These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
Pubmed for Handhelds
PUBMED FOR HANDHELDS
Search MEDLINE/PubMed
Title: Endurance training reduces end-exercise VO2 and muscle use during submaximal cycling. Author: Saunders MJ, Evans EM, Arngrimsson SA, Allison JD, Cureton KJ. Journal: Med Sci Sports Exerc; 2003 Feb; 35(2):257-62. PubMed ID: 12569214. Abstract: INTRODUCTION: End-exercise VO2 during heavy, constant-load exercise is reduced after endurance training, due to an attenuated VO2 slow component. PURPOSE/METHODS: To determine whether the training-induced reduction in end-exercise VO2 was associated with reduced muscle use, we measured VO2 and T2 changes in magnetic resonance images in the final minute of two 15-min constant-load cycle rides, one above lactate threshold and the other below lactate threshold. These measures were repeated after a 4-wk period in eight subjects who trained on a cycle ergometer and seven controls. RESULTS: There were no changes in end-exercise VO2 or active muscle after training in either group during low-intensity cycling, in which no VO2 slow component was present. During high-intensity cycling, in which there was a slow component before training, the training group experienced a significant reduction (P < 0.05) in end-exercise VO2 (2625 +/- 673; 2567 +/- 605 mL.min (-1) and the T2 of the vastus lateralis (35.6 +/- 1.4; 34.5 +/- 0.9 ms). CONCLUSION: These results support the hypothesis that reduction in end-exercise VO2 (and the VO2 slow component) after training is due to reduced muscle use during heavy, constant load cycling.[Abstract] [Full Text] [Related] [New Search]