These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

234 related articles for article (PubMed ID: 9973365)

  • 1. Cloning and characterization of arylamine N-acetyltransferase genes from Mycobacterium smegmatis and Mycobacterium tuberculosis: increased expression results in isoniazid resistance.
    Payton M; Auty R; Delgoda R; Everett M; Sim E
    J Bacteriol; 1999 Feb; 181(4):1343-7. PubMed ID: 9973365
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Evidence towards the role of arylamine N-acetyltransferase in Mycobacterium smegmatis and development of a specific antiserum against the homologous enzyme of Mycobacterium tuberculosis.
    Payton M; Gifford C; Schartau P; Hagemeier C; Mushtaq A; Lucas S; Pinter K; Sim E
    Microbiology (Reading); 2001 Dec; 147(Pt 12):3295-302. PubMed ID: 11739761
    [TBL] [Abstract][Full Text] [Related]  

  • 3. The structure of arylamine N-acetyltransferase from Mycobacterium smegmatis--an enzyme which inactivates the anti-tubercular drug, isoniazid.
    Sandy J; Mushtaq A; Kawamura A; Sinclair J; Sim E; Noble M
    J Mol Biol; 2002 May; 318(4):1071-83. PubMed ID: 12054803
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Binding of the anti-tubercular drug isoniazid to the arylamine N-acetyltransferase protein from Mycobacterium smegmatis.
    Sandy J; Holton S; Fullam E; Sim E; Noble M
    Protein Sci; 2005 Mar; 14(3):775-82. PubMed ID: 15722451
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Structural investigation of mutant Mycobacterium smegmatis arylamine N-acetyltransferase: a model for a naturally occurring functional polymorphism in Mycobacterium tuberculosis arylamine N-acetyltransferase.
    Kawamura A; Sandy J; Upton A; Noble M; Sim E
    Protein Expr Purif; 2003 Jan; 27(1):75-84. PubMed ID: 12509987
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Arylamine N-acetyltransferase of Mycobacterium tuberculosis is a polymorphic enzyme and a site of isoniazid metabolism.
    Upton AM; Mushtaq A; Victor TC; Sampson SL; Sandy J; Smith DM; van Helden PV; Sim E
    Mol Microbiol; 2001 Oct; 42(2):309-17. PubMed ID: 11703656
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Expression, purification, characterization and structure of Pseudomonas aeruginosa arylamine N-acetyltransferase.
    Westwood IM; Holton SJ; Rodrigues-Lima F; Dupret JM; Bhakta S; Noble ME; Sim E
    Biochem J; 2005 Jan; 385(Pt 2):605-12. PubMed ID: 15447630
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Arylamine N-acetyltransferases in mycobacteria.
    Sim E; Sandy J; Evangelopoulos D; Fullam E; Bhakta S; Westwood I; Krylova A; Lack N; Noble M
    Curr Drug Metab; 2008 Jul; 9(6):510-9. PubMed ID: 18680471
    [TBL] [Abstract][Full Text] [Related]  

  • 9. The mabA gene from the inhA operon of Mycobacterium tuberculosis encodes a 3-ketoacyl reductase that fails to confer isoniazid resistance.
    Banerjee A; Sugantino M; Sacchettini JC; Jacobs WR
    Microbiology (Reading); 1998 Oct; 144 ( Pt 10)():2697-2704. PubMed ID: 9802011
    [TBL] [Abstract][Full Text] [Related]  

  • 10. High throughput screening of 7-methylpicene-1,2-diol as arylamine N-acetyltransferase (NAT) inhibitor to establish a isoniazid supplement in anti-tubercular therapy.
    Chowdhury A; Paul P; Choudhury MD
    Comb Chem High Throughput Screen; 2013 Nov; 16(9):721-5. PubMed ID: 23701008
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Oxidative stress response and its role in sensitivity to isoniazid in mycobacteria: characterization and inducibility of ahpC by peroxides in Mycobacterium smegmatis and lack of expression in M. aurum and M. tuberculosis.
    Dhandayuthapani S; Zhang Y; Mudd MH; Deretic V
    J Bacteriol; 1996 Jun; 178(12):3641-9. PubMed ID: 8655566
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Kinetic and chemical mechanism of arylamine N-acetyltransferase from Mycobacterium tuberculosis.
    Sikora AL; Frankel BA; Blanchard JS
    Biochemistry; 2008 Oct; 47(40):10781-9. PubMed ID: 18795795
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Characterisation of a putative AraC transcriptional regulator from Mycobacterium smegmatis.
    Evangelopoulos D; Gupta A; Lack NA; Maitra A; ten Bokum AM; Kendall S; Sim E; Bhakta S
    Tuberculosis (Edinb); 2014 Dec; 94(6):664-71. PubMed ID: 25443504
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Variants of katG, inhA and nat genes are not associated with mutations in efflux pump genes (mmpL3 and mmpL7) in isoniazid-resistant clinical isolates of Mycobacterium tuberculosis from India.
    Unissa AN; Dusthackeer VNA; Kumar MP; Nagarajan P; Sukumar S; Kumari VI; Lakshmi AR; Hanna LE
    Tuberculosis (Edinb); 2017 Dec; 107():144-148. PubMed ID: 29050763
    [TBL] [Abstract][Full Text] [Related]  

  • 15. AhpC, oxidative stress and drug resistance in Mycobacterium tuberculosis.
    Sherman DR; Mdluli K; Hickey MJ; Barry CE; Stover CK
    Biofactors; 1999; 10(2-3):211-7. PubMed ID: 10609885
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Arylamine N-acetyltransferases: a pharmacogenomic approach to drug metabolism and endogenous function.
    Sim E; Pinter K; Mushtaq A; Upton A; Sandy J; Bhakta S; Noble M
    Biochem Soc Trans; 2003 Jun; 31(Pt 3):615-9. PubMed ID: 12773167
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Expression and purification of the rifamycin amide synthase, RifF, an enzyme homologous to the prokaryotic arylamine N-acetyltransferases.
    Pompeo F; Mushtaq A; Sim E
    Protein Expr Purif; 2002 Feb; 24(1):138-51. PubMed ID: 11812235
    [TBL] [Abstract][Full Text] [Related]  

  • 18. [Establishment and drug susceptibility test of isoniazid resistant Mycobacterium smegmatis].
    Jia PP; Zhao LL; Li XY; Zhang Q; Liu ZL; Wang X; Yu LY; Zhao LX; Cen S
    Yao Xue Xue Bao; 2011 Apr; 46(4):377-82. PubMed ID: 21751489
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Rv1255c, a dormancy-related transcriptional regulator of TetR family in Mycobacterium tuberculosis, enhances isoniazid tolerance in Mycobacterium smegmatis.
    Gopi Reji J; K Edison L; Raghunandanan S; Pushparajan AR; Kurthkoti K; Ajay Kumar R
    J Antibiot (Tokyo); 2023 Dec; 76(12):720-727. PubMed ID: 37821540
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Overexpression of inhA, but not kasA, confers resistance to isoniazid and ethionamide in Mycobacterium smegmatis, M. bovis BCG and M. tuberculosis.
    Larsen MH; Vilchèze C; Kremer L; Besra GS; Parsons L; Salfinger M; Heifets L; Hazbon MH; Alland D; Sacchettini JC; Jacobs WR
    Mol Microbiol; 2002 Oct; 46(2):453-66. PubMed ID: 12406221
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 12.