huber R; Schlessinger D; Pilia G Gene; 1998 Jul; 214(1-2):35-44. PubMed ID: 9651473 [TBL] [Abstract][Full Text] [Related]
15. Overgrowth of a mouse model of the Simpson-Golabi-Behmel syndrome is independent of IGF signaling. Chiao E; Fisher P; Crisponi L; Deiana M; Dragatsis I; Schlessinger D; Pilia G; Efstratiadis A Dev Biol; 2002 Mar; 243(1):185-206. PubMed ID: 11846487 [TBL] [Abstract][Full Text] [Related]
16. Hedgehog pathway modulation by glypican 3-conjugated heparan sulfate. Liu YC; Wierbowski BM; Salic A J Cell Sci; 2022 Mar; 135(6):. PubMed ID: 35142364 [TBL] [Abstract][Full Text] [Related]
17. Simpson Golabi Behmel syndrome: progress toward understanding the molecular basis for overgrowth, malformation, and cancer predisposition. DeBaun MR; Ess J; Saunders S Mol Genet Metab; 2001 Apr; 72(4):279-86. PubMed ID: 11286501 [TBL] [Abstract][Full Text] [Related]
18. Overgrowth syndromes and genomic imprinting: from mouse to man. Li M; Squire JA; Weksberg R Clin Genet; 1998 Mar; 53(3):165-70. PubMed ID: 9630066 [TBL] [Abstract][Full Text] [Related]
19. GPC3 mutation analysis in a spectrum of patients with overgrowth expands the phenotype of Simpson-Golabi-Behmel syndrome. Li M; Shuman C; Fei YL; Cutiongco E; Bender HA; Stevens C; Wilkins-Haug L; Day-Salvatore D; Yong SL; Geraghty MT; Squire J; Weksberg R Am J Med Genet; 2001 Aug; 102(2):161-8. PubMed ID: 11477610 [TBL] [Abstract][Full Text] [Related]
20. GPC6, a novel member of the glypican gene family, encodes a product structurally related to GPC4 and is colocalized with GPC5 on human chromosome 13. Paine-Saunders S; Viviano BL; Saunders S Genomics; 1999 May; 57(3):455-8. PubMed ID: 10329016 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]